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1  of GAPDH and Siah1 in the dopamine-enriched corpus striatum.
2 dopamine from slices and synaptosomes of rat corpus striatum.
3 cerebellum, and D(1A) is highly expressed in corpus striatum.
4 r adherence to plastic were infused into the corpus striatum.
5 ei; the posterior hypothalamic area; and the corpus striatum.
6 oncentrations within the olfactory bulbs and corpus striatum.
7 were determined from the olfactory bulbs and corpus striatum.
8 trically stimulated superfused slices of rat corpus striatum.
9  neuropathology is largely restricted to the corpus striatum.
10 ter (-6%), accompanied by hypertrophy of the corpus striatum (+14%).
11  to examine 5-HT release in the hippocampus, corpus striatum, amygdala, and prefrontal cortex of the
12  neural dysfunction and death, mainly in the corpus striatum and cerebral cortex, resulting in pathog
13                                       In the corpus striatum and cerebral cortex, the D3- and D4-rece
14 e cerebral cortex, and reduced volume of the corpus striatum and hippocampus.
15 or no beta(2) expression was observed in the corpus striatum and in granule cells of the olfactory bu
16 lating MPP(+)-induced dopamine output in the corpus striatum and nucleus accumbens of ovariectomized
17 he MPP(+)-evoked dopamine signal in both the corpus striatum and nucleus accumbens.
18 lectrodes stereotaxically passed through the corpus striatum and nucleus accumbens.
19  in midbrain, pons and medulla, hippocampus, corpus striatum and spinal cord of mice following an inj
20 pable of attenuating degeneration within the corpus striatum, and in this way serve as a neuroprotect
21                                 Cells of the corpus striatum arise from the lateral ganglionic eminen
22 ade injections into the IC and amygdala plus corpus striatum as well into the IC and auditory cortex
23 MK-801 was increased in pons and medulla and corpus striatum but decreased in midbrain and hippocampu
24 ntly smaller increase in 5-HT release in the corpus striatum compared to the other three sites.
25 tic l-amino acid decarboxylase (AADC) in the corpus striatum (CS) and substantia nigra (SN) of the ra
26 5 is highly expressed in the olfactory bulb, corpus striatum, dentate gyrus, CA1 region of the hippoc
27 lectrochemical detection from superfusion of corpus striatum fragments with Kreb's ringer phosphate b
28 ume of the temporo-occipital regions and the corpus striatum; (ii) emotional problems and grey matter
29 -OHDA lesioned and non-lesioned sides of the corpus striatum in these animals.
30 shown to modulate synaptic plasticity in the corpus striatum in vitro by activation of the tyrosine k
31     Despite a massive loss of neurons in the corpus striatum, NO-generating neurons are intact.
32 nephrine within both the olfactory bulbs and corpus striatum of mice receiving the intranasal infusio
33 stischemic cell death in the hippocampus and corpus striatum of rats.
34 DDC and 5-HTPDC) in the substantia nigra and corpus striatum of reserpine-treated rats.
35 umoylation, which is markedly reduced in the corpus striatum of Rhes-deleted mice.
36                                 Then, in the corpus striatum, the brain area enriched in dopamine, th
37                                       In the corpus striatum, we found a close anatomical correlation
38  highly selective and profound damage to the corpus striatum, which regulates motor function.

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