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1 tal, lateral temporal, and lateral occipital cortices).
2 , anterior cingulate and supplementary motor cortices.
3 brain rhythms in left and right sensorimotor cortices.
4 refrontal, and superior and lateral parietal cortices.
5 c premotor, parietal, and posterior temporal cortices.
6 specific regions within frontal and parietal cortices.
7 en mainly in gyral regions in more posterior cortices.
8 oint-angles (intrinsic coordinates) in motor cortices.
9 neral regions of the prefrontal and parietal cortices.
10 n anterior insular, parietal, and prefrontal cortices.
11 l neuroplasticity in prefrontal and auditory cortices.
12 ctional connectivity between primary sensory cortices.
13 na incerta, and the visual and retrosplenial cortices.
14 and overlap with the inputs from the sensory cortices.
15 poral, anterior cingulate, and orbitofrontal cortices.
16 mined by FRET analysis both at cell and bleb cortices.
17 nnectivity between auditory and sensorimotor cortices.
18 ateral medial frontal and posterior parietal cortices.
19 on that extend into barrel and retrosplenial cortices.
20 the granular layer 4 (L4) typical of sensory cortices.
21 subregions of frontal, parietal and temporal cortices.
22 arly visual, temporal, parietal, and frontal cortices.
23 ubdural electrodes over frontal and temporal cortices.
24 and the dorsolateral and anterior prefrontal cortices.
25 hy in the medial temporal lobes and adjacent cortices.
26 fine the boundaries of the SVZ in developing cortices.
27 r bone volume and poorly defined metaphyseal cortices.
28 confined to the left temporal and occipital cortices.
29 ventrolateral prefrontal and dorsal premotor cortices.
30 hemisphere in both the insular and temporal cortices.
31 nction of adaptation in higher-order sensory cortices.
32 ended more broadly into parietal and frontal cortices.
33 rhinal, orbitofrontal, piriform, and insular cortices.
34 processing that may extend to other sensory cortices.
35 es in superior temporal and inferior frontal cortices.
36 ipsilaterally in the entorhinal and piriform cortices.
37 connected to the primary and secondary motor cortices.
38 rom the neighboring somatosensory and visual cortices.
39 tex is well described in the primary sensory cortices.
40 cingulate, somatosensory, and motor/premotor cortices.
41 ally observed during adolescence in anterior cortices.
42 reasing length from auditory to higher-order cortices.
43 underpinned by temporal and inferior-frontal cortices.
44 identified projection neurons across sensory cortices.
45 annabis-exposed children had thicker frontal cortices.
46 hological functions attributed to associated cortices.
47 onnectivity between visual and somatosensory cortices.
48 e-treated cases were investigated in femoral cortices.
49 insular, superior prefrontal, and cerebellar cortices.
50 limit the accurate localization of eloquent cortices.
51 1) in the superior frontal and frontal polar cortices.
52 t its ion-impermeable mutant, in the insular cortices.
53 n the superior frontal and superior parietal cortices.
54 relative abundance of bRG in human and mouse cortices.
55 cell assemblies in the frontal and temporal cortices.
56 pathology may originate in limbic/paralimbic cortices.
57 he basal ganglia, cerebellum, and prefrontal cortices.
58 within a conversation only engaged auditory cortices.
59 phase synchrony between visual and auditory cortices.
60 ty between limbic structures and pre-frontal cortices.
61 codes differ between sensory and association cortices.
62 en mainly in gyral regions in more posterior cortices.
63 tion between lateral prefrontal and parietal cortices.
64 eral prefrontal and lateral temporo-parietal cortices.
65 ch structurally connects the bilateral motor cortices.
66 of within the medial prefrontal and fusiform cortices.
67 ange in the middle frontal gyrus or parietal cortices.
68 l areas over primary motor and somatosensory cortices.
69 ecame a mosaic in Pcdh19 heterozygous female cortices.
70 tex development, Eomes and Pax6, in EFhd2 KO cortices.
71 s with the sensory modalities and prefrontal cortices.
72 y, from primary sensory and motor to frontal cortices.
