ライフサイエンスコーパス: corpora lutea

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1 egenerating antral follicles, and absence of corpora lutea.

2 xhibited a marked reduction in the number of corpora lutea.

3 produced fewer oocytes and also showed fewer corpora lutea.

4 ntation-promoting activity has been found in corpora lutea.

5 e, whereas they undergo differentiation into corpora lutea.

6 ary to VEGF expression in both follicles and corpora lutea.

7 tions of follicles, they ovulate and develop corpora lutea.

8 wing preovulatory follicles as well as early corpora lutea.

9 was weak in atretic follicles and regressing corpora lutea.

10 o ovulate and develop numerous inappropriate corpora lutea.

11 educed numbers of large antral follicles and corpora lutea.

12 rrested folliculogenesis and failure to form corpora lutea.

13 and atrophic reproductive tracts with absent corpora lutea.

14 ndations were adherent in most instances for corpora lutea (88%), simple cysts (56%), and cysts sugge

15 C/EBP beta-deficient ovaries lack corpora lutea and fail to down-regulate expression of th

16 le infertility with abnormal ovaries lacking corpora lutea and increase in luteinizing hormone levels

17 ing washed mitochondria isolated from bovine corpora lutea and purified recombinant His-tag StAR prot

18 ed imaging of the ovary revealed cyst walls, corpora lutea, and ovarian medulla.

19 zyme 20alpha-hydroxysteroid dehydrogenase in corpora lutea (CL) inactivated of Lgr5.

20 f ischemic necrosis were demonstrated in the corpora lutea (CLs) of treated animals.

21 in wild type recipients, significantly more corpora lutea containing un-ovulated oocytes were presen

22 nsplants displayed normal estrous cycles and corpora lutea, despite DHT treatment, implying extraovar

23 her percentage of antral follicles and fewer corpora lutea; follicles progressed to the antral stage

24 aphase II oocytes can be retrieved from, and corpora lutea form in, ovaries of aged Bax-/- females fo

25 ning, we identified individual follicles and corpora lutea in intact ovaries.

26 contrast, progesterone, which is secreted by corpora lutea, increased SLA and beta(2)m in luminal epi

27 n mice on lower P:C (1:8), and the number of corpora lutea, indicative of recent ovulations, was grea

28 increase in NUR77 mRNA was observed in mice corpora lutea just before parturition at a time when 20a

29 In normally developing corpora lutea, macrophages were intimately juxtaposed wi

30 ncreased fertility based on ovulated egg and corpora lutea numbers.

31 Analysis of PKC delta activity in corpora lutea obtained during pregnancy by both the IC k

32 sessed the activation status of PKC delta in corpora lutea obtained when the corpus luteum is exposed

33 ithelial and granulosa cells and also in the corpora lutea of GREKO(-/-) mice.

34 munohistochemical analyses demonstrated that corpora lutea of p27(Kip1), p21(Cip1) double-null mice s

35 ibit defects in luteinization, with numerous corpora lutea, some of which contain central trapped, fu

36 Similar to corpora lutea, the lesions were highly vascularized, alt

37 support development and maintain functional corpora lutea through the production of estrogen.

38 ssion was evaluated in ovarian follicles and corpora lutea utilizing immunohistochemical techniques a

39 70, inhibition of endometrial maturation and corpora lutea was observed.

40 o found to be severely subfertile, and fewer corpora lutea were found to form in response to exogenou

41 Corpora lutea were present in all groups.

42 No corpora lutea were present.

43 In the ovaries of macrophage-depleted mice, corpora lutea were profoundly abnormal, with elevated Pt

44 turation of secondary ovarian follicles into corpora lutea, which express high levels of p27, was mar

45 of follicle maturation, most notably around corpora lutea, without significantly affecting follicula

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