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1 c antiporter capable of using protons as the counter ion.
2 one organic anion is coupled to efflux of a counter-ion.
3 mmonium had the same effect as sodium as the counter-ion.
4 iff base with anionic residues that form its counter-ion.
5 ere repulsion is dominated by the entropy of counter ions.
6 g transport, possibly by the co-migration of counter ions.
7 le hydrophobic tails, a neutral solvent, and counter ions.
8 terials are compensated by covalently bonded counter-ions.
9 s provide evidence that Cl- is the principal counter ion accompanying endosomal and Golgi compartment
13 The technique relies on divalent organic counter ions and solvent mixtures to drive the organizat
15 volves ion exchange (swapping of co-ions for counter-ions), and rarely occurs by counter-ion adsorpti
18 these complexes and assumes that release of counter-ions associated with the free DNA plays the domi
19 a detailed picture of kappaB DNA hydration, counter ion binding, and conformation in the absence of
22 s, with the electrostatic effects of Manning counter-ion condensation and explicit magnesium ions are
25 state and, moreover, that the nature of the counter-ion does not affect the overall fold of this qua
28 es: (a) addition to coordinating groups; (b) counter-ion exchange in charged frameworks; or, (c) host
30 K(+)-permeable cation channels that provide counter ions for Ca(2+) handling in intracellular stores
31 pparently no other anion can substitute as a counter-ion for free K+ -- and that K+ glutamate is requ
32 ly permeable to H+, and that Cl- serves as a counter-ion for H+ transport and likely helps to maintai
34 acrylate hydrogel by exchanging the divalent counter-ions in the superficial gel layer to monovalent
36 in SR, which in combination with the reduced counter-ion movement may lead to instability of Ca(2+) m
37 6 of cytoplasmic loop 3 (CL3) is proposed as counter-ion of Asp-835, Asp-836, or Glu-838 of NEG2 to p
38 cation (TRIC) channels on SR as an essential counter-ion permeability pathway associated with rapid C
40 ble, in cell free systems in the presence of counter-ions, revealing structures not predicted by poly
44 that by using pH buffer solutions containing counter-ions that are beyond a specific size, the sensor
45 ne into the catalytic pocket could provide a counter ion to promote ionization of the sulfhydryl grou
46 nserved basic residue that seems to act as a counter ion to the DXDD motif, enhancing the ability of
47 d facilitate coupling of the passive flow of counter ions to active transcellular transport, thereby
48 g this time so that an increased movement of counter ions via the paracellular pathway could have shu
49 nimisation performed with explicit water and counter ions, was used to identify residues participatin
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