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1  stenosis and low to intermediate-complexity coronary artery disease.
2 n of patients with known or suspected stable coronary artery disease.
3 irin (ASA) desensitization for patients with coronary artery disease.
4 mpared with MT alone in patients with stable coronary artery disease.
5 the potent P2Y12 inhibitors in patients with coronary artery disease.
6 cluding hypertension, diabetes mellitus, and coronary artery disease.
7 phoma Kinase) is linked to a protection from coronary artery disease.
8  links to BMI, adiposity-related traits, and coronary artery disease.
9 , bipolar disorder, Alzheimer's disease, and coronary artery disease.
10 s in patients with multivessel and left main coronary artery disease.
11 old balloon angioplasty in the management of coronary artery disease.
12 c plaque, which may be a subsequent risk for coronary artery disease.
13 n hypercholesterolemic pigs with preexisting coronary artery disease.
14 utpatients (age: 63+/-9 years; 76% men) with coronary artery disease.
15 stitutes a serious problem for subjects with coronary artery disease.
16 edical therapy (OMT) for patients with known coronary artery disease.
17          This paper reports on patients with coronary artery disease.
18 enotypes of patients with known or suspected coronary artery disease.
19 ody weight affects outcomes in patients with coronary artery disease.
20 re, cardiac masses, pericardial disease, and coronary artery disease.
21 tient-tailored strategy for the treatment of coronary artery disease.
22 ic morphologic and physiologic assessment of coronary artery disease.
23 pen-label clinical trial in 50 patients with coronary artery disease.
24  patients with chest pain and nonobstructive coronary artery disease.
25 d in epicardial AT of humans with or without coronary artery disease.
26 vasive coronary angiography and had no known coronary artery disease.
27 xpression of their genetic susceptibility to coronary artery disease.
28  precursor have been associated with risk of coronary artery disease.
29 e, 89% white race, and 12% with a history of coronary artery disease.
30 on revascularization in patients with stable coronary artery disease.
31 r against each other in patients with stable coronary artery disease.
32 onary physiology in the modern management of coronary artery disease.
33  hemorrhagic shock in swine with preexisting coronary artery disease.
34  complications in the presence of underlying coronary artery disease.
35 pressure-lowering treatment in patients with coronary artery disease.
36 h significant or suggestive association with coronary artery disease.
37 or evaluating patients with suspected stable coronary artery disease.
38 recludes assessing physiological severity of coronary artery disease.
39 G for the treatment of unprotected left main coronary artery disease.
40 for PCI with OMT versus OMT alone for stable coronary artery disease.
41 utpatients (age: 63+/-9 years; 76% men) with coronary artery disease.
42 AIS patients had no angiographic evidence of coronary artery disease.
43 large cohort of patients suspected of having coronary artery disease.
44 raphy, as compared with patients with stable coronary artery disease.
45 ain at least 90% of the attributable risk of coronary artery disease.
46 cibility of FFRangio in patients with stable coronary artery disease.
47  disease in comparison with controls without coronary artery disease.
48  relevance for patients with risk factors or coronary artery disease.
49                    Prevalence of obstructive coronary artery disease (1%-64%), ACS (1%-44%), downstre
50 vation myocardial infarction and multivessel coronary artery disease: 1-stage percutaneous coronary i
51 rtension (77%), diabetes mellitus (31%), and coronary artery disease (15%).
52 were sudden arrhythmic death syndrome (31%), coronary artery disease (22%), and hypertrophic cardiomy
53 agnosis of cardiovascular disease, including coronary artery disease (26.6 to 12.6% of admissions) an
54 nsion (47 % and 31 % vs 15 %, P < 0.001) and coronary artery disease (29 % and 18 % vs 3 %, P < 0.001
55 test declining primary cardiac diagnosis was coronary artery disease (32.3%-19.0%; P<0.001).
