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1 clerosis, transplant vascular sclerosis, and coronary restenosis.
2 roversy continues regarding the mechanism of coronary restenosis.
3 alphavbeta3 integrin, in the pathogenesis of coronary restenosis.
4 s and is associated with atherosclerosis and coronary restenosis.
5 nts in the prevention and treatment of human coronary restenosis.
6 d has been associated with atherogenesis and coronary restenosis.
7 hat although the use of DES reduces rates of coronary restenosis after percutaneous coronary interven
8 otential for the limitation and treatment of coronary restenosis after percutaneous revascularization
11 erative diseases such as atherosclerosis and coronary restenosis are leading causes of morbidity and
12 sis, myocardial ischemia, coronary stenosis, coronary restenosis, cerebrovascular accident, randomize
13 , short-term study of patients with previous coronary restenosis, coronary stenting followed by cathe
16 herapy techniques to the clinical problem of coronary restenosis has generated tremendous attention a
18 analysis was used to examine the process of coronary restenosis in a large cohort of patients at ris
19 nary gamma radiation plus stenting to reduce coronary restenosis in patients with previous restenosis
20 y with that for other treatments that reduce coronary restenosis, including alternative drug-eluting
24 ficant progress has been made in the area of coronary restenosis, particularly in identifying target
25 .kg-1.d-1 BID for 28 days following PTCA) on coronary restenosis, PTCA was performed in hypercholeste
26 linical benefits observed after treatment of coronary restenosis with (192)Ir appear durable at late
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