ライフサイエンスコーパス: corpus

1 he appearance of SLFN4+ MDSCs in the gastric corpus.

2 basal bodies, destroyed in the mid-to-lower corpus.

3 nd 85.98% on the independently annotated CLL corpus.

4 rpus Gellus and CLL, a focused target domain corpus.

5 , heterogeneous mass located in the anterior corpus.

6 noids from surgical samples of human gastric corpus.

7 fy the contextual properties in the clinical corpus.

8 n annotated in earlier versions of the CRAFT corpus.

9 tations to the previous release of the CRAFT corpus.

10 frequency of its use in the American English corpus.

11 ternatives on both a biological and clinical corpus.

12 unoreactivity significantly decreased in the corpus (1.27 +/- 0.20 vs 0.46 +/- 0.15, P = 0.02), while

13 nd CI + HA conditions, using the Sung Speech Corpus, a database of monosyllabic words produced at dif

14 The resulting corpus adds nearly 30,000 annotations to the previous re

15 Simple content analysis of this corpus allowed us to detect specific events, like wars,

16 h the concept annotations on a gold standard corpus and also by document retrieval on a collection of

17 nd fundus or corpus or all of these, corpus, corpus and antrum, and antrum.

18 r metaplasia of the lower esophagus, gastric corpus and antrum.

19 Empirical evidence from dialogue, both corpus and experimental, highlights the importance of in

20 itional Random Fields, trained on the Gellus corpus and supported with a dictionary of cell line name

21 in 87% of cases, which essentially presented corpus and/or cardia-type mucosa.

22 nor effects of target predictability, target corpus, and target/masker similarity were also found.

23 gical examination of the pancreatic head and corpus at day 7 revealed less edema for the study group

24 in the empirical performance and pitfalls of corpus-based quality score compression schemes in genera

25 iomedical domain knowledge from a large text corpus built from Medline and embed it into word feature

26 obvious differences in the morphology of the corpus bursa and the heavy muscular area of the ductus e

27 ings are differentiated from the rest of the corpus by subtle but important stylistic features.

28 aracteristic extracortical features, such as corpus callosal, basal ganglia, and cerebellar abnormali

29 The analyzed corpus callosi had distinct proteomic profiles depending

30 iling was performed on early postnatal mouse corpus callosi, for which limited evidence has been obta

31 dentify differences in the effect of virtual corpus callosotomies and the neurodevelopmental disorder

32 echanical hypersensitivity of hindlimbs, but corpus callosotomy eliminated the analgesic effect that

33 as achieved with FA index measurement of the corpus callosum (51%).

34 ic individuals with isolated agenesis of the corpus callosum (ACC) without intellectual disability.

35 tion in 25 participants with agenesis of the corpus callosum (AgCC) and 21 matched neurotypical indiv

36 c dominance in patients with agenesis of the corpus callosum (AgCC) and found reduced laterality (i.e

37 e investigated the effect of agenesis of the corpus callosum (AgCC), one of the most common brain mal

38 re present in frontal and temporal lobes and corpus callosum (all p values <0.01).

39 Lesions in the corpus callosum (CC) are important radiological clues to

40 The corpus callosum (cc) contains nitric oxide (NO)-producin

41 anual responses to these unseen stimuli, the corpus callosum (CC) dynamically recruited areas in the

42 While microstructural alterations in corpus callosum (CC) have been identified as a consisten

43 The corpus callosum (CC) is one of the most commonly reporte

44 The corpus callosum (CC) is the largest fiber tract in the m

45 1 controls the formation of the layer II/III corpus callosum (CC) projections through the development

46 m have been often reported to have a smaller corpus callosum (CC) than control subjects.

47 the widths of the frontal horn (FH) and the corpus callosum (CC) were not significantly different be

48 vity (MD) and radial diffusivity (RD) in the corpus callosum (CC), superior longitudinal fasciculus (

49 connect the two cerebral hemispheres via the corpus callosum (CC).

50 hite matter (WM) damage, particularly to the corpus callosum (CC).

