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1 ipheral transplantation tolerance induced by costimulation blockade.
2 tion of transplantation tolerance induced by costimulation blockade.
3 ss resist prolongation of skin allografts by costimulation blockade.
4 allograft survival was readily prolonged by costimulation blockade.
5 of sublethally irradiated mice treated with costimulation blockade.
6 es some of the survival advantage induced by costimulation blockade.
7 T cell apoptotic pathway, on the effects of costimulation blockade.
8 l for the induction of hyporesponsiveness by costimulation blockade.
9 opoietic chimerism, lymphocyte depletion and costimulation blockade.
10 lanted tissues when induced by coreceptor or costimulation blockade.
11 r saturation as a pharmacodynamic measure of costimulation blockade.
12 partial regimens, or a full regimen based on costimulation blockade.
13 tic and skin allografts in mice treated with costimulation blockade.
14 dramatically prolonged graft survival under costimulation blockade.
15 slet allograft survival in mice treated with costimulation blockade.
16 deficient mice show early graft loss despite costimulation blockade.
17 for long-term allograft survival induced by costimulation blockade.
18 skin allograft survival in mice treated with costimulation blockade.
19 ection driven by CD8(+) T cells resistant to costimulation blockade.
20 llograft survival in recipients treated with costimulation blockade.
21 graft survival was compared in untreated and costimulation blockade (500 microg anti-CD40L and 500 mi
22 - mice than in IFN-gamma+/+ mice, and T-cell costimulation blockade abrogated alloantigen-induced T-c
23 by treating mice with either B7-CD28 T-cell costimulation blockade alone or B7-CD28 T-cell costimula
24 -/-) mice treated with either B7-CD28 T-cell costimulation blockade alone or B7-CD28 T-cell costimula
25 we find that the full tolerance regimen, or costimulation blockade alone, specifically inhibits alre
28 neutralizing IL-4 in addition to CD40-CD154 costimulation blockade and CD8+ T-cell depletion prevent
29 ng protocol with ABT-737 in combination with costimulation blockade and low-dose cyclosporine A resul
30 cy may be a valid pharmacodynamic measure of costimulation blockade and provide the first direct clin
33 etermined the effect that CD28/CD40-directed costimulation blockade and sirolimus have on this diseas
35 gate the role of innate immunity, mice given costimulation blockade and skin allografts were coinject
36 te induction of transplantation tolerance by costimulation blockade and that IL-2/Idd3 is a critical
37 stem cell transplantation protocols based on costimulation blockade and tolerance induction may requi
39 ed strategy using a single dose of busulfan, costimulation blockade, and T cell-depleted donor bone m
42 t a donor-specific CD8 T cell response under costimulation blockade as well as for the graft to survi
43 ansion and combinatorial therapy with novel, costimulation blockade-based immunosuppression strategie
44 ed-efficacy as in young recipients employing costimulation blockade-based or T-cell depletion-based c
45 urmount these impediments, we have adapted a costimulation blockade-based protocol developed for soli
47 roduction may represent an important part of costimulation blockade-based strategies to promote allog
51 bed a nonirradiation-based regimen combining costimulation blockade, busulfan, and donor bone marrow
52 in chemically diabetic NOD mice treated with costimulation blockade but is prolonged further in NOD I
53 previously reported that continuous 24-month costimulation blockade by abatacept significantly slows
54 that TLR signaling abrogates the effects of costimulation blockade by preventing alloreactive CD8+ T
58 d depletion of CD4 and/or CD8 pos T cells or costimulation blockade can substitute for ALS and preser
59 ells (Tmem), particularly those resistant to costimulation blockade (CB), are a major barrier to tran
60 c process dependent on the level of residual costimulation blockade, CD4+ regulatory cells, and activ
63 stimulation blockade alone or B7-CD28 T-cell costimulation blockade combined with donor splenocyte tr
64 stimulation blockade alone or B7-CD28 T-cell costimulation blockade combined with donor splenocyte tr
65 th on IFN-gamma-deficient recipients despite costimulation blockade could be explained by the lack of
66 ly tolerized by either rapamycin or combined costimulation blockade (CTLA-4Ig plus anti-CD40L mAb).
