ライフサイエンスコーパス: coronary sinus

1 es additional epicardial ablation within the coronary sinus.

2 sion and MR was placed percutaneously in the coronary sinus.

3 ng a novel annuloplasty device placed in the coronary sinus.

4 nd proximal (p = 0.02) and distal (p < 0.01) coronary sinus.

5 teral right atrium, His bundle position, and coronary sinus.

6 sively moved from the distal to the proximal coronary sinus.

7 circumflex (CFX) arteries (CBF(LAD+CFX)) and coronary sinus.

8 complex lead systems including coils in the coronary sinus.

9 His bundle, posterior triangle of Koch, and coronary sinus.

10 the left atrial electrograms recorded in the coronary sinus.

11 dage, at the fossa ovalis, and in the distal coronary sinus.

12 ar diameter, with catheters in the aorta and coronary sinus.

13 od and blood continuously withdrawn from the coronary sinus.

14 om the high right atrium and from the distal coronary sinus.

15 ced into right atrium, pulmonary artery, and coronary sinus.

16 need for epicardial ablation from within the coronary sinus.

17 IIb/IIIa receptor occupancy measured in the coronary sinus.

18 s were taken from the femoral artery and the coronary sinus.

19 (161+/-22 vs 167+/-26 ms, P=0.05) and distal coronary sinus (162+/-20 vs 168+/-22 ms, P=0.01) sites.

20 roduced dose-related increases in aortic and coronary sinus 6-keto-prostaglandin F1 alpha and the tra

21 significantly less ablation from within the coronary sinus (7.0% versus 71.9%; P<0.01).

22 Working through the coronary sinus, a specialized catheter system was easily

23 The distal coronary sinus activation mapping did not permit distinc

24 e right atrial septum and proximal-to-distal coronary sinus activation, 2) craniocaudal activation of

25 perimitral ATs, 13 focal ATs with sequential coronary sinus activation, and 13 other macroreentrant l

26 subeustachian isthmus and proximal-to-distal coronary sinus activation, and 3) caudocranial right atr

27 sed catheter ablation in the left atrium and coronary sinus after pulmonary vein isolation, were enro

28 the region extending from the AVN toward the coronary sinus along the tricuspid valve (posterior noda

29 n of electrograms recorded on the multipolar coronary sinus and ablation catheters was undertaken to

30 Transcardiac (difference between coronary sinus and aorta) NGF levels were also assayed.

31 or function and intimal thickness as well as coronary sinus and aortic cytokine concentrations (tumor

32 locker," thus precluding "steal" through the coronary sinus and forcing retroperfusion of the anterio

33 delivered between decapolar catheters in the coronary sinus and lateral right atrium.

34 nsmitral conduction block using differential coronary sinus and left atrial appendage pacing techniqu

35 ral isthmus linear lesion using differential coronary sinus and left atrial appendage pacing techniqu

36 re placed in the right ventricular apex, and coronary sinus and left ventricular epicardium.

37 een transvenous catheters, one in the distal coronary sinus and one in the right atrial appendage.

38 in seven swine; serial blood sampling of the coronary sinus and peripheral vein before, during, and a

39 dynamic responses, epinephrine levels in the coronary sinus and systemic circulation, and drug deposi

40 Blood samples from the coronary sinus and the femoral vein were collected at th

41 l block was identified between the os of the coronary sinus and the low lateral right atrium for both

42 of FAEE production and ethanol levels in the coronary sinus and the peripheral circulation.

43 Smaller venous structures, such as the coronary sinus and the pulmonary veins, would also be ex

44 ds were placed transvenously into the distal coronary sinus and the right atrial appendage.

45 pping by pacing at adjacent sites within the coronary sinus and the right atrium.

46 undle position, a multipolar catheter in the coronary sinus, and a deflectable quadripolar catheter a

47 s were implanted into the right atrium (RA), coronary sinus, and left pulmonary artery of 14 dogs.

