ライフサイエンスコーパス: corticotrope

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1 ostral anterior pituitary gland (location of corticotropes).

2 anterior pituitary cells highly enriched in corticotropes.

3 nce of the resting membrane potential of rat corticotropes.

4 Ba(2+)-treated (77 +/- 15 spikes (5 min)-1) corticotropes.

5 ogranin processing in AtT-20 mouse pituitary corticotropes.

6 works governing the initial specification of corticotropes, a major component of this axis, are not f

7 ise to gonadotrope, thyrotrope, somatotrope, corticotrope and lactotrope cells in the anterior lobe a

8 Dll3 is expressed only in the presumptive corticotrope and melanotrope cells.

9 hat Notch signaling is sufficient to prevent corticotrope and melanotrope differentiation, resulting

10 adrenocorticotropin (ACTH) of pars distalis corticotropes and alpha-melanocyte-stimulating hormone (

11 cascade inhibits the differentiation of both corticotropes and melanotropes and results in the suppre

12 sary to repress premature differentiation of corticotropes and to promote proliferation of pituitary

13 pes, thyrotropes, somatotropes, lactotropes, corticotropes, and melanotropes-appear to be determined,

14 ssion system was used to construct an AtT-20 corticotrope cell line capable of inducible PAM-1 expres

15 te to the severe reduction in differentiated corticotrope cells and lower expression of the corticotr

16 xpressed in the anterior pituitary or AtT-20 corticotrope cells.

17 ch signaling must be suppressed in order for corticotrope differentiation to proceed and whether Notc

18 increased numbers of lactotropes and loss of corticotropes in the anterior pars distalis (APD), incre

19 ocorticotropic hormone (ACTH) secretion from corticotropes, inhibited IK(IR) by 25% and depolarized t

20 he activation of CRFR1 on anterior pituitary corticotropes, leading to the release of glucocorticoids

21 and survival, delineates the melanotrope and corticotrope lineage boundary, contributing to establish

22 n of the late (Pit1) lineage into the early (corticotrope) lineage.

23 ll specification, and an increased number of corticotropes, melanotropes, and gonadotropes in the pit

24 omelanocortin (POMC) gene, the anterior lobe corticotropes, producing adrenocorticotropin, and the in

25 her Notch signaling is sufficient to promote corticotrope proliferation, we examined the effects of p

26 ing, cell survival, and normal expression of corticotrope-specific transcription factors, which are n

27 Double mutants exhibit delayed corticotrope specification and complete failure of all o

28 RH-binding protein (CRH-BP) is secreted from corticotropes, the pituitary CRH target cells, suggestin

29 rticotrope cells and lower expression of the corticotrope transcription factors, TPIT and NEUROD1.

30 Corticotrope tumor cell lines expressing integral membra

31 When expressed in corticotrope tumor cells and primary anterior pituitary

32 homeostasis in neuroendocrine cells, we used corticotrope tumor cells in which AP-1 function was dimi

33 Overexpression of membrane PAM in corticotrope tumor cells reorganizes the actin cytoskele

34 Corticotrope tumor cells stably expressing P-CIP10 and P

35 was localized to the Golgi region of AtT-20 corticotrope tumor cells, and expression of integral mem

36 As in corticotrope tumor cells, expression of PAM or Myc-TMD/C

37 nules by stably expressing rat DBM in AtT-20 corticotrope tumor cells, which contain regulated granul

38 ant of PAM-1) was stably expressed in AtT-20 corticotrope tumor cells.

39 3A) was expressed in HEK293 cells and AtT-20 corticotrope tumor cells.

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