68 tive preconditioning (low-dose busulfan) and costimulation blockade (CTLA4-Ig and anti-CD40L) to prod
70 ntribution of Tregs to immune suppression by costimulation blockade depends on the concentration of C
71 from diabetes by a short course of combined costimulation blockade, despite the continued diabetogen
72 mice with very severe diabetes treated with costimulation blockade did not reverse diabetes, showing
73 llograft-protective effects of CD40-directed costimulation blockade do not require sCD154 blockade, c
76 monoclonal antibody-mediated coreceptor and costimulation blockade enables long-term engraftment and
77 N-gamma+/+ and IFN-gamma-/- mice, and T-cell costimulation blockade enhanced alloantigen-induced T-ce
79 The data also suggest that B7-CD28 T-cell costimulation blockade exerts immunosuppressive actions
80 e almost completely protected from diabetes, costimulation blockade failed to prolong skin allograft
85 ne or rapamycin were compared as adjuncts to costimulation blockade in the murine BALB/c to C3H/He he
87 d transplantation, where treatments based on costimulation blockade, in particular CD40 ligand (CD40L
88 linated neoantigens and clinical efficacy of costimulation blockade indicate a general defect in main
89 asL is not required for the establishment of costimulation blockade induced hyporesponsiveness, but r
94 e role of Foxp3 regulatory T (Treg) cells in costimulation blockade-induced dominant tolerance to por
96 sis that NOD mice would also be resistant to costimulation blockade-induced rat xenograft tolerance.
97 nity and that autoimmunity and resistance to costimulation blockade-induced transplantation tolerance
102 of NOD mice to resist tolerance induction by costimulation blockade is mediated by both CD4+ and CD8+
104 ft survival in NOD.B6 Idd3 mice treated with costimulation blockade is prolonged compared with NOD mi
106 vival in (NOD x C57BL/6)F1 mice treated with costimulation blockade is short, suggesting a strong gen
107 cipients with anti-CD40 ligand and CTLA-4Ig (costimulation blockade) is a powerful promising albeit n
110 er the diagnosis of type 1 diabetes and that costimulation blockade may exert its beneficial therapeu
112 hypothesize that CD8(+) T cell "escape" from costimulation blockade might be a IL-15/IL-15R dependent
113 olerance, we hypothesized that "escape" from costimulation blockade might represent a CD8(+) and IL-1
117 erefore, besides direct inhibition of T-cell costimulation, blockade of B7/CD28 may facilitate induct
119 depleting mechanism of action and not merely costimulation blockade plays a substantial role in the t
120 .TNFR2-/- and B6.IL-12R-/- mice treated with costimulation blockade plus LPS also exhibited short ski
122 ovide insight into potential risks following costimulation blockade posed by chronic or latent viral
124 lycytidylic acid (TLR3) to mice treated with costimulation blockade prevents alloreactive CD8(+) T ce
130 ection, recipients treated with a CD28-CD154 costimulation blockade regimen achieved sustained insuli
133 resents a new and potent strategy to prevent costimulation blockade-resistant CD8(+) T cell-driven re
136 in cells of hemopoietic origin and that the costimulation blockade-resistant phenotype is dominant.
137 A-1-specific induction therapy to neutralize costimulation blockade-resistant populations of T cells
139 nfection, but, strikingly, failed to mediate costimulation blockade-resistant rejection after challen
141 he ability of donor-reactive Tmem to mediate costimulation blockade-resistant rejection during a reca
142 is study, we report that CD8(+) Th17 mediate costimulation blockade-resistant rejection in T-bet(-/-)
150 memory T (TM) cells have been implicated in costimulation blockade-resistant transplant rejection, d
153 r of NF-kappa B translocation, together with costimulation blockade, synergistically impairs memory T
154 y) total body irradiation and treatment with costimulation blockade, T-cell depletion, or rapamycin.
155 nd support further investigation of combined costimulation blockade targeting the CD28 and CD40 pathw
156 ration occurred in protected recipients, yet costimulation blockade temporarily blunted early T-cell
157 ival of skin allografts in mice treated with costimulation blockade through a CD8 T cell-dependent, M
158 treatment drastically blunts the ability of costimulation blockade to produce long-term engraftment.
159 cyclosporine therapy blocked the capacity of costimulation blockade to produce permanent engraftment,
160 rlying mechanisms, we studied the ability of costimulation blockade to prolong islet allograft surviv
161 ermanent engraftment, combined rapamycin and costimulation blockade treatment produced permanent engr
162 strain combination known to be refractory to costimulation blockade treatment, combined treatment wit
163 vented in the early posttransplant period by costimulation blockade using CD154 or anti-inducible cos
164 aft tolerance in mice induced through either costimulation blockade using CD154-specific antibody the
165 ipheral transplantation tolerance induced by costimulation blockade using donor-specific transfusion
166 depletion in (NOD x CBA)F1 mice treated with costimulation blockade was impaired compared with simila
168 cluding short-term depleting anti-CD4 mAb or costimulation blockade, would affect the disease progres
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