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1 ys after coronary angioplasty or directional coronary atherectomy.
2 ion abnormalities associated with rotational coronary atherectomy.
3 independent risk factor for restenosis after coronary atherectomy.
4 tational atherectomy and adjunct directional coronary atherectomy.
5 and to 145 +/- 87 degrees after directional coronary atherectomy.
6 tational atherectomy and adjunct directional coronary atherectomy.
7 rences in the mechanisms by which rotational coronary atherectomy and coronary angioplasty produce th
8 on abnormalities are common after rotational coronary atherectomy and have a longer duration than tho
9 n provided, which includes PTCA, directional coronary atherectomy and high speed rotational atherecto
10 sluminal coronary angioplasty or directional coronary atherectomy and whose peak CK levels did not ex
11 ates from 16 patients undergoing directional coronary atherectomy (DCA) and control samples from the
13 estenosis in patients treated by directional coronary atherectomy (DCA) in the Coronary Angioplasty V
20 ined from 20 patients undergoing directional coronary atherectomy for stable angina were analyzed for
21 consecutive patients undergoing directional coronary atherectomy for symptomatic coronary artery dis
22 mflex artery) retrieved by using directional coronary atherectomy from 25 patients at 0.5 to 23 (mean
24 rimary lesions that develop restenosis after coronary atherectomy have more macrophages and smooth mu
25 tween rotational atherectomy and directional coronary atherectomy in the treatment of calcific lesion
26 domized trials of balloon angioplasty versus coronary atherectomy, laser angioplasty, or cutting ball
28 elevation was the performance of directional coronary atherectomy (odds ratio, 4.1; P < .0001), follo
30 ansluminal coronary angioplasty, directional coronary atherectomy, rotational atherectomy, or excimer
35 from patients with DM were compared with 18 coronary atherectomy specimens with restenosis after PTC
36 with rotational atherectomy and directional coronary atherectomy than in those treated with rotation
37 t wall motion abnormalities after rotational coronary atherectomy, the prevalence and duration of the
42 mens from 90 symptomatic patients undergoing coronary atherectomy were tested for the presence of Chl
43 c plaques in patients undergoing directional coronary atherectomy with a variety of control specimens
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