56 n DNA sequence variants for association with coronary artery disease (4,831 cases and 115,455 control
57 were common, including hypertension (82.5%), coronary artery disease (42.6%), and diabetes mellitus (
58                  Main comorbidities included coronary artery disease (51.5%), renal insufficiency (27
59 gists by 2 1 method required the presence of coronary artery disease, a common interpretation of the
60 e kidney transplantation commonly focuses on coronary artery disease, a comprehensive pretransplant c
61 s associated with mortality in patients with coronary artery disease, a finding that is independent o
62      INTERPRETATION: In patients with stable coronary artery disease, addition of rivaroxaban to aspi
63 ors of potentially fatal arrhythmias without coronary artery disease admitted to our institutions fro
64 Antiplatelet therapy is of proven benefit in coronary artery disease and a number of other clinical s
65  found to be appropriate because obstructive coronary artery disease and ACS were more prevalent in p
66           Among patients who had multivessel coronary artery disease and acute myocardial infarction
67 Cardiac risk was assessed by noting baseline coronary artery disease and calculating the WHO/Internat
68 e 4-BRS was also associated with severity of coronary artery disease and composite end points.
69 21.3 region are consistently associated with coronary artery disease and coronary artery calcificatio
70                         Patients with stable coronary artery disease and DM exhibit a burden of angin
71 ated loci for Alzheimer's disease, eight for coronary artery disease and five for type 2 diabetes.
72                                              Coronary artery disease and flail leaflet were seen in 1
73 iously reported significant association with coronary artery disease and GRS153 of 153 single-nucleot
74 onal cardiologists to assess the severity of coronary artery disease and guide treatment, coronary an
75 r mixed methods investigation (compared with coronary artery disease and hypertension).
76        Among patients with documented stable coronary artery disease and in whom no revascularization
77                                              Coronary artery disease and its risk factors are some of
78 becoming increasingly popular in the area of coronary artery disease and its risk factors, many of th
79  other or medical treatment in patients with coronary artery disease and left ventricular ejection fr
80 istics of published mixed methods studies on coronary artery disease and major risk factors (diabetes
81 ated to treatment (two in the control group [coronary artery disease and multiorgan failure] and thre
82 ty patients with a low pretest likelihood of coronary artery disease and normal findings on stress pe
83 he context of a recent GWAS meta-analysis of coronary artery disease and provide a list of targeted e
84 atins have been used for 30 years to prevent coronary artery disease and stroke.
85 robiota-dependent metabolite associated with coronary artery disease and stroke.
86 Dyslipidemia is an important risk factor for coronary artery disease and stroke.
87 we describe GWAB-boosted candidate genes for coronary artery disease and supporting data in the liter
88                BMI, systolic blood pressure, coronary artery disease and type 2 diabetes showed negat
89 revent sudden cardiac death in patients with coronary artery disease and unstable ventricular tachyar
90  T are associated with more atherosclerosis, coronary artery disease, and cardiovascular events.
91 ified by age, sex, risk factors, presence of coronary artery disease, and early presentation.
92 ion, hypercholesterolemia, family history of coronary artery disease, and known coronary artery disea
93 outh), BMI, height, systolic blood pressure, coronary artery disease, and type 2 diabetes using data
94         Epicardial adipose tissue volume and coronary artery disease are strongly associated, even af
95  intervention (PCI) for radiation-associated coronary artery disease are unknown.
96 nically, CVD in this population manifests as coronary artery disease, arrhythmias, stroke, or congest
97                       Although virtually all coronary artery disease associated single-nucleotide pol
98 ients were older, with a lower prevalence of coronary artery disease, atrial fibrillation, and diabet
99 lead AAA single nucleotide polymorphisms and coronary artery disease, blood pressure, lipids, or diab
100 ally attributable to an elevated subclinical coronary artery disease burden composed of noncalcified
101 computed tomographic angiography to quantify coronary artery disease burden.
102 y is an important tool for the evaluation of coronary artery disease but often correlates poorly with
103 medical therapy (MT) in patients with stable coronary artery disease, but PCI was guided by angiograp
104 ated with lower risks of type 2 diabetes and coronary artery disease, but their relations with interm
105 ance imaging, after exclusion of obstructive coronary artery disease by angiography.
106  liter) (P=1.0x10(-16)), and a lower risk of coronary artery disease (by 34%; 95% confidence interval
107 sterol, low-density lipoprotein cholesterol, coronary artery disease, C-reactive protein, HbA1c, heig
108 tion and Meta-analysis [CARDIoGRAM] plus The Coronary Artery Disease [C4D] Genetics; combined total 6
109 ce standard for the functional assessment of coronary artery disease (CAD) ( 1 , 2 ).