73 ring and enriches the nascent daughter cell cortices.
74 hronization and activity in right prefrontal cortices.
75 the anterior cingulate and medial prefrontal cortices.
76 hemispheric reflections of sensory and motor cortices.
77 treamlines leave the prefrontal and temporal cortices.
78 lateral occipital and the inferior temporal cortices.
79 reduced ITD-related changes in both auditory cortices.
80 several neocortical areas, including frontal cortices.
81 th sporadic CAA without dementia had thinner cortices (2.17 mm [SD 0.11]) than the two healthy contro
82 The 26 patients with HCHWA-D had thinner cortices (2.31 mm [SD 0.18]) than the 28 healthy control
83 anges in activity in the auditory and visual cortices after auditory deprivation in the adult rats.
84 the anterior cingulate and medial prefrontal cortices, along with posterior cingulate, sensory associ
86 ht frontopolar and right inferior prefrontal cortices, although this activation was greater in female
87 tromedial prefrontal, dorsomedial prefrontal cortices, amygdala, hippocampus, septal region, and hypo
88 howed cortical thinning of the motor-related cortices and a distributed involvement of the prefrontal
89 l parietal, hippocampal, and parahippocampal cortices and amygdala, as well as lower NAA:Cr and Cho:C
90 iatum and the medial prefrontal and parietal cortices and between the dorsal striatum and the somatos
91 stered separately from the external mycelial cortices and fruiting bodies of Chinese Cordyceps from d
92 s), worse executive functioning, and thinner cortices and less brain activation in diffusely distribu
93 the nexus of visual inputs from retinotopic cortices and linguistic input from the frontotemporal la
94 vFTD, with the addition of the orbitofrontal cortices and nucleus accumbens in patients with semantic
95 d methylome profiling in normal and DS fetal cortices and observed a significant hypermethylation in
97 cingulate, and posterior cingulate cerebral cortices and the cerebellar cortex of 87 end-of-life pat
98 onally, the activities of the right auditory cortices and the parahippocampus, areas that generate ti
99 ctivations to food cues (parietal and visual cortices) and emotional and limbic activity (insula, amy
101 al targets (extrinsic coordinates) in visual cortices, and across movements with equivalent joint-ang
102 the frontal, temporal, parietal, and insular cortices, and in some subcortical regions, including the
103 in sulcal regions in prefrontal and temporal cortices, and negative relationships seen mainly in gyra
106 l genes, includes the medial and dorsomedial cortices, and the majority of the dorsal cortex, except
107 temporal gyrus (STG), the sensory and visual cortices, and two higher-order regions within the intrap
108 the APOE epsilon4 allele, thinner entorhinal cortices, and worse longitudinal trajectory of Logical M
109 area, whereas the somatosensory and auditory cortices are connected to the primary and secondary moto
110 LP, the medial and lateral secondary visual cortices are connected with lateral LP and a portion of
111 an brains and fetal brains show that thicker cortices are consistently found in gyral regions and gyr
112 sory interactions in primary and association cortices are governed by distinct computational principl
114 The primary (MOp) and secondary (MOs) motor cortices are known to produce specific output projection
115 rature has demonstrated that primary sensory cortices are not exclusively unimodal, but can respond t
116 how that fMRI response patterns in the motor cortices are similar for both arms if the movement direc
119 scillatory activity at the bilateral insular cortices as well as connectivity patterns that reflect a
120 of dorsal frontal and lateral orbitofrontal cortices as well as the effects of family disadvantage o
121 ols in the occipital, parietal, and temporal cortices as well as the posterior cingulate gyrus, precu
122 interactions between the auditory and visual cortices, as opposed to fusion of AV percepts in a multi
125 rators identified at right and left auditory cortices at 6 and 12 months and also at frontal cortex a
129 circuits involving SCm, Cg, secondary visual cortices, auditory areas, and the dysgranular retrospeni
130 ry is defined to express the neuron or brain cortices based on the biology and graph theories, and we
133 nsistent activation in the target visuomotor cortices, both with and without perceptual awareness, sp
135 interneurons (CINs) are found in adult mouse cortices, but the mechanism generating their diversity r
137 llum, pons, left amygdala, and orbitofrontal cortices (cluster level, family-wise error corrected, P
138 y somatosensory, motor, and ventral premotor cortices coded preferentially the number of digits while
140 al orbitofrontal and dorsolateral prefrontal cortices compared with healthy men and female patients.