48 le, complex electrograms in the left atrium, coronary sinus, and superior vena cava were targeted for

49 The fossa ovalis, the distal coronary sinus, and the right atrial appendage were outs

50 th paroxysmal AF underwent balloon-occlusion coronary sinus angiograms to identify the vein of Marsha

51 Coronary sinus APDs delivered during HRA pacing also wer

52 Concomitant arterial and coronary sinus (ART/CS) blood samples were collected to

53 utilization of substrates was calculated as coronary sinus-arterial difference times coronary flow.

54 Transmyocardial (coronary sinus-arterial) plasma norepinephrine (tNEPI),

55 were to evaluate the efficacy and safety of coronary sinus aspiration (CSA) procedure to reduce the

56 ial pacing in sinus rhythm from the proximal coronary sinus at a cycle length of 600 ms, until isthmu

57 During constant pacing from the distal coronary sinus, atrial activation close to the His bundl

58 Percutaneous coronary sinus-based mitral annuloplasty reduces chronic

59 FCL at the right atrial appendage and distal coronary sinus before attempting internal cardioversion

60 ary artery was rapid, reaching levels in the coronary sinus blood 4 to 10 times greater than that fou

61 ount to achieve a high drug concentration in coronary sinus blood causes a deterioration of LV systol

62 tes in serial paired peripheral arterial and coronary sinus blood effluents obtained from 37 patients

63 ary sinus blood sampling and measurements of coronary sinus blood flow were made during rest and atri

64 tery occlusion, the content of bradykinin in coronary sinus blood increases.

65 14)C-glucose were coinfused and arterial and coronary sinus blood sampled to measure cardiac free fat

66 Arterial and coronary sinus blood samples (ART/CS) were analyzed for

67 Coronary sinus blood samples were obtained before cardio

68 ter FDG injection, paired basal arterial and coronary sinus blood samples were taken for the measurem

69 cts also underwent simultaneous arterial and coronary sinus blood sampling (to derive transcardiac co

70 Arterial and coronary sinus blood sampling and measurements of corona

71 labeled microspheres and paired arterial and coronary sinus blood sampling, respectively.

72 IVUS and Doppler, combined with arterial and coronary sinus blood sampling.

73 ery high concentrations of FAEEs detected in coronary sinus blood.

74 e ascorbate free radical (AFR) signal in the coronary sinus blood; AFR is a measure of total oxidativ

75 heterogeneous within Koch's triangle and the coronary sinus, both for the entire population and for i

76 tricular lead placement was performed at the coronary sinus branch.

77 Classifying the coronary sinus branches and tributaries by the segment o

78 oplegia and maintained with tepid retrograde coronary sinus cardioplegia for a total of 1 hour.

79 Myocardial oxygen uptake was measured with a coronary sinus catheter.

80 yocardial oxygen uptake was measured using a coronary sinus catheter.

81 recordings, including the use of a decapolar coronary sinus catheter.

82 MBq (5-mCi) bolus of FDG during arterial and coronary sinus catheterization.

83 Coronary venous blood was obtained by coronary sinus catheterization; NOx concentrations in co

84 hian ridge (ER), and from the His bundle and coronary sinus catheters.

85 increased to 100 ng/min, corresponding to a coronary sinus concentration of 175 +/- 45 pg/mL (P < .0

86 ron paramagnetic resonance, we monitored the coronary sinus concentration of ascorbate free radical (

87 Coronary sinus concentrations of unlabeled triglycerides

88 m connection was seen; however, all showed a coronary sinus constriction during atrial systole, indic

89 tly evaluated by measuring the length of the coronary sinus contraction during atrial systole.

90 In the normal group, coronary sinus contraction was seen in 60 of the 65 pati

91 In the atrial fibrillation group, no coronary sinus contraction was seen.

92 e continuity is likely the primary cause for coronary sinus contractions.

93 zed that both the patterns and the timing of coronary sinus (CS) activation could facilitate AT mappi

94 lasma samples were obtained from aorta (AO), coronary sinus (CS) and anterior interventricular vein (

95 vein (PV) ostium and simultaneously from the coronary sinus (CS) and posterior right atrium (RA) duri

96 s to describe the prevalence and ablation of coronary sinus (CS) arrhythmias after left atrial ablati

97 elationships between mitral annulus (MA) and coronary sinus (CS) as well as CS and left circumflex co

98 icular pacemaker insertion have a failure of coronary sinus (CS) cannulation.

