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1  constitutive low signaling by titrating out Costal.
2        The Drosophila kinesin-family protein Costal 2 (Cos2) and its mammalian ortholog Kif7 play dua
3 uding the role of the kinesin-family protein Costal 2 (Cos2), which directs Ci processing in Drosophi
4  with or without the kinesin-related protein Costal 2 (Cos2).
5 -threonine kinase Fused (Fu) and the kinesin Costal 2 (Cos2, also known as Cos), yet Fu does not have
6  Fused (Fu), Suppressor of fused (Sufu), and Costal-2 (Cos2) or the vertebrate homologs Kif27/Kif7.
7 plex containing Ci, the kinesin-like protein Costal-2 (Cos2), the serine-threonine kinase Fused (Fu),
8 lded and stabilized by the atypical kinesin, Costal-2 (Cos2).
9    Further our data show that loss of PKA or Costal-2 activity does not fully mimic Hh signaling, dem
10 erary border cells, was found to disrupt the costal-2 locus, indicating a role for Hedgehog signaling
11 es now show that the kinesin-related protein Costal-2 provides a physical link between the transmembr
12 ontaining Ci and the kinesin-related protein Costal-2 to bind microtubules.
13  find Sex-lethal in a complex with Fused and Costal-2, both downstream components of the pathway.
14 mponents, including the kinesin-like protein Costal-2, the serine-threonine kinase Fused, and Suppres
15 )), and with the microtubule-binding protein Costal-2.
16  patterns of response were identical for the costal and crural diaphragms.
17 rating Smoothened cytoplasmic tails activate Costal and Fused, driving the regulatory complex to its
18  be found in other tetrapods that breathe by costal aspiration and locomote with a lateral undulatory
19           Purpose To assess the incidence of costal cartilage (CC) fractures in whole-body computed t
20  29), prominent convexity of anterior rib or costal cartilage (n = 19), prominent asymmetric costal c
21 tal cartilage (n = 19), prominent asymmetric costal cartilage (n = 20), well-defined paracostal subcu
22 n addition, embryos with paternal UPD12 have costal cartilage defects and hypo-ossification of mesode
23 imal aesthetic outcomes and morbidity at the costal cartilage donor site.
24 all (< 1-cm) subcutaneous nodule adjacent to costal cartilage in five.
25 tilted" sternum in six; prominent asymmetric costal cartilage in four; bifid rib in one; and well-def
26                      Extensive subperiosteal costal cartilage resection and perichondrial sheath deta
27                  After exposing the deformed costal cartilages, a short chip was resected medially ad
28                                              Costal chondrocyte and mixture constructs were morpholog
29                                              Costal chondrocyte constructs produced almost 40 times m
30  between human NK cells and isolated porcine costal chondrocytes (PCC).
31 uman articular chondrocytes and immortalized costal chondrocytes (TC28 cells).
32 ondrocytes and immortalized NTPPPH-deficient costal chondrocytes (TC28 cells).
33       This study demonstrates the ability of costal chondrocytes to produce extracellular matrix that
34 linically relevant cell sources by comparing costal chondrocytes, dermal fibroblasts, a mixture of th
35 ects in collagen matrix protein secretion by costal chondrocytes.
36 consist of highly disorganised vertebrae and costal defects, are similar to those associated with the
37 ol I, fifteen normal subjects maintained the costal diaphragm at inferior/superior positions by full
38 fiber type proportions and FEV1, we obtained costal diaphragm biopsies on 40 subjects whose FEV1 rang
39                                              Costal diaphragm biopsies were taken from five patients
40 n microvascular PO2 (PO2m) within the medial costal diaphragm of control (C, n = 10) and emphysematou
41 c and sternohyoid muscles, as well as in the costal diaphragm.
42                                              Costal diaphragmatic biopsy samples were obtained from 7
43        We obtained biopsy specimens from the costal diaphragms of 14 brain-dead organ donors before o
44 odel where low signaling is initiated when a Costal inhibitory site on the Smoothened cytoplasmic tai
45 dition, alterations in transverse processes, costal joints, and zygapophyses were detected.
46 esence of splenomegaly > or = 5 cm below the costal margin (BCM) or thrombocytosis > or = 700 x 10(9)
47 s underestimated the distance from the right costal margin to the liver edge by only about 2.4 centim
48 alpable splenomegaly (>/=5 cm below the left costal margin), Eastern Cooperative Oncology Group perfo
49 f the ribs into the turtle shell negates the costal movements that effect lung ventilation in other a
50    Depiction of the undersurface of the long costal muscle slips of the diaphragm on supine plain rad
51                         On the CT scans, the costal muscle slips were clearly defined as bands or sma
52                                  SmoC mimics costal mutants.
53                          Muscle slips of the costal portion of the diaphragm were depicted in the rig
54  muscle were obtained from the anterolateral costal regions of the stimulated and inactive hemidiaphr
55 ce a regulatory complex that includes Fused, Costal, Suppressor of Fused and Cubitus interruptus.
56 ride length [10], and they inhibit effective costal ventilation [9, 11].

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