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1 individuals enrolled in the PET pathological correlation study).
2 Clinicohistologic correlation study.
3 This is a longitudinal correlation study.
4 s to undertake a detailed genotype-phenotype correlation study.
5 tagenesis, and facilitate genotype-phenotype correlation studies.
6 es in model organisms and genotype-phenotype correlation studies.
7 l provide a template for future mRNA-protein correlation studies.
8 ethods and confirmed by a series of chemical correlation studies.
9 relation were used for group comparisons and correlation studies.
10 nonfluorescent quenchers in single-molecule correlation studies.
11 s by performing combined histopathologic-MRI correlation studies.
12 gl1, as validated by functional and clinical correlation studies.
13 that can now be tested in clinicopathologic correlation studies.
14 dependent queries and genome wide expression correlation studies.
15 cancer image analysis and clinical data for correlation studies.
16 of interest outside the context of neuronal correlations studies.
21 bar-segmental analysis in structure-function correlation study and provided the anatomical basis for
22 owing amount of data from genotype-phenotype correlation studies, and the introduction of commerciall
23 systems, retrospective preclinical-clinical correlation studies are available, which suggest that im
25 conditions are unnatural [4], and long-term correlation studies based on historical collections [5].
26 al analysis followed by a genotype-phenotype correlation study based on the clinical characteristics
32 ing, and Participants: This genetic-clinical correlation study examined BRAF V600E and TERT promoter
35 he past decade, scores of genotype-phenotype correlation studies in both hypertrophic cardiomyopathy
36 tasis in patient-derived xenograft model and correlation studies in lung cancer patients further vali
37 is aided by the well-defined geometry and a correlation study in scanning electron microscopy and to
41 a typical dataset in heterozygosity-fitness correlation studies involving 10-20 microsatellites and
44 inding by HMGB domain proteins is given by a correlation study of NHP6A and 32 other HMGB domains bel
50 In classic Rett syndrome, genotype-phenotype correlation studies suggest that X chromosome inactivati
53 composite-phenotypes, beyond family or twin correlation studies that initially demonstrate evidence
54 spill, we constructed diagrams and conducted correlation studies that measure the similarity between
58 ence microscopy supplemented by fluorescence correlation studies to determine the structural factors
62 is, in conjunction with structure-reactivity correlation studies, we show that para-substituted benzy
65 e mutation and additional genotype-phenotype correlation studies will provide fundamental insight int
66 trochemical results was demonstrated through correlation studies with the results obtained with conve
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