51 The anterior corona radiata (d=0.40) and corpus callosum (d=0.39), specifically its body (d=0.39)

52 microstructural integrity of the body of the corpus callosum (FA, beta = 0.01 [P = .01]; RD, beta = -

53 First, whereas FA in anterior corpus callosum (genu) correlated with word-matching BPA

54 iculi (IFOF), genu (GCC) and splenium of the corpus callosum (SCC), posterior limbs of the internal c

55 l neuropathy with or without agenesis of the corpus callosum (SLC12A6).

56 in a priori regions of interest: splenium of corpus callosum (SPCC) and posterior limb of internal ca

57 ated with word-matching BPA, FA in posterior corpus callosum (splenium-occipital) correlated with fac

58 Astrogliosis significantly increased in corpus callosum (TBI = 6.7 +/- 0.69, Sham = 2.5 +/- 0.38

59 onal connectivity in individuals in whom the corpus callosum (the major commissure between the hemisp

60 erc1), thinner (Kif21b and Wdr89), or absent corpus callosum (Wdr47), revealing a common role for WDR

61 ieved with the volumetric measurement of the corpus callosum - the values were 73% and 71%, respectiv

62 lities included intracranial calcifications, corpus callosum abnormalities, abnormal cortical formati

63 bility in the structural organization of the corpus callosum accounts for these differences.

64 precursor protein aggregates in axons of the corpus callosum after traumatic brain injury as compared

65 isorders, such as autism spectrum disorders, corpus callosum agenesis, Joubert syndrome, Kallmann syn

66 ctrum disorder, intellectual disability, and corpus callosum agenesis.

67 and all cases exhibited partial or complete corpus callosum agenesis.

68 However, it is unclear whether corpus callosum alterations are related to the underlyin

69 diffusion anisotropy of the splenium of the corpus callosum and adjacent parietal white matter (P <

70 osomal recessive inheritance pattern of thin corpus callosum and axonal Charcot-Marie-Tooth disease i

71 sive hereditary spastic paraplegia with thin corpus callosum and axonal peripheral neuropathy (SPG7/P

72 RD, and MD in more extended portions of the corpus callosum and beyond (eg, corona radiata and infer

73 nction leads to insufficient angiogenesis in corpus callosum and catastrophic axon loss.

74 ng, white matter injury was prominent in the corpus callosum and internal capsule on day 3 and then p

75 We examined the corpus callosum and its subregion volumes and their rela

76 a reduced number of myelinated axons in the corpus callosum and optic nerves.

77 cific damage to the genu and splenium of the corpus callosum and parahippocampal tract bilaterally (P

78 sive hereditary spastic paraplegia with thin corpus callosum and peripheral axonal neuropathy, and ac

79 es and attenuated white matter growth of the corpus callosum and pons relative to nondrinkers.

80 ad lower mean diffusivity in the genu of the corpus callosum and the anterior thalamic tracts.

81 of proteins regulating the formation of the corpus callosum and their respective developmental funct

82 small normally proportioned cerebellum, and corpus callosum anomalies.

83 ntry of new astrocytes from the SVZ into the corpus callosum appears to be balanced by astroglial apo

84 Brain malformations involving the corpus callosum are common in children with developmenta

85 We found significantly increased corpus callosum area and thickness in children with auti

86 enetic fate-mapping, we demonstrate that new corpus callosum astrocytes are continuously generated fr

87 These nestin fate-mapped corpus callosum astrocytes are uniformly postmitotic, ex

88 oglial apoptosis, because overall numbers of corpus callosum astrocytes remain constant during normal

89 ces were particularly robust in the anterior corpus callosum at the 6 and 12 month time points.

90 Corpus callosum bisections demonstrated that premotor co

91 ignificant decrease in FA in the genu of the corpus callosum characterized both conditions.

92 also evident in the anterior portions of the corpus callosum connecting left and right frontal lobes.

93 the increased white matter integrity in the corpus callosum connecting these regions, suggesting an

94 hypothesis that congenital disruption of the corpus callosum constitutes a major risk factor for deve

95 Remarkably, all seven genes showed corpus callosum defects, including thicker (Atg16l1, Cor

96 ial-temporal lobe epilepsy, microcephaly and corpus callosum deficiency, and by postnatal Day 21, mic

97 e crossing of an axonal subpopulation of the corpus callosum derived from the anterior cingulate cort

98 The role that corpus callosum development has on the hemispheric speci

99 rce for identifying new proteins integral to corpus callosum development that will provide new insigh

100 previously-identified proteins in aspects of corpus callosum development, and identifies new candidat

101 r evidence that the presence of any residual corpus callosum differentiated those who exhibited curre

102 ffecting directional axonal growth, triggers corpus callosum enlargement due to the errant CB1 cannab

103 losum is a congenital condition in which the corpus callosum fails to develop; such individuals exhib

104 bilateral atrophy of the dorsal cingulum and corpus callosum fibers, which we interpret as a conseque

105 interhemispheric connectivity by controlling corpus callosum formation remains unclear.

