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1 n changes might precede diffuse atrophy with cortical thinning.
2 Children with ADHD had the slowest rate of cortical thinning.
3 ith and contribute additively to hippocampal cortical thinning.
4 es were associated with discrete patterns of cortical thinning.
5 ain development, including hydrocephalus and cortical thinning.
6 of known protective factors for age-related cortical thinning.
7 extrapolated to further adult aging-related cortical thinning.
8 an be characterised by a specific pattern of cortical thinning.
9 osure group showed significant and bilateral cortical thinning.
10 of reduced anterior cingulate activation and cortical thinning.
11 sive enlargement of the medullary cavity and cortical thinning.
13 oking is associated with diffuse accelerated cortical thinning, a biomarker of cognitive decline in a
14 s, increased CAC scores were associated with cortical thinning across several brain regions, which ge
15 obar CMBs were independently associated with cortical thinning across the entire cortical regions whi
16 low-risk groups, detecting large expanses of cortical thinning across the lateral surface of the righ
17 isk families also revealed large expanses of cortical thinning across the lateral surface of the righ
19 apeutic effect on knee OA pain by preventing cortical thinning and decreases in functional connectivi
21 d hydronephrosis and hydroureter with marked cortical thinning and multiple bilateral PET-avid pulmon
23 thy is associated with a distinct pattern of cortical thinning and reduced functional connectivity.
24 methodology allows for a distinction between cortical thinning and reduction in cortical area and rev
25 e relationship between the rates of regional cortical thinning and severity of symptoms of hyperactiv
26 We compared regional percentage change of cortical thinning and subcortical atrophy over 18 months
27 , a significant association between regional cortical thinning and total functional capacity, current
28 uli in direct proportion to the magnitude of cortical thinning and white matter hypoplasia within the
30 lobally, sulcal widening was associated with cortical thinning and, for the frontal cortex, with loss
31 gional change in macrostructure (atrophy and cortical thinning), and microstructure (diffusion metric
32 re children also showed frontal and parietal cortical thinning, and an inverse dose-response relation
33 urological problems, including microcephaly, cortical thinning, and blindness during early developmen
34 or to cortical volume reductions in aging is cortical thinning, and that magnitude of change varies w
35 for Alzheimer's disease was associated with cortical thinning as a function of time since injury.
36 causes selective elimination of neurons and cortical thinning as observed in some forms of mental re
37 how a similar but less pronounced pattern of cortical thinning as patients with a psychotic disorder,
43 extensive atrophy and greater percentage of cortical thinning compared to Parkinson's disease with n
44 irst study indicating that prefrontotemporal cortical thinning constitutes a relevant cortical pathom
45 sability and impaired cognitive performance; cortical thinning correlated with increased thalamic TSP
46 aralleled at postmortem by striatal atrophy, cortical thinning, degeneration of medium spiny neurons,
50 ural brain networks that undergo coordinated cortical thinning during adolescence, which is in part g
51 ally, they examined whether a slower rate of cortical thinning during late childhood and adolescence,
52 particular, the association of exercise with cortical thinning had regional specificity in the bilate
53 nalysis of the fetal brain, diffuse cerebral cortical thinning, high ZIKV RNA loads, and viral partic
56 at 25 gestational weeks was associated with cortical thinning in 19% of the whole cortex and 24% of
57 eukocytes associated with annualized rate of cortical thinning in a subsample of the NAPLS cohort (N=
58 mpal activity was negatively correlated with cortical thinning in a subset of regions, including the
59 es have found reduced gray matter volume and cortical thinning in acutely underweight patients to nor
62 n conclusion, the characteristic patterns of cortical thinning in Alzheimer's disease and FTD suggest
63 through cortical thickness, suggesting that cortical thinning in Alzheimer's disease-vulnerable brai
64 ar neuron depletion and progressive cerebral cortical thinning in an animal model of Canavan disease.
65 orms of childhood abuse were associated with cortical thinning in areas critical to the perception an
66 ively normal adults, CAC was associated with cortical thinning in areas related to cognitive function
67 ood and discrepantly accelerated age-related cortical thinning in autism spectrum disorders suggest t
70 sing magnetic resonance imaging, we detected cortical thinning in frontal and parietal lobes in group
71 ognitive impairment demonstrated more severe cortical thinning in frontal and temporo-parietal cortic
72 evidence linking the complement system with cortical thinning in humans, a process potentially invol
73 th antisocial personality disorder exhibited cortical thinning in inferior mesial frontal cortices.
