ライフサイエンスコーパス: corticocortical

1 ements; this may be the result of increasing corticocortical activity to compensate for striatal dysf

2 In the present study, we investigated the corticocortical afferent projections of one of these sub

3 sensory systems, central olfactory pathways, corticocortical and commissural connections, and pathway

4 rm two major types of long-range connections-corticocortical and cortico-subcortical.

5 ene expression leads to aberrantly motorized corticocortical and corticofugal output connectivity.

6 gyrus, and they suggest an important role of corticocortical and corticolimbic forward connections in

7 orphologically diverse cell groups that have corticocortical and corticosubcortical projections.

8 hypoconnectivity dominated, particularly for corticocortical and interhemispheric functional connecti

9 might be a general feature of long-distance corticocortical and thalamocortical circuits.

10 thalamic areas that are themselves linked by corticocortical and thalamocortical connections.

11 role that layer 3 pyramidal neurons play in corticocortical and thalamocortical connectivity, we hyp

12 order to determine the origin and extent of corticocortical and thalamocortical projections to layer

13 dense synaptic zone consisting of horizontal corticocortical and widespread layer VII projections, in

14 l extrinsic projection systems (commissural, corticocortical, and thalamocortical) in all AC areas.

15 ynapses suggests the involvement of DISC1 in corticocortical as well as thalamocortical connections.

16 We found lower membrane excitability of the corticocortical axons and normal intracortical gamma-ami

17 rior part of secondary motor cortex receives corticocortical axons from the rostral retrosplenial cor

18 amine, basal forebrain, thalamocortical, and corticocortical axons is mainly responsible for this reg

19 connections tend to be larger than those of corticocortical, but the projection foci are less dense.

20 ur classes of efferent neurons were studied: corticocortical (CC) neurons with ipsilateral projection

21 lamocortical (TC) synapses are stronger than corticocortical (CC) synapses.

22 mouse AC, defined by their output as either corticocortical (CCort) or corticocollicular (CCol).

23 dicating that corticothalamic and interareal corticocortical cells in the subgranular layers represen

24 erlap between corticothalamic and interareal corticocortical cells in the subgranular layers.

25 he structural and functional properties of a corticocortical circuit that could enable movement-relat

26 se findings establish an excitatory RSC-->M2 corticocortical circuit that engages diverse types of ex

27 , we demonstrate that distinct intrinsic and corticocortical circuitries arise from barrel and septal

28 Our work in a rodent model of corticocortical circuitry demonstrates that feedback pat

29 nt with abnormalities in thalamocortical and corticocortical circuitry, suggesting that disruption of

30 y a central role in both thalamocortical and corticocortical circuitry.

31 a model, we characterized the intrinsic and corticocortical circuits arising in the major propriocep

32 ct amyloid deposition suggests that specific corticocortical circuits express selective, but late, vu

33 ract tracing analyses of the organization of corticocortical circuits of identified layers of primary

34 uce differential patterns of intra-areal and corticocortical circuits.

35 By influencing network state, corticocortical communication from motor cortex may ensu

36 dulatory functions can be expected to act on corticocortical communication in addition to their actio

37 esults emphasize the importance of recurrent corticocortical communication in the maintenance of cons

38 The efficacy of spiking synchrony in corticocortical communication is poorly understood.

39 Long-range corticocortical communication may have important roles i

40 relays seem especially important to general corticocortical communication, and this challenges and e

41 halamo-cortical pathway that is critical for corticocortical communication.

42 der thalamic relays), thus serving a role in corticocortical communication.

43 her primates, is well positioned to regulate corticocortical communication.

44 cy range (30-100 Hz) are thought to subserve corticocortical communication.

45 from corticosubcortical to more predominant corticocortical communications in the human brain.

46 We corroborated our findings using a corticocortical computational model representing perturb

47 ignificant because PPC and AGm are linked by corticocortical connections and are both critical compon

48 eaved auditory processing modules related to corticocortical connections and embedded in the isofrequ

49 us may provide a substrate for plasticity in corticocortical connections and Schaffer collateral syna

50 to simple cells, and others have argued that corticocortical connections are likely to be important i

51 Direct corticocortical connections are often paralleled by tran

52 oanatomical studies have long indicated that corticocortical connections are organized in networks th

53 Ascending corticocortical connections from subgriseal neurons were

54 These data demonstrate that corticocortical connections in cat somatosensory cortex

55 it arises with the evolutionary expansion of corticocortical connections in primates, crossing the th

56 essor of Ctip2 and regulatory determinant of corticocortical connections in the developing cerebral c

57 Knowledge of AC corticocortical connections is modest other than in the

58 We studied the corticocortical connections of architectonically defined

59 ration of neurons that furnish glutamatergic corticocortical connections that subserve cognition.