141 int, the inactivation of the entire auditory cortices completely prevented the formation of new memor
144 altered in App (NL-G-F/NL-G-F) and human AD cortices correlated with the inflammatory response or im
146 t NM had higher FCD in visual and prefrontal cortices, default mode network regions and thalamus, whi
148 self-produced speech, suppression of sensory cortices, did not occur during joint synchronized speech
150 S over the bilateral dorsolateral prefrontal cortices (dlPFCs) improved learning and performance of c
151 M coding arises as a property of association cortices downstream from the early stages of sensory pro
152 that memories stored in higher-order sensory cortices drive BLA activity when distinguishing between
153 responses of neurons in frontal and parietal cortices during a pulse-based accumulation of evidence t
154 l cortex, posterior cingulate, and occipital cortices during evaluation of positive social feedback.
155 in medial prefrontal and posterior cingulate cortices during goal-directed action selection in the tr
156 temporal correlations) of the frontocentral cortices during rest and follow-up neural and behavioral
157 han competitive interaction of the two motor cortices during skill learning and suggest that bihemisp
159 sham tDCS over their dorsolateral prefrontal cortices during two 30-minute mathematics training sessi
160 late and dorsal and ventrolateral prefrontal cortices) during cognitive modulation and behavioral con
162 xtending through frontal and temporoparietal cortices, especially in those with the most severe sympt
163 d in the hemodynamic activity of vocal-motor cortices, even after individual motor and sensory compon
164 activated regions in prefrontal and parietal cortices (excluding superior IPS) did not exhibit such a
165 more, in blindness, number-responsive visual cortices exhibited increased functional connectivity wit
166 lu increased in the primary visual and motor cortices for eyes open and finger tapping, respectively
168 rior parietal (PPC), and frontal motor (fMC) cortices for sensorimotor mapping in mice during perform
170 of multisensory processing in primary visual cortices further indicates that neural reuse is a basic
172 tofrontal (OFC) and anterior cingulate (ACC) cortices has been linked with several psychiatric disord
173 al reorganization in the auditory and visual cortices has been reported after hearing and visual defi
174 analysis of neural dynamics in several brain cortices has consistently uncovered low-dimensional mani
175 ed that the temporal and heteromodal insular cortices have a central role in propagating these neural
177 associated with human occipital and temporal cortices, here we show, by measuring fMRI response patte
178 D4T (Stavudine 10 mg/kg/day) for 5 days, and cortices, hippocampi and spinal cords were collected for
179 g reduced correlation between left and right cortices (homotopic correlation) within the visual netwo
180 hite matter tracts connecting frontoparietal cortices (i.e., structural connectivity, SC), coordinate
181 edial orbitofrontal, and posterior cingulate cortices, i.e., several of the core regions of the defau
182 higher functional MRI activity, with thicker cortices in dorsolateral prefrontal brain regions, and w
183 s does not differ between frontal and visual cortices in either species, but is significantly higher
185 in the left posterior temporal and occipital cortices in patients with the logopenic variant, within
186 striatum and bilateral frontal and parietal cortices in response to conditioned reward stimuli in th
187 in the attention-related parietal and visual cortices in response to highly palatable food cues at 1
188 nhibition (SICI) was reduced over both motor cortices in stroke patients without diabetes compared wi
189 um, parietal lobe, and prefrontal and visual cortices in the brain that may serve to perpetuate consu
190 osterior parietal (PPC) and prefrontal (PFC) cortices in two male monkeys that performed spatial and
191 lities during adolescence in BDI in anterior cortices, including altered developmental trajectories o
192 08 with gray matter (GM) volume variation in cortices, including the vicinity of the Perisylvian hete
193 in a network linking motor and somatosensory cortices increased with trial-to-trial changes in direct
194 between orbitofrontal (OFC) and rhinal (Rh) cortices influences the judgment of reward size, we reve
198 c premotor, parietal, and posterior temporal cortices is activated even under subliminal perceptual c
199 n the medial temporal, parietal, and frontal cortices is correlated with tau-related cerebrospinal fl
201 ciated immediate early gene in rat olfactory cortices is uninterrupted by propofol, an intravenous ge
203 spatial representations, whereas in parietal cortices, it determined the influence of task-irrelevant
204 anterior paralimbic and heteromodal frontal cortices, key structures in emotional regulation process
205 essively from posterior to anterior temporal cortices, leaving threat as the dominant explanatory var
206 addition, Pcdh19 overexpression in wild-type cortices led to ectopic clustering of Pcdh19-positive ne
207 the superior, middle, and inferior temporal cortices, little is known about the real-time brain acti
208 We recently reconstituted minimal actin cortices (MACs) and here advanced our assay to investiga
210 es sensory percepts, suggesting that sensory cortices might be intrinsically susceptible to SDs.
