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1 ive action of distinct subsets of unisensory corticofugal afferents, afferents whose sensory combinat
2 ed for the formation and maintenance of both corticofugal and intracortical pyramidal cell population
6 f Pax6 function on thalamocortical (TCA) and corticofugal axon (CFA) pathfinding during the period of
9 usion, both the recovery of motor skills and corticofugal axonal plasticity are promoted by intracere
10 ty, ascending thalamocortical and descending corticofugal axons first intermingle at the pallial-subp
11 docannabinoid action, to excessive growth of corticofugal axons into the sub-ventricular zone in vivo
13 ganglionic eminence-derived corridor and on corticofugal axons, but not on thalamocortical axons, an
14 fraction of small-to-medium-sized long-range corticofugal axons, which also emit collaterals that inn
20 conclusion, the tonotopic and non-tonotopic corticofugal connections of AI can potentially serve for
22 tions of the circuitry of the basal ganglia, corticofugal connections, topographic maps in sensory sy
24 B1 signaling is known to modulate long-range corticofugal connectivity, we analyzed the impact of THC
28 r colliculus receive differential degrees of corticofugal control; (5) cochleotopically organized are
29 track degeneration related to projection and corticofugal descending tracks associated with the right
32 uited for exploration of the function of the corticofugal (descending) system and the neural mechanis
38 It now appears that the connectivity of the corticofugal feedback pathway is also fundamentally link
43 ed disruption of the development of auditory corticofugal fibers may interfere with the ability of th
44 as the corpus callosum, anterior commissure, corticofugal fibers, lateral lemniscus, and cerebellar p
45 including the optic nerve, corpus callosum, corticofugal fibers, thalamocortical axons, lateral olfa
49 n the cerebral cortex itself, secondarily in corticofugal fibres and the subcortical targets with whi
50 ns indicate that strength is conveyed by the corticofugal fibres destined for the spinal cord, wherea
52 s confirm that selective disruption of motor corticofugal fibres influences functional reorganization
53 al pathway, the conduction velocities of the corticofugal fibres that mediate the responses were esti
61 transmission through the posterior thalamus, corticofugal input may be essential for normal processin
73 ecific effects can be explained by selective corticofugal modulation of individual olivocochlear effe
77 ateral and contralateral MEPs indicates that corticofugal motor fibres other than the fast-conducting
78 indicate that the widespread distribution of corticofugal motor projections may account for the favor
80 the timing of critical fate decisions during corticofugal neuron production and thus subtype-specific
81 ntrols the sequential generation of distinct corticofugal neuron subtypes by preventing premature eme
82 ies of the three principal sequentially born corticofugal neuron subtypes: subplate neurons, corticot
83 acquisition of cell fate for closely related corticofugal neurons and indicate that differential dosa
85 ion factor, Fezf2, is sufficient to generate corticofugal neurons from progenitors fated to become me
88 contralateral projection (CCCs), descending corticofugal neurons of layer V (CF5s), and those of lay
89 of efferent neurons were studied: descending corticofugal neurons of layer V (CF5s), those of layer V
90 ence of excitatory amino acids released from corticofugal neurons on dopaminergic activity in the bas
91 addition, experimental generation of either corticofugal neurons or callosal neurons below the corte
93 FIB is strongly expressed in radial glia and corticofugal neurons throughout cortical development.
95 in controlling the sequential generation of corticofugal neurons--SOX5 overexpression at late stages
98 aryal postsynaptic staining, suggesting that corticofugal output neurons may be modulated particularl
99 s 5 and 6) of primary sensory cortex provide corticofugal output to thalamus and they also project to
101 cortex by focal stimulation of the cerebral corticofugal pathway was investigated in anaesthetised r
102 onses evoked from different locations in the corticofugal pathway, the conduction velocities of the c
107 neurite inhibitory proteins, lesion-induced corticofugal plasticity is possible even in the adult ce
112 our understanding of the organization of the corticofugal projection in this critical brain region, w
113 losal projection neurons (CPNs) into induced corticofugal projection neurons (iCFuPNs) increases inhi
116 erentation is dependent upon both descending corticofugal projections and ascending trigeminothalamic
119 the mechanisms that control the guidance of corticofugal projections as they extend along different
121 d to quantify the contribution of descending corticofugal projections on (i) the normal organization
123 a connection to be reciprocal and documented corticofugal projections to the facial nucleus, surround
124 ugh considerable overlap characterized these corticofugal projections, a general topography was disce
132 unctions, but also a possible reciprocity of corticofugal speech and music tuning, providing neurophy
133 auditory system, however, indicates that the corticofugal system adjusts and improves auditory signal
135 hypothesis is that, during conditioning, the corticofugal system evokes subcortical BF shifts, which
138 Facilitation and inhibition evoked by the corticofugal system have been hypothesized to be respect
142 Recent studies have shown that the auditory corticofugal system modulates and improves signal proces
144 tric stimulation of cortical neurons via the corticofugal system modulates cochlear hair cells in a h
149 hat the collicular change is mediated by the corticofugal system; and that the IC itself can sustain
150 These data support previous reports that corticofugal systems work together with widespread later
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