75 reduced higher visual function at baseline, cortical thinning in parietal, occipital and frontal cor
76 an unbiased estimate of the distribution of cortical thinning in patients (relative to controls) by
78 objectives were to test the hypothesis that cortical thinning in patients with schizophrenia (relati
79 g reductions in brain volume and progressive cortical thinning in patients with schizophrenia who are
80 rnalizing behavior (p < .05) was mediated by cortical thinning in prefrontal areas of the right hemis
82 better clinical outcome, is associated with cortical thinning in regions important in attentional co
83 ontrast, emotional abuse was associated with cortical thinning in regions relevant to self-awareness
84 omplement pathway may contribute to aberrant cortical thinning in schizophrenia prodromes and reduced
86 elative to controls, patients showed greater cortical thinning in temporal-prefrontal ROIs than in co
88 eference shift was statistically mediated by cortical thinning in the dorsomedial prefrontal cortex.
89 was specifically associated with pronounced cortical thinning in the genital representation field of
90 y developing cohort without ADHD showed that cortical thinning in the group not taking psychostimulan
91 The group difference was due to more rapid cortical thinning in the group not taking psychostimulan
92 ood disorders is associated with progressive cortical thinning in the left inferior frontal and prece
93 peduncle, and smaller grey matter volume and cortical thinning in the leg area of the primary motor a
94 rly inattentive symptoms, so did the rate of cortical thinning in the medial and dorsolateral prefron
100 ere to detect the characteristic patterns of cortical thinning in these two types of dementia, to tes
101 ntrast with previous findings of age-related cortical thinning in this same autism spectrum disorders
102 se of one symptom of adult ADHD, the rate of cortical thinning increased by .0018 mm (SE = .0004, t =
103 ated with cortical thinning, suggesting that cortical thinning is spatially associated with white mat
104 reduction in cortical area and reveals that cortical thinning is the most important factor in volume
105 there was a decrease in the rate of regional cortical thinning of 0.0054 mm/year (SE=0.0019 mm/year).
106 the group not taking psychostimulants (mean cortical thinning of 0.16 mm/year [SD=0.17], compared wi
107 nges in bone composition, and trabecular and cortical thinning of bone accompanied by increased bone
108 The preference shift was associated with cortical thinning of the dorsomedial prefrontal cortex a
109 s who improved were more likely to have less cortical thinning of the left medial frontal cortex and
112 spinal cord lesions, and global and regional cortical thinning) parameters were assessed in a trainin
113 so in a manner partially mediated by rate of cortical thinning (point estimate=0.078 (95% CIs: 0.003,
114 unters to some extent the vulnerability that cortical thinning poses for developing familial depressi
115 peractivity/impulsivity had a slower rate of cortical thinning, predominantly in prefrontal cortical
117 nce of CSS was independently associated with cortical thinning primarily in the bilateral frontal reg
118 exposed control subjects was associated with cortical thinning, primarily in the superior frontal and
119 cognitive impairment demonstrated widespread cortical thinning relative to controls and atrophy of th
120 underlying white matter was correlated with cortical thinning, suggesting that cortical thinning is
121 l intensity differences were consistent with cortical thinning/sulcal widening and ventricular enlarg
123 ening (from ages 8-14) and widespread linear cortical thinning that have dissociable relationships wi
124 with chronic schizophrenia showed widespread cortical thinning that particularly affected the prefron
125 ntipsychotics was associated with prefrontal cortical thinning, treatment was also associated with be
127 prenatal depression coupled with subsequent cortical thinning was associated with presence of extern
128 erters and healthy controls, and the rate of cortical thinning was correlated with levels of proinfla
129 anatomical differences, significant regional cortical thinning was found in lithium-free euthymic pat
133 st that central vision loss may give rise to cortical thinning, while in the same group of people, co
134 The selective topographical associations of cortical thinning with clinical features of HD suggest t
136 action: with increasing age, more pronounced cortical thinning would be observed in autism spectrum d
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