211 l areas, such as the parietal and prefrontal cortices, mnemonically encode the remembered stimulus.
213 in both ipsilateral and contralateral motor cortices, neural populations have effector-invariant cod
214 and somatosensory, piriform, and entorhinal cortices of all three strains of p75(NTR) mutant mice.
220 ivity from prefrontal, parietal and temporal cortices of healthy adults (n = 19) during memory encodi
221 Previous studies of prefrontal and premotor cortices of macaque monkeys have found neural signals as
222 rimary (S1) and secondary (S2) somatosensory cortices of mice performing a tactile detection task.
223 arying signals in auditory and somatosensory cortices of monkeys is the opponent model of rate coding
225 nes examined, seven were misexpressed in the cortices of Tbr1 knockout mice, including six with incre
226 -3) neuropil of visual (V1) and frontal (FC) cortices of the adult mouse and compared findings to tho
227 ite matter adjacent to the motor and sensory cortices of the hand and the entire cerebral white matte
228 cal neurons in prefrontal, motor, and visual cortices of the Siberian tiger (Panthera tigris altaica)
229 eficits in gamma frequency in the prefrontal cortices of transgenic mice overexpressingDyrk1A We also
230 demonstrated in doxycycline-treated dTg-211 cortices overrepresentation of synaptic activity, Ca(2+)
231 ith volume loss in the auditory and premotor cortices (P < .001), whereas worse performance on the 9-
232 rontal, rostral, and dorsolateral prefrontal cortices (p < .05, corrected), including greater gray ma
233 ortex and the superior and inferior parietal cortices (P = 0.0006, 0.0015, and 0.0023, respectively),
234 EG electrodes over frontal (FC) and parietal cortices (PC) and later tested under vehicle (saline, i.
235 aphy data obtained from frontal and temporal cortices, permitting detailed spatiotemporal analysis of
236 Dorsal premotor (PMd) and primary motor (M1) cortices play a central role in mapping sensation to mov
237 CE STATEMENT The dorsal and ventral premotor cortices (PMd and PMv, respectively) are two specialized
239 ed increases in the motor cortex, prefrontal cortices, posterior parietal cortex, striatum, and thala
240 ns such as parahippocampal and retrosplenial cortices provide critical inputs that allow cognitive ma
241 hat includes the frontoinsular and cingulate cortices, provides a unique lesion model for elucidating
242 population findings in prefrontal and motor cortices, providing essential context to those studies.
243 eft rostral and pregenual anterior cingulate cortices (rACC/pgACC), which control parasympathetic act
244 ow the GABA levels of parietal and occipital cortices related to interindividual differences in size,
247 stimulation frequencies at which the sensory cortices respond maximally, influenced the impact of FDP
248 (NPCs) and neurons from developing cerebral cortices, revealing hundreds of differentially spliced e
249 ntral pallidum, bilateral anterior cingulate cortices, right hypothalamus and bilateral amygdala).