99 lock of tissue encompassing the LOM from the coronary sinus (CS) cephalad, between the atrial appenda

100 The coronary sinus (CS) has a myocardial coat (CSMC) with ex

101 treated due to an inability or impediment to coronary sinus (CS) lead implantation.

102 e of this study was to determine whether the coronary sinus (CS) musculature has electrical connectio

103 of atrial tachycardia (AT) arising from the coronary sinus (CS) musculature.

104 atomic mapping during pacing from the distal coronary sinus (CS) or the posterior left atrium.

105 line of conduction block between the IVC and coronary sinus (CS) ostium and forms a second isthmus (s

106 he left atrium is activated, as reflected by coronary sinus (CS) recordings, has not been systematica

107 Arterial and coronary sinus (CS) samples were taken before and after

108 the pacing lead is usually positioned in the coronary sinus (CS) to stimulate the left ventricular (L

109 ation in humans, the release of vWF into the coronary sinus (CS) was measured in 32 patients during o

110 nfiguration, right atrial appendage (RAA) to coronary sinus (CS), was reduced by >50% with the additi

111 (OTVT) from the distal ramifications of the coronary sinus (CS).

112 odes in the right atrial appendage (RAA) and coronary sinus (CS).

113 theters located in the right atrium (RA) and coronary sinus (CS).

114 Ds from the high right atrium (HRA) than the coronary sinus (CS).

115 nefficient cannulation of the orifice of the coronary sinus (CS).

116 catheterization underwent cannulation of the coronary sinus (CS).

117 ther in the superior vena cava (SVC, n = 6), coronary sinus (CS, n = 10) or right pulmonary artery (R

118 y time for left ventricular lead deployment (coronary sinus [CS] cannulation to withdrawal of CS shea

119 in plasma levels between the aortic root and coronary sinus [CS]) in 9 patients undergoing right and

120 her a simpler pacing approach via the distal coronary sinus (CSd) could eliminate AF inducibility by

121 e (RA), left subclavian vein (LSV), proximal coronary sinus (CSos), and distal coronary sinus (DCS) i

122 RAap), distal coronary sinus (DCS), proximal coronary sinus (CSos), main/left pulmonary artery juncti

123 , proximal coronary sinus (CSos), and distal coronary sinus (DCS) in 14 patients with chronic atrial

124 in the right atrial appendage (RAap), distal coronary sinus (DCS), proximal coronary sinus (CSos), ma

125 Two patients had a coronary sinus diverticulum and one a right atrium to ri

126 The area of the coronary sinus during diastole was larger in the atrial

127 ntly increased NO overflow measured from the coronary sinus during SS (93.25+/-59.20 versus 114.82+/-

128 suggested by the elevated drug levels in the coronary sinus effluent.

129 coronary sinus leads, with the most proximal coronary sinus electrode pair straddling the coronary si

130 biventricular systems, with implantation of coronary sinus electrodes, will continue to challenge le

131 tion of far-field left atrium from the local coronary sinus electrograms besides appropriate adjustme

132 vasomotor responses to substance P (SP), and coronary sinus endothelin-1 and NO metabolite levels in

133 46+/-15% below baseline values (P=.007) at a coronary sinus estradiol concentration of 1725+/-705 pmo

134 A strong correlation was found between coronary sinus FAEEs and ethanol concentration (r = 0.92

135 of dietary control, catheters were placed in coronary sinus, femoral arterial and venous, and periphe

136 Coronary sinus flow and arterial-coronary sinus oxygen d

137 LV perfusion was calculated from coronary sinus flow and mass.

138 a resulted in 2.7- and 2.3-fold increases in coronary sinus flow at VEC MR imaging and flow probe CBF

139 CBF(LAD+CFX) and coronary sinus flow at VEC MR imaging showed close corre

140 VEC MR imaging has the potential to measure coronary sinus flow during different physiologic conditi

141 bal LV perfusion was quantified by measuring coronary sinus flow in an oblique imaging plane perpendi