60 siderable information is available about the corticocortical connections to the IPL, much less is kno

61 Most corticocortical connections were patchy, in a manner sug

62 udal AGm parallel their known differences in corticocortical connections, and represent another basis

63 that primarily affects intrahemispheric and corticocortical connections, and that places these two d

64 Despite the importance of corticocortical connections, few published studies have

65 In addition to this cascade of corticocortical connections, the auditory cortex receive

66 Because these cortical areas are linked by corticocortical connections, the present findings indica

67 thalamus and other ipsilateral and callosal corticocortical connections.

68 w that such communication is based on direct corticocortical connections.

69 eurodegenerative diseases that proceed along corticocortical connections.

70 s also a progressive shift in the pattern of corticocortical connections.

71 primate visual cortex have different sets of corticocortical connections.

72 complexes, but differed in their patterns of corticocortical connections.

73 -subcortical connectivity and short-distance corticocortical connections.

74 idate strengthened several corticolimbic and corticocortical connections.

75 rence in the proportion of cortex devoted to corticocortical connectivity across these orders.

76 owed that control subjects exhibited greater corticocortical connectivity among middle cingulate, pos

77 oncerning the existence of neural areas, for corticocortical connectivity among neural areas, and for

78 We first determined the extent of intrinsic corticocortical connectivity between the hand and the fa

79 se was accompanied by a decrease in backward corticocortical connectivity from frontal to parietal co

80 ons of tractography for analyzing interareal corticocortical connectivity in nonhuman primates and a

81 ming earlier reports of the patchy nature of corticocortical connectivity in the adult cat somatosens

82 on pattern to the functional organization of corticocortical connectivity in the mouse cortex.

83 rain magnetic resonance imaging, large-scale corticocortical connectivity was mapped from ages 12 to

84 ea-to-area serial or hierarchical pattern of corticocortical connectivity.

85 the size of layer 3 neurons-and whole-brain corticocortical connectivity.

86 erved a breakdown of subcortico-cortical and corticocortical connectivity.

87 that deficits in thalamocortical, as well as corticocortical, connectivity contribute to auditory dys

88 results are consistent with abnormalities in corticocortical, corticobasal ganglia, mesocortical dopa

89 tem cell-grafted rats demonstrated increased corticocortical, corticostriatal, corticothalamic and co

90 ontains subnetworks corresponding to classic corticocortical, corticosubcortical, and subcortico-subc

91 ygdalocortical communication and distributed corticocortical coupling across multiple functional brai

92 ith theoretical perspectives positing that a corticocortical "disconnection" partly explains cognitiv

93 However, the dynamics of corticocortical effective connectivity has never been di

94 ng rest predict the pattern and magnitude of corticocortical evoked potentials elicited within 500 ms

95 an be made for impairment of hippocampal and corticocortical excitatory pathways, but in general the

96 ping of the dendritic distributions of these corticocortical excitatory synapses onto CSPs in both ar

97 t, regular-spiking neurons, which are mainly corticocortical, exhibited sharp frequency tuning simila

98 We studied the influence of corticocortical feedback in alert monkeys using cortical

99 ts demonstrate the involvement of the direct corticocortical feedback pathway, providing temporally p

100 eep-layer cortical neurons that provide both corticocortical feedforward inputs (to area MT) and cort

101 ity to study the human cortical networks and corticocortical functional connectivity mediating arbitr

102 activity to reveal the cortical networks and corticocortical functional connectivity mediating visuom

103 ion of amygdala impacts amygdalocortical and corticocortical functional connectivity.

104 nt of the underlying cognitive processes and corticocortical functional connectivity.

105 ings inform our understanding of large-scale corticocortical influence as well as the interpretation

106 i may then provide a complementary route for corticocortical information flow.

107 simulations, reveal that the nonlinearity of corticocortical inhibition cancels the nonlinear excitat

108 tatory synaptic drive and actively processes corticocortical input during behavior.

109 work reveals patterns of thalamocortical and corticocortical input unique to the auditory cortex.

110 erlaps PLST, indicating that PLST receives a corticocortical input, either directly or indirectly, fr

111 idal neurons, and relatively weak long-range corticocortical inputs and outputs.

112 have studied the distribution of reciprocal corticocortical inputs to pyramidal cells and gamma-amin

113 These reciprocal corticocortical inputs to SI were concentrated in layer

114 been suggested as one possible mechanism for corticocortical interaction.

115 ral sentence reading and analyzed long-range corticocortical interactions between local neural activa

116 r, little is known about the dynamics of the corticocortical interactions implementing these rapid an

117 This suggests that corticocortical interactions, both within a hemisphere (

118 activity is mediated by mutually inhibitory corticocortical interactions.

119 oss layers and projection classes, including corticocortical/intratelencephalic neurons (reciprocally

120 Corticocortical labeling with 1, 1''-dioleyl-3, 3, 3'',

121 y motor cortex (M2), suggesting a functional corticocortical link from the RSC to M2 and thus a bridg

122 s neurochemical changes in the supragranular corticocortical network to which these areas belong.