110  We also conduct parallel investigations for coronary artery disease (CAD) (viewed as a positive cont
111                            The prevalence of coronary artery disease (CAD) among patients with refrac
112 ave identified multiple loci associated with coronary artery disease (CAD) among predominantly Europe
113 nd local site interpretation for significant coronary artery disease (CAD) and cardiovascular events.
114 lated with QTc prolongation in patients with coronary artery disease (CAD) and investigate the effect
115 to elevated serum calcium levels and risk of coronary artery disease (CAD) and myocardial infarction
116 c factor with therapeutic potential to treat coronary artery disease (CAD) and myocardial infarction
117 obstructive (>/=50% stenosis) left main (LM) coronary artery disease (CAD) are at high risk for adver
118                                 The value of coronary artery disease (CAD) assessment and coronary in
119    Genome-wide association studies (GWAS) in coronary artery disease (CAD) had identified 66 loci at
120 ) trial, patients with 3-vessel or left main coronary artery disease (CAD) had improved long-term out
121 for determining the presence and severity of coronary artery disease (CAD) in patients with sign and
122                                              Coronary artery disease (CAD) is a chronic inflammatory
123                                   RATIONALE: Coronary artery disease (CAD) is a complex phenotype dri
124                                              Coronary artery disease (CAD) is a leading cause of morb
125                                              Coronary artery disease (CAD) is a significant problem d
126 asive testing for a diagnosis of significant coronary artery disease (CAD) is ambiguous, but nuclear
127 gher for women than for men, yet obstructive coronary artery disease (CAD) is less prevalent in women
128                                              Coronary artery disease (CAD) is the leading cause of de
129                                              Coronary artery disease (CAD) is the number one cause of
130   Whether ANGPTL3 deficiency reduces risk of coronary artery disease (CAD) is unknown.
131 ovided a rich collection of approximately 58 coronary artery disease (CAD) loci that suggest the exis
132 oninvasive models to predict the presence of coronary artery disease (CAD) may help reduce the societ
133 ne, has mechanistic links to atherosclerotic coronary artery disease (CAD) pathogenesis and is associ
134 tly associated with cardiovascular events in coronary artery disease (CAD) patients and reducing the
135 n stable symptomatic patients with suspected coronary artery disease (CAD) requiring noninvasive test
136 e molecular off-target effects and increased coronary artery disease (CAD) risk.
137  sought to describe an optimized approach to coronary artery disease (CAD) screening and management i
138                                              Coronary artery disease (CAD) severity was quantified in
139  dawned for the prevention and management of coronary artery disease (CAD) utilizing genetic risk var
140                      METHODS AND Obstructive coronary artery disease (CAD) was defined as >/=50% sten
141                                     Comorbid coronary artery disease (CAD) was present in 24.3% of vi
142 rmediate pretest probability for obstructive coronary artery disease (CAD) were randomly assigned to
143 ion could beneficially affect progression of coronary artery disease (CAD) when compared with transpl
144 for atherosclerosis, the underlying cause of coronary artery disease (CAD), a major cause of morbidit
145 re among the most robust genetic markers for coronary artery disease (CAD), and previous studies have
146 causes, any CVD, atherosclerotic CVD (ACVD), coronary artery disease (CAD), and stroke.
147 ants influencing cholesterol levels, risk of coronary artery disease (CAD), body mass index, as well
148 2+)-activated K(+) channels in subjects with coronary artery disease (CAD), but its mechanisms of act
149 elated with lifespan while susceptibility to coronary artery disease (CAD), cigarettes smoked per day
150                          Among patients with coronary artery disease (CAD), improvement of lifestyle-
151 scribed Chinese medicine for both stroke and coronary artery disease (CAD), its underlying common mol
152 etically higher calcium had a higher risk of coronary artery disease (CAD), myocardial infarction (MI
153 eneity has already been well investigated in coronary artery disease (CAD), the knowledge about monoc
154 status has been related to increased risk of coronary artery disease (CAD), which is a condition that
155 dial infarction (MI) in patients with stable coronary artery disease (CAD).
156 ng studies among patients without history of coronary artery disease (CAD).
157 eating Peripheral Vascular Disease (PVD) and Coronary Artery Disease (CAD).