142 The difference between CBF(LAD+CFX) and MR coronary sinus flow was 3.1 mL/min +/- 8.5 (SD).

143 Measurements of coronary sinus flow were performed in seven dogs by usin

144 ated in milliliters per minute per gram from coronary sinus flow, and LV mass was obtained by using V

145 cessory pathway; and 7) searching within the coronary sinus for a presumed accessory pathway potentia

146 Catheters were placed into the coronary sinus for continuous blood withdrawal.

147 valve surgery, a catheter was placed in the coronary sinus for delivery of cardioplegia.

148 ously from the left main coronary artery and coronary sinus for measurement of Lp-PLA2, lysophosphati

149 amples were withdrawn from the aorta and the coronary sinus for measurement of NO metabolites, O2 con

150 r to ablation of CFAEs in the left atrium or coronary sinus for up to 2 additional hours of procedure

151 The NO synthase inhibitors decrease coronary sinus free radical concentration and ameliorate

152 us valves, and formation of the mouth of the coronary sinus from the cranial muscular wall of the lef

153 n that linked the superior vena cava and the coronary sinus from the CT model with a catheter placed

154 from the atrioventricular node artery to the coronary sinus, from the right coronary artery (RCA) to

155 CT can provide excellent information about coronary sinus function and coronary sinus-left atrium m

156 The coronary sinus generally had a different rate than the r

157 , or tended to have, lower concentrations of coronary sinus growth factors and plasma exerting a weak

158 A coronary sinus guidewire traverses a short segment of th

159 positioned in the right atrial appendage and coronary sinus in 13 patients.

160 .1%), inferior vena cava in 1 (1.5%) and the coronary sinus in 2 (3.0%).

161 atrial tachycardia, which was mapped to the coronary sinus in 3 patients, to the posterolateral righ

162 rtery in 64 of 74 (86%) patients, and in the coronary sinus in 49 of 74 (66%) patients.

163 ptum, roof, or anterior wall in all; and the coronary sinus in 55%.

164 t changes in configuration were noted in the coronary sinus in any lead at packing sites < or = 32 mm

165 sinus catheterization; NOx concentrations in coronary sinus, in arterial and peripheral venous plasma

166 on alone, nitric oxide (NO) release into the coronary sinus increased from 219.8 to 544.9 pmol min-1

167 The coronary sinus is the gateway for left ventricular (LV)

168 Left ventricular (LV) pacing through the coronary sinus is the standard approach for cardiac resy

169 was performed using a right atrial appendage/coronary sinus lead configuration in 38 patients with a

170 Ten patients with previous failed coronary sinus lead implant or with nonresponse to cardi

171 tes is more effective than via pre-implanted coronary sinus lead pacing.

172 ocardial site directly transmural to the CRT-coronary sinus lead tip.

173 V; P=0.001) but not different compared with coronary sinus leads (1.33+/-0.58 V; P=0.994).

174 de atrial activation recorded in the lateral coronary sinus leads, and 12 had the earliest retrograde

175 atrial activation recorded in the posterior coronary sinus leads, with the most proximal coronary si

176 In seven patients (11%), no coronary sinus-left atrium connection was seen; however,

177 es in the atrial fibrillation group showed a coronary sinus-left atrium connection, which was single

178 The length, number, and extent of coronary sinus-left atrium connections were recorded.

179 Coronary sinus-left atrium muscle connections were seen

180 nformation about coronary sinus function and coronary sinus-left atrium muscle connections.

181 vels rose rapidly with dramatic increases in coronary sinus levels indicative of myocardial release.

182 endothelial prostacyclin release, aortic and coronary sinus levels of ET-1 and 6-keto-prostaglandin F

183 m the femoral vein (systemic sample) and the coronary sinus (local cardiac sample).

184 acing (DDD)-LV was used to determine optimal coronary sinus LV lead position.

