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1 of GAPDH and Siah1 in the dopamine-enriched corpus striatum.
2 dopamine from slices and synaptosomes of rat corpus striatum.
3 cerebellum, and D(1A) is highly expressed in corpus striatum.
4 r adherence to plastic were infused into the corpus striatum.
5 ei; the posterior hypothalamic area; and the corpus striatum.
6 oncentrations within the olfactory bulbs and corpus striatum.
7 were determined from the olfactory bulbs and corpus striatum.
8 trically stimulated superfused slices of rat corpus striatum.
9 neuropathology is largely restricted to the corpus striatum.
11 to examine 5-HT release in the hippocampus, corpus striatum, amygdala, and prefrontal cortex of the
12 neural dysfunction and death, mainly in the corpus striatum and cerebral cortex, resulting in pathog
15 or no beta(2) expression was observed in the corpus striatum and in granule cells of the olfactory bu
16 lating MPP(+)-induced dopamine output in the corpus striatum and nucleus accumbens of ovariectomized
19 in midbrain, pons and medulla, hippocampus, corpus striatum and spinal cord of mice following an inj
20 pable of attenuating degeneration within the corpus striatum, and in this way serve as a neuroprotect
22 ade injections into the IC and amygdala plus corpus striatum as well into the IC and auditory cortex
23 MK-801 was increased in pons and medulla and corpus striatum but decreased in midbrain and hippocampu
25 tic l-amino acid decarboxylase (AADC) in the corpus striatum (CS) and substantia nigra (SN) of the ra
26 5 is highly expressed in the olfactory bulb, corpus striatum, dentate gyrus, CA1 region of the hippoc
27 lectrochemical detection from superfusion of corpus striatum fragments with Kreb's ringer phosphate b
28 ume of the temporo-occipital regions and the corpus striatum; (ii) emotional problems and grey matter
30 shown to modulate synaptic plasticity in the corpus striatum in vitro by activation of the tyrosine k
32 nephrine within both the olfactory bulbs and corpus striatum of mice receiving the intranasal infusio
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