106 In slices of corpus callosum from mice subjected to a demyelination p

107 findings suggest novel dual mechanism of the corpus callosum function in spatial attention and have b

108 The corpus callosum has been implicated in the pathogenesis

109 he module (dorsomedial) lying closest to the corpus callosum has the most complete set of commissural

110 These findings suggest that the corpus callosum helps to drive language lateralization.

111 aging showed subtle abnormalities, including corpus callosum hypoplasia and ventriculomegaly.

112 onia, movement disorders, behavior problems, corpus callosum hypoplasia, and epilepsy.

113 fections, respectively; abnormalities of the corpus callosum in 16 of 17 (94%) and 22 of 28 (78%) inf

114 ity before and after surgical section of the corpus callosum in 22 patients with medically refractory

115 but inattention scores were related to AD in corpus callosum in a cluster sized 716 voxels.

116 hese findings suggest a notable role for the corpus callosum in maintaining stable functional communi

117 allosal structure, supporting a role for the corpus callosum in mediating functional asymmetry.

118 against brain injury in the hippocampus and corpus callosum in rats with vascular dementia.

119 abnormalities with sparing of the U fibers, corpus callosum involvement with sparing of the outer bl

120 Agenesis of the corpus callosum is a common brain malformation that can

121 Agenesis of the corpus callosum is a congenital condition in which the c

122 rous brain imaging studies indicate that the corpus callosum is smaller in older children and adults

123 The corpus callosum is the largest fibre tract in the brain,

124 Section of the corpus callosum markedly reduced interhemispheric functi

125 en and adults, our findings suggest that the corpus callosum may be larger in infants who go on to de

126 Together, our findings suggest that the corpus callosum may have a dual inhibitory and excitator

127 ctroscopy data from the anterior body of the corpus callosum of 13 patients with systemic lupus eryth

128 High focal uptake was found in the posterior corpus callosum of a TBI subject.

129 G-ratio of myelin, were also detected in the corpus callosum of adult HdhQ250 mice.

130 he remyelination phase of the CPZ model, the corpus callosum of Cav1.2(KO) animals presented a signif

131 rpus callosum, cortex, and striatum, and the corpus callosum of Cav1.2(KO) animals showed an importan

132 he dentate nucleus, subthalamic nucleus, and corpus callosum of multiple system atrophy, and in all r

133 drocyte differentiation were impaired in the corpus callosum of Olig1-null mice, resulting in hypomye

134 neuropathy, cognitive impairment and a thin corpus callosum on brain MRI.

135 ons surrounding the anterior crossing of the corpus callosum on E18 as well as the persistence of lar

136 c functional connectivity in relation to the corpus callosum presents a case in point.

137 Evoked action potentials in the myelinated corpus callosum projections of Msh2-null mice were small

138 The volumetric assessment of the corpus callosum proved to be a useful tool in discrimina

139 es in white matter tracts of the genu of the corpus callosum that connect the two hemispheres of the

140 r microstructure (FA) within a region of the corpus callosum that projects to the SMA within each hem

141 Reduced corpus callosum thickness confirmed trend-level observat

142 nal anisotropy (FA) index measurement of the corpus callosum using diffusion tensor imaging.

143 pus, on average, but lower right caudate and corpus callosum volume, relative to 22q-del carriers.