158 ts, nor if such results indicate obstructive coronary artery disease (CAD).
159 cytokine in inflammatory diseases, including coronary artery disease (CAD).
160 e (MR) perfusion imaging in the detection of coronary artery disease (CAD).
161 -hip ratio (WHR) with mortality and incident coronary artery disease (CAD).This study followed 130,47
162 s displayed lower TC and were protected from coronary artery disease (CAD); (ii) excluding the CETP l
163 olocalize with known susceptibility loci for coronary artery disease (CARDIoGRAMplusC4D) and large ar
164 nome-wide Replication and Meta-analysis plus Coronary Artery Disease (CARDIoGRAMplusC4D) consortium.
165                             In patients with coronary artery disease, clinical outcome depends on the
166                                         In a coronary artery disease cohort separate from volunteers
167 e women having a lower burden of obstructive coronary artery disease compared with men, the prevalenc
168  pain and had a lower pretest probability of coronary artery disease compared with men.
169  outcomes in relation to diabetic status and coronary artery disease complexity as assessed by the Sy
170 cording to drug-eluting stent generation and coronary artery disease complexity were performed.
171 ension, hyperlipidemia, cardiac arrhythmias, coronary artery disease, congestive heart failure, diabe
172 y conditions include venous thromboembolism, coronary artery disease, congestive heart failure, sleep
173  undergoing initial evaluation for suspected coronary artery disease, coronary CTA was associated wit
174 larization decisions in patients with stable coronary artery disease could be improved by assessment
175 re were 6 patients with positive findings of coronary artery disease detected on MDCT coronary angiog
176  transplantation and coronary bypass grafts, coronary artery disease, diabetic cardiomyopathy, hypert
177                                    Prevalent coronary artery disease did not increase the risk to die
178 istory of coronary artery disease, and known coronary artery disease), each gram increase of posterio
179    CT coronary angiography revealed positive coronary artery disease findings in 16 patients; LAD was
180                      Among participants with coronary artery disease, fluctuation in body weight was
181 .G202V HAND2 variant associated with CHD and coronary artery diseases found in a large Lebanese famil
182 uidelines-Stroke and Get With The Guidelines-Coronary Artery Disease from 2006 to 2009 and treating >
183 SS trial, of whom 24 824 patients had stable coronary artery disease from 558 centres.
184            In patients with hypertension and coronary artery disease from routine clinical practice,
185 -wide Replication and Meta-analysis Plus the Coronary Artery Disease Genetics (CardiogramplusC4D) con
186 t disease (CHD) data from CARDIoGRAMplusC4D (Coronary Artery Disease Genome-wide Replication and Meta
187 s (N = up to 61079 individuals) and from the Coronary Artery Disease Genome-wide Replication and Meta
188 sted for their combined effect on CAD in the Coronary Artery Disease Genome-Wide Replication and Meta
189 Consortium and 194,427 participants from the Coronary ARtery DIsease Genome-wide Replication and Meta
190 e 51+/-8.8 years), 80 (6.5%) had obstructive coronary artery disease (&gt;/=70% stenosis) and 68 (5.5%)
191 c arrest in nearly half of survivors in whom coronary artery disease had been excluded.
192                       Eligible patients with coronary artery disease had to have had a myocardial inf
193 th systolic heart failure that is not due to coronary artery disease has been based primarily on subg
194 symptomatic systolic heart failure caused by coronary artery disease has been well documented.
195         In vitro, MSCs from individuals with coronary artery disease have reduced ability to suppress
196 men, despite having less severe angiographic coronary artery disease, have an increased risk of targe
197  prognostic ability of these risk markers in coronary artery disease, heart failure, and atrial fibri
198 with medical therapy alone, in patients with coronary artery disease, heart failure, and severe left
199 were followed through December 31, 2012, for coronary artery disease, heart failure, and stroke.
200 ril 1, 2002, without prior valvular disease, coronary artery disease, heart failure, cardiac arrhythm
201 e, lower systolic blood pressure, history of coronary artery disease, heart failure, chronic obstruct
202 spital size, teaching hospital status, known coronary artery disease, heart failure, diabetes mellitu
203 l dysfunction, hypertension (HTN), diabetes, coronary artery disease, history of atrial arrhythmias,
204 nfarction in the past 20 years, multi-vessel coronary artery disease, history of stable or unstable a
205 Cs) are promising therapeutic strategies for coronary artery disease; however, donor-related variabil
206 for time of enrollment, age, sex, history of coronary artery disease, hypertension, and diabetes.