185 l annular dimension with a PMA device in the coronary sinus may reduce functional mitral regurgitatio

186 a quadripolar catheter was performed in the coronary sinus (n = 29) and in the right atrium (n = 10)

187 The coronary sinus narrowed 26% from middiastole to atrial s

188 c activity, whereas metoprolol did not lower coronary sinus norepinephrine and actually increased cen

189 Carvedilol selectively lowered coronary sinus norepinephrine levels, an index of cardia

190 ermined by the Fick method from arterial and coronary sinus O2 concentrations and from MBF obtained b

191 uloplasty can be achieved indirectly via the coronary sinus or directly from retrograde left ventricu

192 uloplasty can be achieved indirectly via the coronary sinus or directly from retrograde left ventricu

193 fficulties with efficient cannulation of the coronary sinus orifice in a rare anatomical variant.

194 crista terminalis, tricuspid valve isthmus, coronary sinus orifice, membranous fossa ovalis and pulm

195 xits (n=3) or only SP exits located near the coronary sinus orifice.

196 ophysiological catheter into the VOM via the coronary sinus orifice.

197 coronary sinus electrode pair straddling the coronary sinus orifice.

198 split (n = 3) or turned in the region of the coronary sinus os (n = 6).

199 vidence of a posterior fast pathway near the coronary sinus os.

200 Coronary sinus ostial lead dislodgement was not observed

201 High right atrial and coronary sinus ostial pacing do not differ in efficacy.

202 High right atrial pacing and coronary sinus ostial pacing had similar efficacy for AF

203 aneously pacing at the high right atrium and coronary sinus ostium at an identical rate to the baseli

204 The lack of increase in the His-to-coronary sinus ostium atrial interval during incremental

205 A <10 ms increase in the His-to-coronary sinus ostium atrial timing during low lateral r

206 atrial pacing from the high right atrium and coronary sinus ostium can suppress inducible AF or atria

207 st retrograde activation was recorded at the coronary sinus ostium in 60% and 65% of patients with ty

208 The incremental His-to-coronary sinus ostium maneuver is analogous to the IP ma

209 with the IP maneuver, the incremental His-to-coronary sinus ostium maneuver was consistent with funct

210 period between the high right atrium and the coronary sinus ostium pacing sites was significantly gre

211 pathway in the right atrial septum near the coronary sinus ostium prevented the induction and clinic

212 t atrial pacing modes (high right atrium and coronary sinus ostium) and the long-term need for cardio

213 owed by ablation of zone 2 (fossa ovalis and coronary sinus ostium).

214 - 2.4 in length within 2.2 mm +/- 3.8 of the coronary sinus ostium, and proximal connections measured

215 m, and left posteroseptal mitral annulus and coronary sinus ostium.

216 t a posterior atrial site (P) just below the coronary sinus ostium.

217 as performed at the high right atrium or the coronary sinus ostium.

218 along the tricuspid annulus anterior to the coronary sinus ostium.

219 0 mm +/- 14.0 within 12.0 mm +/- 11.0 of the coronary sinus ostium.

220 10.0 in length within 24.0 mm +/- 8.0 of the coronary sinus ostium.

221 connections, the connection extended to the coronary sinus ostium.

222 ded for 131I-albumin, 42K and 201Tl from the coronary sinus outflow following injection into arterial

223 use of an in-line flow probe and an in-line coronary sinus oximetric catheterizationeter.

224 sponses (P < .05), associated with increased coronary sinus oxygen content, were observed for-ACh (+6

225 rtic pressure, coronary blood flow, arterial-coronary sinus oxygen difference (DeltaAVO(2)), and MVO(

226 Coronary sinus flow and arterial-coronary sinus oxygen difference were measured to determ

227 equency gradient from the left atrium to the coronary sinus (p = 0.02).

228 tery versus 13 of 49 (26%) patients from the coronary sinus (P<0.001).

229 need for epicardial ablation from within the coronary sinus (P<0.01) and the total length of the MIL

230 rded along the ablation line during proximal coronary sinus pacing at sites at which radiofrequency a

231 findings during high right atrium and distal coronary sinus pacing.

