ライフサイエンスコーパス: coronary vasoconstriction

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1 , in CAD patients), acute stress also causes coronary vasoconstriction.

2 vehicle (DMSO) had no effect on ET-1-induced coronary vasoconstriction.

3 and/or alpha1B-AR signaling without causing coronary vasoconstriction.

4 Cocaine caused greater coronary vasoconstriction and greater requirements for o

5 increase in myocardial injury as a result of coronary vasoconstriction and heightened oxidative stres

6 ediated damage, epicardial and microvascular coronary vasoconstriction and/or spasm, and increased ca

7 of arterial stretch receptors with resultant coronary vasoconstriction, and increased vessel-wall inj

8 Acetylcholine elicited coronary vasoconstriction in 8 of 10 subjects.

9 Coronary vasoconstriction in angiographically diseased a

10 n precontracted rat aortic rings, attenuated coronary vasoconstriction in hearts ex vivo, and signifi

11 ynamic determinants of mental stress-induced coronary vasoconstriction in patients undergoing diagnos

12 asodilating and vasoconstricting factors and coronary vasoconstriction in response to the endothelium

13 nd alpha1D, and the alpha1D subtype mediates coronary vasoconstriction in the mouse.

14 In a pharmacological model of coronary vasoconstriction, inhaled NO did not reverse th

15 reoxygenation (16% O(2)) resulted in further coronary vasoconstriction (internal diameter from 105 +/

16 Recent work in humans suggests coronary vasoconstriction occurs with static handgrip wi

17 s for acute migraine, but the side-effect of coronary vasoconstriction restricts their use in patient

18 Thrombin deposition causes local coronary vasoconstriction that may contribute to the dev

19 Estrogen reverses acetylcholine-induced coronary vasoconstriction via the possible facilitation

20 The coronary vasoconstriction was caused by multiple factors

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