250 rostrolateral visual, and medial entorhinal cortices send projections only to the ipsilateral claust
251 al, parietal, and inferotemporal association cortices show robust sustained activity encoding the loc
252 et, brain activity pattern across widespread cortices significantly predicted whether a threshold-lev
253 , engaging prefrontal and anterior cingulate cortices similarly to many types of effortful task switc
255 nal responses in the human superior temporal cortices (STC) we collected fMRI data from deaf and hear
256 l loss in the frontal, parietal and temporal cortices, striatum, substantia nigra and subthalamic nuc
258 dual neurons have not been found in nonhuman cortices, suggesting that these synapses are specific to
259 rhinal, piriform, orbitofrontal, and insular cortices suggests that these regions can integrate multi
260 ed in monkeys composed of bilateral premotor cortices, supplementary motor area, and the right inferi
262 ns within the medial prefrontal and parietal cortices, temporoparietal junction, and anterior tempora
263 semantic dementia but included the cingulate cortices, thalami, and cerebellum in patients with bvFTD
265 At baseline, patients had diffusely thicker cortices than did healthy participants, and patients who
266 al microcircuits in noncanonical association cortices that contrast V1.SIGNIFICANCE STATEMENT Our vis
267 y increases in fMRI activation in both motor cortices that outlasted the stimulation period, as well
268 urons in the posterior parietal and premotor cortices that seem to implement and update such a multis
269 e evenly distributed in layer V of wild-type cortices, their distribution became a mosaic in Pcdh19 h
270 mporal parietal junction, and medial frontal cortices, there were large differences in neural respons
271 my in lateral occipital and ventral temporal cortices, these dimensions fall away progressively from
272 organization of projections from the sensory cortices to the claustrum, whereas frontal inputs are mo
273 ponses of insula and dorsolateral prefrontal cortices to the receipt of large monetary losses, and al
274 ation of descending projections from sensory cortices to the SC has garnered much attention; however,
275 ivity profiles downstream of primary sensory cortices, to investigate neural reorganization in blind
276 es on how macaque premotor and primary motor cortices transform sensory inputs into motor outputs.
278 red 6920 synapses in mouse motor and sensory cortices using three-dimensional electron microscopy.
279 the behavioral role of BLA inputs to sensory cortices, very little is known about the circuit organiz
281 ilability in the cingulate and orbitofrontal cortices was associated with the rate of social laughter
283 ter volume in visual primary and association cortices was significantly correlated with the extent of
284 coherence between auditory and sensorimotor cortices, was stronger in the second listening block.
285 ng primary somatosensory (S1) and motor (M1) cortices, we used convection-enhanced delivery of the vi
286 els of Fgf9, Fgf10, Fgfr2c, and Fgfr3b in S1 cortices were enhanced, and this was accompanied by exub
287 cipants, the IPS and dorsolateral prefrontal cortices were more active during the math task than the
288 ustrum neurons projecting to primary sensory cortices were scant and restricted in distribution acros
289 categories in the fusiform and orbitofrontal cortices were stereotypically biased and correlated with
290 , precuneus, and medial and lateral temporal cortices, were biophysically intact compared with those
292 al, superior temporal, and superior parietal cortices, when the subjects believed that the sisters we
293 nsular, primary somatosensory, and cingulate cortices, whereas hard task difficulty was represented i
294 pace engaged the medial and lateral parietal cortices, whereas self-projection in time engaged a wide
295 both primary Te1 and secondary Te2 auditory cortices, whereas, at late time intervals, memory proces
296 nlikely to be inherited from fronto-parietal cortices, which do not project to SCs, but may be comput
297 erirhinal (PRh) and posterior parietal (PPC) cortices, which seemingly provide visual and tactile obj
298 onnections to temporal, parietal and frontal cortices, while in contrast, the network was driven by b
299 in sulcal regions in prefrontal and temporal cortices, while negative relationships were seen mainly
300 partially collapsed skull; (2) thin cerebral cortices with subcortical calcifications; (3) macular sc
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