207 ier modelling in respect of the diagnosis of coronary artery disease in 197 study subjects and the pr
208 uccessful treatment of unstable, multivessel coronary artery disease in a child with PCI with BRS imp
209 ant implications for the future treatment of coronary artery disease in children and warrants further
210 ters in epicardial AT of human patients with coronary artery disease in comparison with controls with
211  Few studies have assessed the prevalence of coronary artery disease in masters athletes with a low a
212 e efforts have been made to directly measure coronary artery disease in this vulnerable population.
213 erved probability of CED with a CREST score (coronary artery disease, initial heart rhythm, low eject
214                                              Coronary artery disease is a major cause of morbidity an
215 ing process of ACS patients with obstructive coronary artery disease is associated with a high reclas
216 iac arrest or periarrest without significant coronary artery disease is crucial for management and pr
217 ment in the delivery of OMT to patients with coronary artery disease is one possible step to help the
218                                              Coronary artery disease is the leading global cause of m
219 ridaforolimus for treatment of patients with coronary artery disease is undetermined.
220 nd cardiovascular disease due to accelerated coronary artery disease is well established.
221  of known history for obstructive epicardial coronary artery disease, is associated with reduced MFR
222 comes in patients with unprotected left main coronary artery disease (LMCAD) treated with coronary ar
223  a better prognosis than MI with obstructive coronary artery disease (MI-CAD).
224 al noninvasive testing to evaluate suspected coronary artery disease might affect subsequent clinical
225 n risk for five vascular diseases, including coronary artery disease, migraine headache, cervical art
226 potential to provide valuable biomarkers for coronary artery disease mirroring especially alterations
227 stable outpatients presenting with suspected coronary artery disease, most patients experiencing clin
228  (PCI) in diabetic patients with multivessel coronary artery disease (MV-CAD).
229 red between men and women with either stable coronary artery disease (n=320) or acute coronary syndro
230 or dyslipidemia; family history of premature coronary artery disease; never smoking; symptoms unrelat
231                 Patients with nonobstructive coronary artery disease (NOCAD; wall irregularities, ste
232                          Among patients with coronary artery disease, nurse-coordinated referral to a
233 icantly less likely than noncarriers to have coronary artery disease (odds ratio, 0.81; 95% confidenc
234 m VTE genetic risk score was associated with coronary artery disease (odds ratio, 1.08; 95% confidenc
235  factors, have the lowest reported levels of coronary artery disease of any population recorded to da
236 signed 1905 eligible patients with left main coronary artery disease of low or intermediate anatomica
237  yield to identify patients with obstructive coronary artery disease on subsequent invasive coronary
238  PCI between 2005 and 2014 because of stable coronary artery disease or acute coronary syndrome were
239 ents undergoing PCI in the setting of stable coronary artery disease or acute coronary syndromes.
240 ngina pectoris OR acute coronary syndrome OR coronary artery disease OR cardiomyopathy.
241 ema AND stroke OR cerebrovascular disease OR coronary artery disease OR heart failure OR myocardial i
242 F or at risk of HF, such as in patients with coronary artery disease or hypertension.
243 lled, outpatient trial, patients with stable coronary artery disease or peripheral artery disease wer
244  ASA sensitivity with known/suspected stable coronary artery disease or presenting with an acute coro
245 th out-of-hospital cardiac arrest have shown coronary artery disease or symptoms during the hour befo
246 g subgroups of patients at increased risk of coronary artery disease or those with a specific driving
247 cular disease (OR, 0.10; 95% CI, 0.01-0.78), coronary artery disease (OR, 0.37; 95% CI, 0.20-0.67), a
248        Subjects without a history of stroke, coronary artery disease, or peripheral artery disease we
249 ean dose; hazard ratio, 1.03/Gy; P = .002,), coronary artery disease ( P < .001), and WHO/Internation
250 ictors of adverse cardiovascular outcomes in coronary artery disease patients.