232 minibasket catheter during sinus rhythm and coronary sinus pacing.

233 n myocardial oxygen extraction and decreased coronary sinus pH in baboons, but not dogs.

234 phrine, appearance rate of norepinephrine in coronary sinus plasma (cardiac norepinephrine spillover)

235 levels 100-fold (P<0.01), whereas aortic and coronary sinus plasma Ang I and II levels were unaffecte

236 Coronary sinus plasma endothelin-1 and nitrite/nitrate (

237 and sodium nitroprusside (P<0.001), although coronary sinus plasma tPA antigen and activity concentra

238 of NO (nitrate+nitrite=NO(x)) in aortic and coronary sinus plasma using chemiluminescence to assess

239 dial interstitial fluid (ISF) and aortic and coronary sinus plasma were quantified by use of 3H-label

240 coronary blood flow, epicardial diameter and coronary sinus platelet cGMP content during intracoronar

241 pandable, stainless steel, hourglass-shaped, coronary-sinus reducing device creates a focal narrowing

242 is small clinical trial, implantation of the coronary-sinus reducing device was associated with signi

243 (two of whom died) and by perforation of the coronary sinus requiring pericardiocentesis in two other

244 Pacing within the coronary sinus resulted in significant changes in amplit

245 ction during atrial systole, indicating that coronary sinus-right atrium muscle continuity is likely

246 Coronary sinus-right atrium muscle continuity was indire

247 coronary angiography with high accuracy, and coronary sinus sampling distinguished cardiac-derived fr

248 Arterial-coronary sinus sampling was performed to measure the K o

249 The right ventricular-coronary sinus shock vector had lower impedance than the

250 ances of the RA-left pulmonary artery and RA-coronary sinus shock vectors were similar (121 +/- 11 Om

251 ing was performed from electrodes within the coronary sinus showing activation later than adjacent el

252 a norepinephrine spillover measured from the coronary sinus significantly increased during SS and was

253 Three patients acquired postablation coronary sinus stenosis.

254 dal conduction during right and left atrial (coronary sinus) stimulation in 46 patients (27 women and

255 ncentrations were significantly lower in the coronary sinus than in the artery (P < 0.05; extraction

256 the nonpulmonary vein triggers includes the coronary sinus, the anterior part of the septum, the lef

257 ocal narrowing and increases pressure in the coronary sinus, thus redistributing blood into ischemic

258 Coronary sinus to MA distance increases in patients with

259 regurgitation exploits the proximity of the coronary sinus to the mitral annulus, but is limited by

260 Systemic (femoral vein) but not coronary sinus tumor necrosis factor (TNF)-alpha and C-r

261 various regions of the left atrium, and the coronary sinus until AF terminated or all identified com

262 Blood samples were collected from the coronary sinus via a cannula passed through the right ex

263 nd ablation catheters were inserted into the coronary sinus via femoral sheaths and into the right at

264 e CT model with a catheter placed inside the coronary sinus via the superior vena cava.

265 oventricular node artery passed close to the coronary sinus wall (mean distance, 2.1 mm +/- 0.7; rang

266 rug concentration in blood obtained from the coronary sinus was 3.0+/-0.4 (mean+/-SD) mg/L, similar i

267 In 10 of the 65 patients (15%), the entire coronary sinus was attached to the left atrial wall.

268 In CSA group, the coronary sinus was cannulated via subclavian or femoral

269 hat initiated flutter, when right atrium and coronary sinus were activated in sequence.

270 Similarly, the superior vena cava and the coronary sinus were also reconstructed from these images

271 V1 and electrograms from the left atrium and coronary sinus were analyzed to determine the DF of AF b

272 All available tributary veins of the coronary sinus were tested, and the Q-LV interval was me

273 nnulus and a decapole catheter placed in the coronary sinus were used for mapping during initiation t

274 coronary arteries arising from the opposite coronary sinus with IAC deserves further study.

275 n oblique imaging plane perpendicular to the coronary sinus with non-breath-hold VEC MR imaging.

276 sistent elevation of NGF levels in aorta and coronary sinus within 1 month after MI.