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1 line on vagal sensory nerve activity and the cough reflex.
2  the electrical threshold for initiating the cough reflex.
3  the airways and have been implicated in the cough reflex.
4  their primary function is regulation of the cough reflex.
5 irway sensory nerve activity and thereby the cough reflex.
6 ay be more important than sensitivity of the cough reflex.
7 .07-4.57], p = .03), and absence of a gag or cough reflex (3.41 [1.34-8.68], p = .01).
8 95% CI 1.19-6.01; p=0.0173; 1 point), absent cough reflex (4.16, 1.79-9.70; p=0.0009; 2 points), exte
9 tem, spine, or airway nerves can enhance the cough reflex, and can persist in the absence of the init
10  pathophysiology of an abnormally heightened cough reflex, and jeopardising development of improved t
11 ological control of an abnormally heightened cough reflex, and recommend ways to assess the effects o
12 was found to have regained corneal reflexes, cough reflex, and spontaneous respirations.
13        An effective cough requires an intact cough reflex as well as adequate respiratory muscle func
14 ur clinical characteristics (corneal reflex, cough reflex, best motor response, and oxygenation index
15 our variables (absent corneal reflex, absent cough reflex, extensor or absent motor response, and an
16 and chemical stimuli, which is suggestive of cough-reflex hyperresponsiveness.
17                                              Cough reflex hypersensitivity is thought to underlie the
18 irway hyperresponsiveness (AHR), bronchitis, cough reflex hypersensitivity, damage to the airways and
19 communication to the environment, diminished cough reflex, impaired mucociliary clearance, altered al
20 way sensory nerves and an enhancement of the cough reflex in conscious guinea pigs.
21 l sensory nerve activity in vitro and on the cough reflex in guinea pig challenge models.
22 companied by an increased sensitivity of the cough reflex is the most common symptom of inflammatory
23 receptors could mediate sensitisation of the cough reflex, leading to chronic cough.
24 (R-C) positive patients had a more sensitive cough reflex (P = .03) but similar esophageal reflux exp
25 g factors of an intrinsic abnormality of the cough reflex, rather than the cause.
26 flux and the presence of erosive disease and cough reflex sensitivity in unselected patients (ie, inc
27                             We conclude that cough reflex sensitivity is preserved after C-SCI, and t
28          In addition, all patients underwent cough reflex sensitivity testing to citric acid, and 66
29                                              Cough reflex sensitivity to capsaicin (concentration of
30 y age or sex and only weakly correlated with cough reflex sensitivity to capsaicin (log C5 r = -0.36;
31 705498 produced a significant improvement in cough reflex sensitivity to capsaicin at 2 hours and a b
32  and performed spirometry, sputum induction, cough reflex sensitivity to capsaicin, and 24-hour ambul
33 ts of cervical spinal cord injury (C-SCI) on cough reflex sensitivity, we measured responsiveness to
34                     Objective testing of the cough reflex using citric acid cough challenge tests did
35    C-fibre activation may thus sensitize the cough reflex via central mechanisms.
36 0 years, use of inotropes and absence of gag/cough reflexes were predictors of cardiac arrest.

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