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1 esponse to hyperemic stimuli (i.e., abnormal coronary flow reserve).
2 nd attenuated endothelial and nonendothelial coronary flow reserve.
3 helial-dependent and endothelial-independent coronary flow reserve.
4 ardial perfusion reserve matches the reduced coronary flow reserve.
5 cardial layers and vary in relation to local coronary flow reserve.
6 es were previously considered to have normal coronary flow reserve.
7 to quantify resting MBF, hyperemic MBF, and coronary flow reserve.
8 roved pressure-only estimation of underlying coronary flow reserve.
9 flow can also lead to accurate assessment of coronary flow reserve.
10 graphy to quantify myocardial blood flow and coronary flow reserve.
11 including a stress total severity score and coronary flow reserve.
12 her compared with that for hyperemic MBF and coronary flow reserve (0.76; P=0.32 and 0.72; P=0.08, re
13 us 1.66+/-0.38 mL.min(-1).g(-1); P<0.01) and coronary flow reserve (1.59+/-0.49 versus 2.12+/-0.48; P
14 ith abnormal coronary microcirculation (mean coronary flow reserve = 1.84), adenosine increased coron
15 ), %untwMVO (31% versus 27% versus 17%), and coronary flow reserve (14% versus 11% versus 4%), as wel
16 sus 1.78+/-0.43 mL/min per gram; P=0.01) and coronary flow reserve (2.78+/-0.32 versus 2.01+/-0.52; P
17 degrees ), %untwMVO (27.8% versus 35%), and coronary flow reserve (2.8 versus 3.1) and reduced circu
18 control groups (median [interquartile range] coronary flow reserve, 2.9 [2.5-3.4] versus 3.0 [2.4-3.4
19 using speckle-tracking echocardiography, (2) coronary flow reserve, (3) pulse wave velocity and augme
20 -mediated dilation (57+/-4% versus 47+/-5%), coronary flow reserve (37+/-4% versus 29+/-2%), arterial
22 n present as angina pectoris associated with coronary flow reserve abnormalities despite normal coron
24 ought to examine the mechanism of increasing coronary flow reserve after balloon angioplasty and sten
25 positron emission tomography to measure the coronary flow reserve, an integrated measure of coronary
26 icant correlation between MRI assessments of coronary flow reserve and (a) assessments of coronary ar
28 ll conducting vessel densities and increased coronary flow reserve and perfusion-dependent cardiac co
29 duced vasodilatation with exercise = reduced coronary flow reserve and/or vasospasm at rest) might al
31 yocardial perfusion grade, TIMI frame count, coronary flow reserve, and ST-segment resolution were al
32 absolute coronary flow reserve, the relative coronary flow reserve, and the pressure-derived fraction
33 4; 95% confidence intervals [0.69-0.99]) and coronary flow reserve (area under the curve, 0.77; 95% c
37 of an IMR>40, alone or in combination with a coronary flow reserve (CFR</=2.0), in the culprit artery
38 ients with metabolic syndrome showed a lower coronary flow reserve (CFR) (2.5 +/- 1.0) than those wit
40 between major adverse outcomes and baseline coronary flow reserve (CFR) after intracoronary adenosin
41 ediate stenoses were classified in 4 FFR and coronary flow reserve (CFR) agreement groups, using FFR>
42 the hypothesis that increased variability of coronary flow reserve (CFR) among multiple vascular regi
44 rements of fractional flow reserve (FFR) and coronary flow reserve (CFR) and the index of microcircul
49 idated for absolute myocardial perfusion and coronary flow reserve (CFR) by positron emission tomogra
51 isoforms on myocardial blood flow (MBF) and coronary flow reserve (CFR) in volunteers and in (denerv
52 and B had a normal coronary angiogram and a coronary flow reserve (CFR) of > or =2.5 (CFR = hyperemi
54 iastolic/systolic velocity ratio (DSVR), and coronary flow reserve (CFR) were assessed before interve
55 is a new technique for invasively measuring coronary flow reserve (CFR) with a coronary pressure wir
56 d to validate the technique of measuring the coronary flow reserve (CFR) with coronary pressure measu
57 ntitation of myocardial blood flow (MBF) and coronary flow reserve (CFR) with dynamic (82)Rb PET is f
60 en low-level troponin elevation and impaired coronary flow reserve (CFR), an integrated measure of co
63 FR), (2) Doppler wire-derived measurement of coronary flow reserve (CFR), and (3) intravascular ultra
64 his study sought to examine the evolution of coronary flow reserve (CFR), index of microcirculatory r
65 n of maximal myocardial blood flow (MBF) and coronary flow reserve (CFR), termed coronary flow capaci
66 th true microvascular resistance and, unlike coronary flow reserve (CFR), to be independent of the ep
67 relation between habitual dietary sodium and coronary flow reserve (CFR), which is a measure of overa
68 on, as assessed by quantitative estimates of coronary flow reserve (CFR), with respect to prediction
72 11 nondiabetics) underwent quantification of coronary flow reserve (CFR; CFR=stress divided by rest m
73 myocardial blood flow (MBF) and the relative coronary flow reserves (CFR) using (15)O-labeled water (
75 ous coronary intervention, it disagrees with coronary flow reserve classification 30% of the time.