144 tional anisotropy within the splenium of the corpus callosum was found in each NDD group, compared wi

145 silesional corticospinal tract and bilateral corpus callosum was increased but sensorimotor CBF was d

146 Interestingly, the corpus callosum was markedly thinner, a characteristic w

147 Absence of the corpus callosum was noted at screening prenatal head ult

148 Luxol fast blue staining of the corpus callosum was significantly greater in the BCCAO r

149 necting ipsilateral cortical regions and the corpus callosum were significantly heritable, ranging fr

150 Schizencephaly and abnormalities of the corpus callosum were the most often developmental disord

151 Dissociations were found in the genu of corpus callosum which accounted for short-term memory bi

152 In WT mice, microglia expanded in the corpus callosum with age, whereas aged Trem2(-/-) mice h

153 ite matter with sparing of the U fibers, the corpus callosum with sparing of the outer blades, the ba

154 e., fractional anisotropy alterations in the corpus callosum) as a shared feature of ASD, ADHD, and O

155 nd white matter of cerebrum, cerebellum, and corpus callosum).

156 Except for the genu of the corpus callosum, an increase in AD values was also found

157 ions in the hippocampus, degeneration of the corpus callosum, and ataxia and seizures.

158 /- but not Nova1-/- mice had agenesis of the corpus callosum, and axonal outgrowth defects specific t

159 tly present with spastic paraparesis, a thin corpus callosum, and cognitive impairment.

160 plegia, developmental delay, agenesis of the corpus callosum, and enlargement of the third cerebral v

161 late gyrus, cortex of the temporal lobes and corpus callosum, and fractional anisotropy (FA) index me

162 encephalic commissures (anterior commissure, corpus callosum, and hippocampal commissure) along with

163 Small telencephalic commissures (anterior, corpus callosum, and hippocampal), an enlarged posterior

164 nifest stage around the striatum, within the corpus callosum, and in posterior white matter tracts.

165 ficantly higher in the medial cortex, in the corpus callosum, and in the thalamus than in the corresp

166 interconnect auditory and motor structures, corpus callosum, and in tracts interconnecting cortical

167 otonia, developmental delay, thinning of the corpus callosum, and seizures.

168 rain malformation (microcephaly, agenesis of corpus callosum, and simplified gyration), and severe en

169 ith the nodes of Ranvier in the optic nerve, corpus callosum, and spinal cord of young adult mice or

170 regions included genu, body and splenium of corpus callosum, anterior and superior corona radiata, s

171 neurons in the neocortex results in lack of corpus callosum, anterior commissure, and corticospinal

172 aining and MPF in all anatomical structures (corpus callosum, anterior commissure, internal capsule,

173 uding hippocampus hypoplasia and agenesis of corpus callosum, as well as neuromuscular and behavioral

174 )/stria terminalis, genu and splenium of the corpus callosum, bilateral anterior and posterior limbs

175 , as did white matter areas (splenium of the corpus callosum, bilateral superior-parietal lobe, bilat

176 entilation, brain atrophy, dysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facia

177 decreased volume and abnormal signal), thin corpus callosum, cerebellar vermis hypoplasia, optic ner

178 features of the brain, such as the amygdala, corpus callosum, cerebellum, and gyrnecephalic index, al

179 Apart from alterations in white matter (in corpus callosum, cingulum bundle, corona radiata, and su

180 tensor imaging abnormalities observed in the corpus callosum, cingulum, and temporal lobe likely cons

181 l and macrostructural variability within the corpus callosum, consistent with differential effects on

182 e in several brain structures, including the corpus callosum, cortex, and striatum, and the corpus ca

183 d the psALS group, encompassing parts of the corpus callosum, corticospinal tracts and superior longi

184 econdary hypomyelination, microcephaly, thin corpus callosum, developmental delay, intellectual disab

185 n of WM damage that involved the body of the corpus callosum, fornix, and main anterior-posterior pat

186 PN) connect the cerebral hemispheres via the corpus callosum, integrating cortical information and pl

187 g abnormalities of the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, basal foreb

188 Fourth, white matter such as corpus callosum, known to contain negligible numbers of

189 NFIX) results in abnormal development of the corpus callosum, lateral ventricles, and hippocampus.

190 not in FA values, including the splenium of corpus callosum, left posterior corona radiate/posterior

191 on, relevant to limited brain areas like the corpus callosum, or multiple orientations but without th

192 arietal regions, and lower FA in the body of corpus callosum, posterior superior longitudinal fascicu

193 d in some subcortical regions, including the corpus callosum, putamen, and cerebellum.

