ライフサイエンスコーパス: corrected QT interval

1 dia, or apparent risk of prolongation of the corrected QT interval.

2 ngation of the action potential duration and corrected QT interval.

3 ormalities, particularly prolongation of the corrected QT interval.

4 T segment shift; and the duration of QRS and corrected QT intervals.

5 TS patients (84% male; age, 26 +/- 15 years; corrected QT interval, 329 +/- 22 ms) were studied, and

6 rmulas, only 24% to 32% of patients had rate-corrected QT intervals above 500 ms.

7 There was a small increase in corrected QT interval after ibutilide (from442 +/- 61 ms

8 Analysis of corrected QT interval among 74 control subjects from our

9 ts (10 g of ethanol) per day with heart rate-corrected QT interval and heart rate assessed from elect

10 ing therapies may be effective in shortening corrected QT interval and reducing TdP recurrence risk.

11 Mechanisms for longer rate-corrected QT intervals and higher incidences of drug-ind

12 e variability were assessed every 30 min and corrected QT intervals and T-wave morphology every 60 mi

13 ventricular hypertrophy, prolongation of the corrected QT interval, and repolarization changes (ST/T

14 urther, the majority of LQTS patients have a corrected QT interval below this threshold, and a signif

15 ypes were neither associated with heart rate-corrected QT interval duration (QTc) nor cardiac events

16 were not associated with MeanNN, heart-rate-corrected QT interval duration (QTc), deceleration capac

17 gestive features that, along with heart rate-corrected QT interval duration, may risk stratify perina

18 Median corrected QT interval for heart rate was 312 ms (range:

19 in serum K(+) resulted in a decrease in the corrected QT interval from 526 +/- 94 to 423 +/- 36 ms (

20 A total of 1,059 LQTS patients with a corrected QT interval > or =450 ms presenting with synco

21 No patient in either group exhibited a corrected QT interval >/=500 msec.

22 A corrected QT interval >500 msecs was considered prolonge

23 , fatigue in one; hypertension and prolonged corrected QT interval in another) occurred in patients i

24 +/-24.9) was longer than neonatal heart rate-corrected QT interval in both group 2 (491.2+/-27.6; P=0

25 ygotes, revealed an age-dependent heart rate-corrected QT interval increase (1% per additional 10 yea

26 Variants investigated altered heart rate-corrected QT interval irrespective of mutation status, a

27 Prolonged heart rate-corrected QT interval is associated with higher risk of

28 lf is insufficient for diagnosis, unless the corrected QT interval is repeatedly >/=500 ms without an

29 ECG or arrhythmia phenotype, and only 2 had corrected QT interval longer than 500 milliseconds.

30 olic blood pressure, heart rate variability, corrected QT interval, low density lipoprotein (LDL) cho

31 probands displaying ST-segment elevation and corrected QT intervals < or = 360 ms had mutations in ge

32 me is a new clinical entity characterized by corrected QT intervals <300 ms and a high incidence of v

33 cantly (p < 0.05) prolonged, as indexed by a corrected QT interval (mean [+/-SD] 311 +/- 25 to 338 +/

34 The neonatal heart rate-corrected QT interval (mean+/-SE) of group 1 (664.7+/-24

35 n-treatment electrocardiogram occurrences of corrected QT interval more than 500 ms (an indicator of

36 verage age of onset of 10 months, an average corrected QT interval of 676 ms, and a high prevalence o

37 After 72 hours, the corrected QT interval of the electrocardiogram was reduc

38 nicity index was increased (P<0.001) and the corrected QT interval on ECG was prolonged (P<0.001) in

39 Prolonged heart rate-corrected QT interval on electrocardiograms (ECGs) is as

40 associated with uncorrected QT interval, HR-corrected QT interval or high-density lipoprotein-choles

41 RE: rs12734991 in meta-analysis: increase in corrected QT interval per C allele: 9.1 +/- 3.2 ms, p =

42 nation was not predicted by the magnitude of corrected QT interval prolongation but was associated wi

43 No significant associations were seen among corrected QT interval prolongation, repolarization chang

44 The proportion of patients who developed corrected QT-interval prolongation (p = 0.16), extrapyra

45 Arsenic is known to cause corrected QT-interval prolongation and T-wave changes, b

46 As expected, in TdP patients, many known corrected QT interval-prolonging risk factors were simul

47 a 7.7% +/- 0.9% shortening of the heart rate-corrected QT interval (QTc interval) in Kir2.1-transduce

48 All patients developed corrected QT interval (QTc interval) prolongation (media

49 ment depression (STD) >/=50 micro V and rate-corrected QT interval (QTc) >460 ms were examined as mea

50 ic testing correlated significantly with the corrected QT interval (QTc) and clinical diagnostic scor

51 to study the predictive value of heart rate-corrected QT interval (QTc) for incident coronary heart

52 (P<0.05), and an increase in the heart rate-corrected QT interval (QTc) from 379+/-10 to 504+/-11 ms

53 Heart rate-corrected QT interval (QTc) is the traditional method of

54 arrhythmia, of which lengthening of the rate-corrected QT interval (QTc) on the electrocardiogram is

55 , 2 of 15 patients experienced dose-limiting corrected QT interval (QTc) prolongation, pneumonitis, o

56 The corrected QT interval (QTc) should be assessed as a rout

57 n a recent cohort study, prolongation of the corrected QT interval (QTc) was associated with an indep

58 forms in labeling prolongation of heart rate-corrected QT interval (QTc), an arrhythmia risk marker.

59 ying effects of AKAP9 variants on heart rate-corrected QT interval (QTc), cardiac events, and disease

60 in cases with exertional syncope and normal corrected QT interval (QTc).

61 rrhythmia risk factors and quantification of corrected QT interval (QTc).

62 ects differed from control subjects: resting corrected QT interval (QTc, 627 +/- 90 versus 425 +/- 25

63 the risk for TdP included absolute and rate-corrected QT intervals (QTc) on drug therapy, the magnit

64 The median baseline corrected QT intervals (QTc) were 444 ms (gene negative)

65 ized into electrocardiographically affected (corrected QT interval [QTc] > or = 470 ms), borderline (

66 dden cardiac death during childhood included corrected QT interval [QTc] duration > 500 ms (hazard ra

67 males, median age 16 years, average referral corrected QT interval [QTc] of 481 ms) referred with a d

68 G parameters (QRS voltage, QRS duration, and corrected QT interval [QTc]) were evaluated by using mul

69 tion potential and cause prolongation of the corrected QT interval, QTc.

70 alyses controlling for risk factors and rate-corrected QT interval, the PCA ratio remained a signific

71 despite having essentially identical resting corrected QT interval values.

72 nd a significant minority has normal resting corrected QT interval values.

73 Mean corrected QT interval was 403 (standard deviation, 30) m

74 Corrected QT interval was measured by surface ECG.

75 However, a prolonged rate-corrected QT interval was not a consistent feature, indi

76 The average corrected QT interval was significantly shorter in peopl

77 Frontal T axis, heart rate, and heart rate-corrected QT interval were the most significant ECG fact

78 In the 1270 (63%) with ECGs, corrected QT intervals were not different in variant car

79 duration of treatment, flecainide levels and corrected QT intervals were recorded; 24 h Holter monito

80 t alcohol units were not associated with the corrected QT interval, with beta = 1.04 (95% confidence