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1 n and mass reduction would facilitate timely counteraction.
2 e multifunctional proteins to coordinate the counteractions.
4 e results provide evidence in support of the counteraction aspect of the urea:TMAO paradigm linking s
7 tterning in vertebrates is determined by the counteraction between the Sonic Hedgehog (Shh) and the G
8 antagonizes human tetherin and suggest that counteraction by O-Nefs may be suboptimal.IMPORTANCE Pre
11 orster resonance energy transfer to test the counteraction hypothesis of counterbalancing effects bet
13 etic parameters, supporting the premise that counteraction is a property of the solvent system and is
14 suggests that Mip130 activity without DmMyb counteraction may be responsible for the Dm-myb mutant l
15 tional standpoint, our understanding of this counteraction mechanism is hampered by the fact that exi
16 xamined factors that might determine whether counteraction occurs, namely different combinations of a
17 ulting in non-essential immune responses and counteraction of bacterial protective immune responses w
18 those found in M-Vpus and mediate efficient counteraction of both the long and short isoforms of thi
21 vivo findings reveal inhibition of MyD88 via counteraction of IL-1-mediated proteoglycan depletion.
22 MinD binding to the surface is controlled by counteraction of initiation and dissociation complexes;
24 o block both IL10 and PD-1 to strengthen the counteraction of T-cell immunosuppression and to enhance
27 -activated protein kinases p38 and JNK-1, 3) counteraction of the effects of CHG on TNFalpha producti
28 ll of these effects are consistent with TMAO counteraction of the effects of urea on LDH kinetic para
30 GDNF signaling in a feed-forward loop and/or counteraction of the inhibitory pathway regulated by BMP
31 t baboon cultures, suggesting less efficient counteraction of these responses by viral proteins in ma
34 otoxin-induced lethal shock suggest that the counteraction or neutralization of MIF may serve as an a
35 ptors and transmission of signals for proper counteractions through adaptor and effector molecules.
36 d the ISO-stimulated NKCC activity, and this counteraction was sensitive to the p38 mitogen-activated
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