194 volume and microstructural integrity of the corpus callosum, represented the most promising candidat

195 d by 67.4% and 203.0% in the hippocampus and corpus callosum, respectively.

196 corona radiata, right tapetum, and bilateral corpus callosum, statistically moderates whether sleep s

197 esent in the subcortical gray matter nuclei, corpus callosum, superior temporal gyrus, and pre- and p

198 ismatch repair deficiency is agenesis of the corpus callosum, the cause of which has not been establi

199 Why do humans born without the corpus callosum, the major interhemispheric commissure,

200 e fewer axons than in wild-type mice and, in corpus callosum, the myelin is thinner than in controls.

201 capsule (RLIC), the body and splenium of the corpus callosum, the superior and posterior corona radia

202 distribution volume (VT), determined in the corpus callosum, we calculated the binding potential (re

203 ected), mainly in the uncinate, cingulum and corpus callosum, whereas responders were indistinguishab

204 udate nucleus, and cerebellum but not in the corpus callosum, which served as reference region for no

205 particularly significant with regard to the corpus callosum, whose development undergoes several dyn

206 ight frontal cortex to 0.46 mL cm(-3) in the corpus callosum, with intermediate VT values in subcorti

207 The corpus callosum, with its approximately 200 million axon

208 ased in the superior cerebellar peduncle and corpus callosum.

209 ite matter organization, particularly in the corpus callosum.

210 myelination of the axonal projections in the corpus callosum.

211 ct orientation within white matter, e.g. the corpus callosum.

212 cross-hemispheric communication through the corpus callosum.

213 cerebellar vermis and lobules V and VI, and corpus callosum.

214 ith cuprizone, and OPCs were sorted from the corpus callosum.

215 ls of PEDF was capable of infiltration along corpus callosum.

216 f genes related to myelination and increased corpus callosum.

217 trics (FA, MD, RD, and AD), primarily in the corpus callosum.

218 rrhages predominantly in the splenium of the corpus callosum.

219 m aberrant axonal bundles within the rostral corpus callosum.

220 decreased numbers of myelinated axons in the corpus callosum.

221 tal retardation (MR) and malformation of the corpus callosum.

222 he interhemispheric connecting fibers of the corpus callosum.

223 evere mental retardation and agenesis of the corpus callosum.

224 asciculus, as well as in the splenium of the corpus callosum.

225 ossibly associated with the evolution of the corpus callosum.

226 wing demyelinating injury to the adult mouse corpus callosum.

227 which appeared progressive, and a prominent corpus callosum.

228 ivo pathology of the sensorimotor cortex and corpus callosum.

229 orks as well as in the frontal aspect of the corpus callosum.

230 inflammatory microglia only (p=0.01) in the corpus callosum.

231 Brain MRI of the patient showed a thin corpus callosum.

232 mispheric commissural bridges traversing the corpus callosum.

233 tructural connectivity in anterior/posterior corpus callosum.

234 as frontal white matter and the genu of the corpus callosum.

235 also failed to cross the midline to form the corpus callosum.

236 rected P < .05) in posterior portions of the corpus callosum.

237 ng an increase in unmyelinated fibers in the corpus callosum.

238 ces and corticospinal tracts, to include the corpus callosum; frontal, sensory, and premotor cortices

239 nd dermal abnormalities and the absence of a corpus callosum; his immune deficit was fully corrected

240 The anonymized and annotated Dutch clinical corpus can serve as a useful resource for further algori

241 Outcomes and Measures: Incidence of uterine corpus cancer in BRCA+ women who underwent RRSO without

242 We found that the corpus cardiacum (CC) cells, a group of neuroendocrine c

243 demonstrated using a large, real-world data corpus collected by a local hospital on which it is show

244 Here, in a corpus consisting of 709,577 abstracts published between

245 cardia and fundus or corpus or all of these, corpus, corpus and antrum, and antrum.

246 0.02), while there was a trend for decreased corpus cyclin D1 and ki67 expression (0.17 +/- 0.07 vs 0

247 varian serous cystadenocarcinoma and uterine corpus endometrial carcinomas.

248 ditional named entities in the full 97-paper corpus, for an increase of 76% percent in the semantic c

249 own in human gastric epithelial cells of the corpus/fundus and in HGT-1 cells, a model for the study

250 I:PG II ratio (<5), indicating more advanced corpus gastritis, increased the odds of seroconversion o

251 ated for cell line names: the broad-coverage corpus Gellus and CLL, a focused target domain corpus.