251 dies suggest that among patients with stable coronary artery disease, patients with diabetes mellitus
252 etes, chronic obstructive pulmonary disease, coronary artery disease, peripheral arterial disease, hy
253 gnificance of smoking in relation to genetic coronary artery disease predisposition may merit further
254 ic role of elevated WBC across a spectrum of coronary artery disease presentations are warranted.
255 ients with diabetes mellitus and multivessel coronary artery disease presenting with non-ST-segment-e
256  55+/-10 years), mostly with an intermediate coronary artery disease probability, between cardiac CT
257 iation of biological therapy with changes in coronary artery disease progression, measured by repeate
258 mparison to Functional Testing for Suspected Coronary Artery Disease) prospectively randomized 350 pa
259 larization) at 1 year in 2,008 patients with coronary artery disease randomized to BVS versus cobalt-
260  hemorrhagic shock in swine with preexisting coronary artery disease reduced renal dysfunction and ca
261 table, symptomatic outpatients without known coronary artery disease referred for noninvasive testing
262 en symptomatic patients without a history of coronary artery disease referred for SPECT and CAC scori
263 ere is angiographic evidence for obstructive coronary artery disease, remains unknown.
264 hletes with atherosclerotic risk factors for coronary artery disease report higher coronary artery ca
265  Although much less frequent than in adults, coronary artery disease requiring revascularization may
266 s familial hypercholesterolemia and unstable coronary artery disease requiring revascularization.
267 e genetic association between rs11556924 and coronary artery disease risk by characterizing its effec
268                                 Conventional coronary artery disease risk factors might potentially e
269 t population of the Bolivian Amazon with few coronary artery disease risk factors, have the lowest re
270 e-industrial lifestyle and low prevalence of coronary artery disease risk factors, we examined the Ts
271 lipoprotein (LDL) has been shown to increase coronary artery disease risk in hemodialysis patients, b
272 similar age, sex, and low Framingham 10-year coronary artery disease risk scores with an echocardiogr
273 en genome-wide significantly associated with coronary artery disease risk.
274 s of inflammation may translate into reduced coronary artery disease risk.
275 clinical outcomes among patients with stable coronary artery disease (SCAD) treated medically.
276 to predict who will develop diseases such as coronary artery disease, stroke, heart failure, and arrh
277 ion (T2MI2007); and 1 method did not require coronary artery disease, the 2012 universal definition (
278 gulating protein] gene increases the risk of coronary artery disease, the leading cause of death worl
279 n scanning showed that CCDC92 likely affects coronary artery disease through insulin resistance pathw
280 ing the total number of loci associated with coronary artery disease to 95 at the time of analysis.
281 reserve with best MT in patients with stable coronary artery disease to assess clinical outcomes and
282 redicted lower BMI, systolic blood pressure, coronary artery disease, type 2 diabetes, and taller sta
283                             In patients with coronary artery disease undergoing elective PCI, an incr
284 65.2+/-7.6 years) with exertional angina and coronary artery disease underwent cardiac catheterizatio
285 le patients (n = 4,057) without a history of coronary artery disease underwent CZT SPECT MPI.
286 a prospective study, 85 patients with stable coronary artery disease underwent percutaneous coronary
287 an initial investigation of suspected stable coronary artery disease using coronary CTA resulted in a
288 k prediction and management of patients with coronary artery disease warrants further study.
289 pt for those 31 to 35 years of age, for whom coronary artery disease was the most common finding.
290 s for diabetic patients who have multivessel coronary artery disease, we combine the results of the F
291 agnosed from 1997 to 2012 without history of coronary artery disease were identified using Danish nat
292  history of statin intolerance, diabetes, or coronary artery disease were not eligible.
293 rmediate pretest probability for obstructive coronary artery disease were randomized to FT (exercise
294 ls (90% on statins) with no prior history of coronary artery disease who had a screening CACS >/=300
295                         Patients with severe coronary artery disease with a clinical indication for r
296 carotid artery stenosis of at least 50%), or coronary artery disease with an ankle-brachial index of
297 nts with stable single-vessel or multivessel coronary artery disease with reduced fractional flow res
298 diagnostic strategy for women with suspected coronary artery disease, with potential benefits in term
299 cantly from those of trials with multivessel coronary artery disease without left main LMCA stenosis.
300 antially reduce morbidity and mortality from coronary artery disease worldwide.

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