76 is more accurate for assessment of impaired coronary flow reserve compared with SPECT MPI, potential
77 tment kinetic model and were used to compute coronary flow reserve (coronary flow reserve equals stre
79 othesis that an acute critical limitation in coronary flow reserve could rapidly recapitulate the phy
81 In the 5 men who received both meals, mean coronary flow reserve decreased by 0.79 after the high-f
83 sive fractional flow reserve and noninvasive coronary flow reserve, depends on their ability to impro
84 ow-mediated dilation of brachial artery; (2) coronary flow reserve, ejection fraction, systemic arter
85 were used to compute coronary flow reserve (coronary flow reserve equals stress divided by rest myoc
88 the ratio of hyperemic to resting velocity (coronary flow reserve), have been more commonly studied.
90 groups, reflecting the greater diminution in coronary flow reserve in group 2 dogs (LAD/LCx flow rati
92 the microvascular (endothelium-independent) coronary flow reserve in response to intracoronary adeno
93 microcirculatory responsiveness and impaired coronary flow reserve in smokers, which provides evidenc
94 er and thus may be well suited for assessing coronary flow reserve in the acute phase of reperfusion.
96 intended to identify regional limitations in coronary flow reserve in viable myocardium need to ident
98 e ischemia in an area of chronically reduced coronary flow reserve induces regional myocyte loss via
100 ent of maximal saline- and adenosine-induced coronary flow reserve (intraclass correlation coefficien
103 , the regional perfusion reserve matched the coronary flow reserve (linear regression with a slope of
106 aracterized as having impaired post-stenotic coronary flow reserve < 2.0 and pressure-derived fractio
107 Coronary flow reserve was quantified, and coronary flow reserve <2.0 was used to define the presen
111 al change (17.3% versus 17.09%; P=0.91), and coronary flow reserve measurements (2.63 versus 2.53; P=
112 flow (IDV) (mL/min)+17 (mL/min), r=.89, and coronary flow reserve (MRI) =0.79 x coronary velocity re
113 n humans with impaired endothelium-dependent coronary flow reserve of the coronary epicardial and mic
116 ence interval, 0.75-086] per 10% increase in coronary flow reserve; P<0.0001) and resulted in favorab
117 canine myocardial infarction model with some coronary flow reserve preservation, 99mTc-N-NOET imaging
118 s evident when angiography was compared with coronary flow reserve (r=.43), and the angiogram did not
120 yocardial infarction in relation to tests of coronary flow reserve; surveys the extensive literature
124 ude the measurement of poststenotic absolute coronary flow reserve, the relative coronary flow reserv
126 he prognostic value of IMR was compared with coronary flow reserve, TIMI myocardial perfusion grade,
136 high-risk patients with ACS undergoing PCI, coronary flow reserve was greater with bivalirudin than
137 s significantly higher (+95%, p = 0.001) and coronary flow reserve was lower (-0.21, p = 0.02) in twi
142 (n = 754), the primary end point of post-PCI coronary flow reserve was significantly greater with biv
143 this pilot study, impaired hyperemic MBF and coronary flow reserve were associated with VA inducibili
144 sistance (IMR), fractional flow reserve, and coronary flow reserve were measured before stenting in t
147 ation between endothelial and nonendothelial coronary flow reserve with vascular remodeling in patien
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