ライフサイエンスコーパス: corticomedullary

1 was greater than that of oncocytomas in the corticomedullary (125 HU vs 106 HU, P = .045) and excret

2 s greater than that of papillary RCCs in the corticomedullary (125 HU vs 54 HU, P < .001), nephrograp

3 greater than that of chromophobe RCCs in the corticomedullary (125 HU vs 74 HU, P < .001) and excreto

4 On both the corticomedullary and nephrographic phase images, clear c

5 The tumor-to-cortex enhancement indexes at corticomedullary and nephrographic phases were largest f

6 nical MR protocol was used before and after (corticomedullary and nephrographic phases) intravenous a

7 ctal properties, resulting in a considerable corticomedullary area.

8 y localized to renal tubules situated at the corticomedullary boundary, consistent with the region of

9 n of Bowman's capsule followed temporally by corticomedullary cysts involving collecting ducts, proxi

10 tained before transplantation in donors, the corticomedullary difference was reduced in allografts (P

11 and bone bark), metaphyseal undulation, and corticomedullary differences were evaluated qualitativel

12 ureteral dilatation, pelvic dilatation, and corticomedullary differentiation were analyzed and compa

13 ion in thymus size, cellularity, and lack of corticomedullary differentiation.

14 stnatal sheep the irregularly shaped adrenal corticomedullary interface is innervated by corticotropi

15 eys in one fetus showed tubular cysts at the corticomedullary junction and diffuse effacement of the

16 itic cells and macrophages were found at the corticomedullary junction and in foci in the thymic cort

17 of the peritubular capillary network in the corticomedullary junction and increased numbers of bone

18 ng cells were found in the cortex and at the corticomedullary junction in both mouse strains.

19 revealed increased apoptotic cells near the corticomedullary junction in lymphoid follicles and in t

20 is a major cause of tubular necrosis in the corticomedullary junction of isolated perfused kidneys,

21 ed predominantly in intestinal villi and the corticomedullary junction of the kidney, whereas guanyli

22 Pericytes at the corticomedullary junction produce the S1P egress signal,

23 Most affected is the outer medullary or corticomedullary junction region where blood flow remain

24 , in situ apoptosis in the cortex and at the corticomedullary junction was responsible for a synchron

25 d kidney size, cysts are concentrated at the corticomedullary junction, and tubulointerstitial fibros

26 situ by thymic epithelium in the cortex and corticomedullary junction, two locations at which VCAM-1

27 ce revealed extensive ischemic injury at the corticomedullary junction, whereas kidneys from sham HO-

28 ure thymocytes exit via blood vessels at the corticomedullary junction.

29 in the cortex and is most abundant near the corticomedullary junction.

30 n the degree of interstitial fibrosis in the corticomedullary junction.

31 ed in tubular cells of the outer medulla and corticomedullary junction.

32 ed kidneys and on active reabsorption in the corticomedullary junction.

33 rring in 10.3 +/- 9.5% and 32.3 +/- 17.8% of corticomedullary-junction tubules by conventional light

34 the boundary between DP and SP thymocytes at corticomedullary junctions was disrupted and medullary s

35 the initiation of cytotoxic gene expression, corticomedullary migration, or thymus exit.

36 hasic protocol, which included non-contrast, corticomedullary, nephrographic and urographic phases.

37 tember 2007, the prenephrectomy multiphasic (corticomedullary, nephrographic, and excretory phases),

38 ar positions on images from the precontrast, corticomedullary, nephrographic, and excretory phases.

39 CT with as many as four phases (unenhanced, corticomedullary, nephrographic, and excretory).

40 revisiting the complex and poorly described corticomedullary organization of the thymus.

41 , Osp94 mRNA expression paralleled the known corticomedullary osmolality gradient showing highest exp

42 ephrographic phase compared with that in the corticomedullary phase (P = .0002 and P < .0001, respect

43 an in clear cell carcinomas, but only in the corticomedullary phase (P = .0231).

44 nal fat-suppressed T1-weighted gradient-echo corticomedullary phase acquisitions, obtained before and

45 was demonstrated in 11 neoplasms during the corticomedullary phase and in all neoplasms in the nephr

46 Signal intensity changes on corticomedullary phase images were the most effective pa

47 During the corticomedullary phase, ccRCCs with the loss of chromoso

48 relative contrast enhancement values in the corticomedullary phase.

49 86%, 86-88% and 74-78%, respectively, in the corticomedullary phase.

50 orresponded to "cyst attenuation" during the corticomedullary phase.

51 ypovascular tumors analyzed during the early corticomedullary phase.

52 lear cell RCCs and oncocytomas peaked in the corticomedullary phase; mean enhancement of papillary an

53 ode), during (helical mode, 25-second delay, corticomedullary-phase images), and after (axial mode, 1

54 We reveal that CTLA-4 is expressed in the corticomedullary region of the thymus.

55 improved injury scores in both cortical and corticomedullary regions.

56 lated in the perivascular channels of thymic corticomedullary venules.