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1 t pain persisted despite administration of a coronary vasodilator.
2 ore and after the venous administration of a coronary vasodilator.
3 ata have suggested that it can function as a coronary vasodilator.
4 support a role for exogenous CNP as a potent coronary vasodilator.
5                        Adenosine is a potent coronary vasodilator and causes an increase of coronary
6 reperfusion, and the A2 effects include both coronary vasodilator and leukocyte inhibitory effects.
7 hin the infarct bed, MCE in conjunction with coronary vasodilators can accurately predict infarct siz
8 reserve (CFR), which is a measure of overall coronary vasodilator capacity and microvascular function
9 d platelet aggregation, and are a target for coronary vasodilator drugs.
10                                              Coronary vasodilator dysfunction is a powerful, independ
11 nary conductance and resistance arteries the coronary vasodilator effects of the angiotensin-converti
12                            Nesiritide exerts coronary vasodilator effects on both the coronary conduc
13                           The improvement in coronary vasodilator function from the time of acute PTC
14 is unknown whether noninvasive assessment of coronary vasodilator function in patients with suspected
15 milar endothelium-dependent and -independent coronary vasodilator function in subjects with both type
16       Noninvasive quantitative assessment of coronary vasodilator function with positron emission tom
17 tabolic syndrome is associated with impaired coronary vasodilator function, a marker of atherosclerot
18            Statins, antiplatelet agents, and coronary vasodilators protect against microembolization
19 quantify changes in transmural perfusion and coronary vasodilator reserve (CVR), a measure of microci
20 ory dysfunction, demonstrated by an impaired coronary vasodilator reserve (CVR).
21 imed to assess the effect of TMLR on MBF and coronary vasodilator reserve (CVR).
22 ely provide a noninvasive method for testing coronary vasodilator reserve and may emerge as a new met
23  The calculated "noninvasive" and "invasive" coronary vasodilator reserve ratios were similar ([mean
24 to examine the timing and degree of regional coronary vasodilator reserve recovery in patients who ar
25                                "Noninvasive" coronary vasodilator reserve was determined by measuring
26 s that correction of hyperlipidemia improves coronary vasodilator response and maximal perfusion in m
27                                          The coronary vasodilator response to nitroglycerin is not si
28 ical concentrations on endothelium-dependent coronary vasodilator responsiveness in postmenopausal wo
29                           CNP functions as a coronary vasodilator through activation of cGMP by way o
30  receptor agonist WRC-0470 is a short-acting coronary vasodilator with potential utility for pharmaco

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