252 the compound repository and patent document corpus; given the wealth of knowledge hidden in patent d

253 Lineage tracing experiments of the gastric corpus in mice have not established whether SPEM derives

254 The ADR types were adapted from a DDI corpus, including i) adverse effect; ii) effect at molec

255 on marks quiescent stem cells in the gastric corpus isthmus.

256 However, B&P overlook a vast (corpus-)linguistic literature that converges with - but

257 , smaller ovaries with a decreased number of corpus luteum, and an increased number of cystic/atretic

258 critical questions about Latin literature, a corpus marked by an extraordinary concentration of inter

259 study we aimed to compare pattern of gastric corpus microbiota in ESCC with normal esophagus.

260 GABAergic currents and modifies the gastric corpus motility response to OXT.

261 he first to show that composition of gastric corpus mucosal microbiota differs in early ESCC and ESD

262 s high performance on diseases (NCBI Disease corpus, NER f-score: 0.829, normalization f-score: 0.807

263 ore: 0.807) and chemicals (BioCreative 5 CDR corpus, NER f-score: 0.914, normalization f-score 0.895)

264 We introduce a corpus of 1499 PubMed abstracts annotated according to t

265 on and recall when evaluated on a literature corpus of 150 abstracts with F-scores close to 0.90 on t

266 on the test portion of a manually annotated corpus of 320 Medline abstracts, a 4-fold improvement ov

267 racy of the tagger on a new manually curated corpus of 600 Encyclopedia of Life (EOL) species pages.

268 ng the network properties of a comprehensive corpus of 70 complex diseases.

269 We introduce a new ultradense corpus of audio and video recordings of a single child's

270 e of several unsupervised methods on a large corpus of biomedical abstracts collected from PubMed.

271 t diseases that is scattered across the vast corpus of biomedical literature.

272 le the existence of superassemblies with the corpus of data that drove the field to abandon the early

273 erwhelmed or confused when faced with such a corpus of detailed insights, nomenclature rules, and deb

274 ate the identified neighborhoods for a large corpus of diseases.

275 The corpus of documentation of these resources is fragmented

276 stem has been tested on a manually annotated corpus of journal articles related to phylogeography usi

277 Our model leverages a corpus of k-mers to reduce the entropy of the quality sc

278 evious work and validated using a large data corpus of medical records from a local hospital.

279 ddresses these problems by assembling a vast corpus of regional newspapers from the United Kingdom, i

280 Finally the role of a corpus of robustly tested medicinal chemistry knowledge

281 bined across multiple papers to appraise the corpus of scientists, institutions, journals, or countri

282 The accelerating growth in the corpus of sequencing data that underpins such analysis i

283 machine-learning model trained on a standard corpus of text from the World Wide Web.

284 as esophageal, cardia, cardia and fundus or corpus or all of these, corpus, corpus and antrum, and a

285 ] vs 2.91 +/- 0.3 [control group, pancreatic corpus]; p = 0.025).

286 ri (strain J99) from a patient who developed corpus-predominant gastritis and hypochlorhydia over a 6

287 This corpus provides the first direct comparison, to our know

288 The corpus spans 150 years and is formed by millions of arti

289 ade injections into the IC and amygdala plus corpus striatum as well into the IC and auditory cortex

290 highly selective and profound damage to the corpus striatum, which regulates motor function.

291 DNA was extracted from snap-frozen gastric corpus tissues and 16S rRNA was sequenced on GS-FLX Tita

292 he classification model trained with the DDI corpus to identify the drugs that might interact with th

293 We use this corpus to train a system that classifies PubMed literatu

294 without needs to have access to the language corpus underneath.

295 21%-55%), breast (31%; 95% CI, 11%-51%), and corpus uteri (27%; 95% CI, 11%-43%).

296 Corpus uteri, postmenopausal breast, and colon cancers a

297 The corpus was manually annotated with coreference relations

298 e F-measure performance on the gold standard corpus was observed.

299 Colorado Richly Annotated Full Text (CRAFT) corpus with coreference relations.

300 e System, we identified medical terms in the corpus with exact matching.