ライフサイエンスコーパス: corpus cavernosum

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1 e was studied in vitro using isolated rabbit corpus cavernosum.

2 PDE5, which causes cGMP to accumulate in the corpus cavernosum.

3 rotein was present in the endothelium of the corpus cavernosum.

4 ived nitric oxide is present in the diabetic corpus cavernosum.

5 nhibitor rosuvastatin (RSV) were examined on corpus cavernosum and aorta from streptozotocin-induced

6 monophosphate hydrolysis [corrected] in the corpus cavernosum and therefore increases the penile res

7 Since NO synthase is found in human clitoral corpus cavernosum and vagina, we hypothesized that human

8 -) currents underlie detumescent tone in the corpus cavernosum, and that modulation of this mechanism

9 Many tonic SMCs (vascular, airway and corpus cavernosum) are driven by a cytosolic Ca2+ oscill

10 A Rho-kinase inhibitor increases corpus cavernosum (CC) pressure in an in vivo rat model

11 - mice, and this effect was reproduced in WT corpus cavernosum exposed to NOS inhibitors.

12 In rabbit penile corpus cavernosum, intracellular cGMP was determined to

13 tissues, including the smooth muscle of the corpus cavernosum of the penis.

14 increased blood flow into the penis, raising corpus cavernosum pressure to culminate in penile erecti

15 ntagonism of Rho-kinase results in increased corpus cavernosum pressure, initiating the erectile resp

16 Relaxation of arterial and corpus cavernosum smooth muscle (CCSM) is necessary to i

17 o study membrane currents in isolated rabbit corpus cavernosum smooth muscle cells.

18 oavailability to NO synthase in human penile corpus cavernosum smooth muscle, the inhibition of human

19 ent smooth muscle relaxation in human penile corpus cavernosum tissue that is required for erection.

20 ate that arginase is present in human penile corpus cavernosum tissue, and that the arginase inhibito

21 ed the functional role of BK channels in the corpus cavernosum utilizing a knock-out mouse lacking th

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