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1 diameter, volumetric coronary blood flow, or coronary vascular resistance.
2 CAB-2 also retarded the onset of increased coronary vascular resistance.
3 brillation and the accompanying increases in coronary vascular resistance.
4 t percent changes in coronary blood flow and coronary vascular resistance (-31 +/- 4% versus -7 +/- 3
6 ects on energy metabolism without effects on coronary vascular resistance and coronary blood flow.
7 fic antagonist of nitric oxide synthesis, on coronary vascular resistance and epicardial coronary art
8 MA, the 13 +/- 4% (mean +/- SEM) increase in coronary vascular resistance and the 4 +/- 1% lumen diam
9 h a higher coronary effluent PCO2, increased coronary vascular resistance, and development of ischemi
10 elopment of ischemic contracture, increasing coronary vascular resistance, and favoring the developme
11 length area, coronary artery blood flow and coronary vascular resistance, and myocardial oxygen cons
12 [*p < 0.05 vs. baseline value]) and reduced coronary vascular resistance (baseline, 1.6 +/- 0.3 mm H
13 -8%, P<.05, respectively) and an increase in coronary vascular resistance (CVR) (242+/-18% versus 110
14 (13)N]ammonia PET imaging to measure MBF and coronary vascular resistance (CVR) at rest and during a
15 and Doppler flow velocity were measured, and coronary vascular resistance (CVR) was calculated during
17 mulation was performed, measuring changes in coronary vascular resistance (% delta CVR) before and af
20 Hg/s; P < .01) and coronary vasoconstrictor (coronary vascular resistance: first dose, +38 +/- 9% fro
21 decreased coronary blood flow and increased coronary vascular resistance in normal dogs; this effect
22 unction, determined as the percent change in coronary vascular resistance in response to ACH (P=0.009
23 e percent changes in coronary blood flow and coronary vascular resistance in the TIVCC compared with
26 ompromised by other mechanisms that increase coronary vascular resistance or reduce coronary driving
27 o significant effect on coronary blood flow, coronary vascular resistance, or coronary artery diamete
29 (+15 +/- 4 from 41 +/- 4 ml/min), while the coronary vascular resistance response was greater in bab
30 stemic pressor, inotropic, chronotropic, and coronary vascular resistance responses to cocaine (1 mg/
31 ry artery diameter, coronary blood flow, and coronary vascular resistance were calculated on the basi
32 sclerotic patients (32.2 +/- 9% reduction in coronary vascular resistance with 10(-6) mol/liter acety
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