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1 microvasculature but interrogates the entire coronary circulation.
2  not have to restore efficient blood flow in coronary circulation.
3 rdial and proepicardial contributions to the coronary circulation.
4 er to the different flow dynamics typical of coronary circulation.
5  E14.5, concurrent with the establishment of coronary circulation.
6 n microvessels that are perfused by the host coronary circulation.
7 sociated with functional disturbances of the coronary circulation.
8 transplantation and integrates with the host coronary circulation.
9  in patch clamp recordings and in the intact coronary circulation.
10 vasodilation plays a vital role in the human coronary circulation.
11 d of myocytes and vessels connected with the coronary circulation.
12 and reducing the vasodilatory reserve of the coronary circulation.
13  of the mechanisms action of NE in the human coronary circulation.
14 diated responses are potentiated only in the coronary circulation.
15 ence of CMV in atheromatous plaques from the coronary circulation.
16 may have contributed to the evolution of the coronary circulation.
17 ell known to affect vascular function in the coronary circulation.
18 timates of microvascular shear stress in the coronary circulation.
19 sts that they may be important regulators of coronary circulation.
20  an extremely rare congenital anomaly of the coronary circulation.
21 r acetylcholine in the atherosclerotic human coronary circulation.
22 constrictor properties of ET-1 in the canine coronary circulation.
23 gnificant elevations in NO production by the coronary circulation (46 +/- 23, 129 +/- 44, and 63 +/-
24 n microvessels anastomosed with the rat host coronary circulation and delivered blood to the grafts.
25 the suggestion that inhaled NO may reach the coronary circulation and have a negative inotropic effec
26 ental injection to directly access the fetal coronary circulation and injection at E12.5 when the mas
27 ial dysfunction, and atherothrombosis in the coronary circulation and may explain the greater efficac
28 (a) provide an overview of the physiology of coronary circulation and myocardial perfusion; (b) descr
29 methyl-L-thiocitrulline (SMTC), on the human coronary circulation and on flow-mediated dilatation in
30 al reference range for studies assessing the coronary circulation and post-stenotic coronary vasodila
31 ium, but their relative contributions to the coronary circulation and the molecular mechanisms regula
32 lator effects of testosterone in vivo in the coronary circulation and the potential mechanisms of its
33 diated vasodilation in either the femoral or coronary circulations and did not alter SNP responses in
34 based gene delivery is poorly adapted to the coronary circulation, and current gene transfer vectors
35 1, is involved in vascular reactivity in the coronary circulation, and has a protective role in ische
36 ved CNP is involved in the regulation of the coronary circulation, and NPR-C activation underlies the
37  new vessels were connected with the primary coronary circulation, and this increase in vascularizati
38 elaxation was impaired during hypoxia in the coronary circulation, and this resulted in greater cardi
39 atherosclerotic epicardial and microvascular coronary circulation; and 2) acetylcholine-induced coron
40                                   While some coronary circulation anomalies are associated with signi
41 , although their mechanisms of action on the coronary circulation are less well understood.
42  impairs microvascular dilation in the human coronary circulation as a result of reduced NO bioavaila
43 nctional and/or structural alteration of the coronary circulation associated with coronary risk facto
44                    Basal shear stress in the coronary circulation averaged 10 dyn/cm2 in small arteri
45 ne encoding eNOS were infused into the donor coronary circulation before transplantation.
46 airs vascular function in the peripheral and coronary circulations, but its effects on cerebral arter
47          Estrogen is cardioprotective of the coronary circulation by mechanisms incompletely understo
48 ina, synergistic changes in the systemic and coronary circulations combine to improve vascular-ventri
49                           BQ-123 dilated the coronary circulation; D increased by 5.6+/-1.0% (P<0.000
50                             Isolation of the coronary circulation during cardiac surgery may allow fo
51                     The absence of NO in the coronary circulation during exercise does not affect lev
52                                 Although the coronary circulation during sepsis is maintained or even
53 fter arrest and were allowed to dwell in the coronary circulation during the cross-clamp period.
54 ies (ITAs) are frequently anastomosed to the coronary circulation for bypass grafting.
55                            The effect on the coronary circulation has been the leading explanation fo
56 phrine (NE)-mediated regulation of the human coronary circulation has yet to be elucidated.
57                The effects of cocaine on the coronary circulation have been studied extensively in ex
58 s interventions probably by offering reserve coronary circulation, if in-stent restenosis were to occ
59 scence to assess NO(x) production across the coronary circulation in chronically instrumented dogs du
60  beneficial effects of ACE inhibitors on the coronary circulation in DCM that are not shared by AT1 r
61 rcumflex arteries, suggesting a role for the coronary circulation in determining local drug depositio
62 maging strategy, to define the 3-dimensional coronary circulation in the young and aged heart and its
63 icrovascular diameters and velocities in the coronary circulation in vivo and used these variables to
64 coronary artery stems, and thus a functional coronary circulation, in the embryos injected with VEGF-
65 n on their impact on the ischemic/reperfused coronary circulation is available.
66                     The establishment of the coronary circulation is critical for the development of
67                         NO production by the coronary circulation is increased with exercise and bloc
68 he progression of atherosclerotic plaques in coronary circulation is modulated by several risk factor
69                 However, the acute effect on coronary circulation is not known.
70                   However, its effect on the coronary circulation is not known.
71                     The establishment of the coronary circulation is one of the final critical steps
72     The vasodilatory reserve capacity of the coronary circulation is reduced in AS.
73 oth cohorts), a culprit location in the left coronary circulation (left anterior descending or circum
74          Ten years after implantation to the coronary circulation, LITA grafts show intimal thickenin
75 tion generates a distinct compartment of the coronary circulation located within the inner half of th
76 n of the interventional cardiologist and the coronary circulation, now in 2001 nearly every vascular
77 marrow could be administered safely into the coronary circulation of healthy dogs.
78  but not scrambled control peptide, into the coronary circulation of isolated perfused hearts rapidly
79 may reduce vasodilator responsiveness in the coronary circulation of subjects with coronary disease o
80 rmined the changes in NO production from the coronary circulation of the conscious dog during exercis
81 g of their effects on the myocardium and the coronary circulation, optimization of delivery technique
82 ment of the device without any damage to the coronary circulation or to mitral valve function.
83   In contrast to any other vascular bed, the coronary circulation receives its perfusion mostly durin
84      The effect of testosterone on the human coronary circulation remains unknown.
85 lly digested via retrograde perfusion of the coronary circulation, resulting in rapid and extensive b
86 oglycerin causes changes in the systemic and coronary circulation that combine to reduce myocardial o
87                                       In the coronary circulation, the ADP-ribosylation signaling pat
88  Whether the abnormal responses of the human coronary circulation to acetylcholine in patients with h
89 f preconditioning, has been infused into the coronary circulation to induce therapeutic preconditioni
90                Lp-PLA2 net production in the coronary circulation was higher in patients compared wit
91              Alterations at the level of the coronary circulation with aging may play an important ro

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