123 ation by prolonged recruitment of long-range corticocortical networks.

124 Corticostriatal neurons in layer 5A and corticocortical neurons (callosal projection neurons sim

125 om M2 monosynaptically excited M2-projecting corticocortical neurons in the RSC, especially in the su

126 probability of feed-forward connections from corticocortical neurons to corticotectal neurons is appr

127 of layer V (CF5s), those of layer VI (CF6s), corticocortical neurons with ipsilateral projection (CCI

128 how that Cre-positive neurons are CT and not corticocortical or corticoclaustral types.

129 robability of monosynaptic connections among corticocortical or corticotectal cells.

130 l neurons is significantly higher than among corticocortical or corticotectal pyramidal neurons.

131 cause of this hypoactivity may be defective corticocortical or thalamocortical connections.

132 ly outnumbered white-dominant neurons in the corticocortical output layers 2/3, but the numbers of bl

133 e that opposite STDP-like effects induced by corticocortical PAS are associated with increased commun

134 s connecting frontal and parietal regions: a corticocortical pathway and a subcortical pathway.

135 unction, underscoring its role as a critical corticocortical pathway linking the medial prefrontal, c

136 When the direct corticocortical pathway was interrupted, secondary somat

137 Corticocortical pathways interconnect cortical areas ext

138 e understanding of the role of transthalamic corticocortical pathways is to determine the nature of t

139 ception by modulating corticosubcortical and corticocortical pathways.

140 messages carried through the well-documented corticocortical pathways.

141 inate multiple levels of thalamocortical and corticocortical processing to rapidly learn, and stably

142 frequency range consistent with distributed corticocortical processing, whereas responses to standar

143 al projection neurons (cCStrPNs) and crossed-corticocortical projection neurons (cCCPNs).

144 2 or Ctip2 can alter the axonal targeting of corticocortical projection neurons and cause them to pro

145 e monkey neocortical pyramidal neurons: long corticocortical projection neurons from superior tempora

146 the distribution of neurofilament protein in corticocortical projection neurons in areas V1, V2, V3,

147 more subtle abnormalities, with the largest corticocortical projection neurons of layer IIIc express

148 ex can be classified into two major classes: corticocortical projection neurons, which are concentrat

149 ted to other distinctive properties of these corticocortical projection neurons.

150 by modulating network oscillations in S1 via corticocortical projections and subcortical feedforward

151 paradigm to characterize the organization of corticocortical projections from primary somatosensory (

152 Corticocortical projections from the dorsal and ventral

153 supports the view that the release of latent corticocortical projections from tonic inhibition throug

154 sis (ALS), and the preferential loss of long corticocortical projections in Alzheimer's disease (AD).

155 esis, altered neuronal identity and aberrant corticocortical projections in the developing mouse brai

156 Corticocortical projections mainly terminated in the dor

157 mmediate neighboring barrel columns, whereas corticocortical projections reach the second somatosenso

158 Corticocortical projections targeted areas projected to

159 Septal corticocortical projections terminate in the dysgranular

160 may be a more important unifying feature for corticocortical projections than morphology.

161 Long-range corticocortical projections thus act through local micro

162 Corticocortical projections to the caudal and rostral ar

163 th the order observed in thalamocortical and corticocortical projections, and which characterizes all

164 understanding of these two components of the corticocortical projections, I studied the distribution

165 drive multisensory functions as strongly as corticocortical projections.

166 ant component in the evolution of long-range corticocortical projections.

167 an SI and subsequently via intrahemispheric (corticocortical) projections to the SI hand region.

168 We examined the corticocortical receptive field organization of resting-

169 connectivity following deafness may reflect corticocortical rewiring affording acoustically deprived

170 to interact with each other by multisynaptic corticocortical routes in strategy implementation.

171 e population has been suggested to determine corticocortical signaling efficacy, but others have argu

172 gamma remains unclear, and the influence on corticocortical signaling largely untested.

173 ough multiple downstream networks as in some corticocortical signaling schemes.

174 Do thalamocortical and corticocortical synapses differ in their plasticity and

175 se is required at thalamocortical but not at corticocortical synapses for building the whisker to bar

176 e explained either by synaptic depression in corticocortical synapses or by an inhibition-mediated su

177 The labeled corticocortical terminals in the primary motor (MI) and

178 mediodorsal thalamus (MDT) is a higher-order corticocortical thalamic nucleus involved in cognition a

179 emporal to basal forebrain structures versus corticocortical tract disconnection.

180 rum imaging to compare anterior-to-posterior corticocortical tracts between primates and other mammal

181 ell as an expansion of anterior-to-posterior corticocortical tracts compared with other mammals.

182 increased bursting may be more important in corticocortical transmission than in transmission of pri

183 entially reflecting a greater sensitivity of corticocortical versus thalamocortical projections to th

184 functional connectivity is likely driven by corticocortical white matter connections but with comple