ライフサイエンスコーパス: countertransport

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1 nse to EAAC1-mediated Na+ cotransport and K+ countertransport.

2 l competition between similar molecules, and countertransport.

3 ibrium exchange conditions while maintaining countertransport acceleration.

4 and they did not show the expected level of countertransport acceleration.

5 t NHA genes may contribute to sodium-lithium countertransport activity and salt homeostasis in humans

6 These are hallmarks of sodium-lithium countertransport activity, a highly heritable trait corr

7 These ion gradients drive co- and countertransport and are critical for establishing the m

8 Countertransport and reversibility studies demonstrate t

9 plasm is known to be indirectly linked to H+ countertransport and, hence, stromal pH and photosynthet

10 Uptake of 5-FTHF is characterized by countertransport because adding unlabeled 5-FTHF in the

11 wed that ORP5 and ORP8 could mediate PI4P/PS countertransport between the ER and the PM, thus deliver

12 ion and release as a consequence of the H(+) countertransport by VMAT that accompanies vesicular upta

13 the electrochemical gradient of the co- and countertransported ions.

14 current and the concentration of analyte and countertransporting ions and is found to correspond well

15 nosomes preloaded with tyrosine demonstrated countertransport of 10 microM [3H]tyrosine, indicating c

16 ort of a proton and three Na(+) ions and the countertransport of a K(+) ion.

17 e net flux and complete a transport cycle by countertransport of K+, ASCT-1 mediates only homo- and h

18 mic acid with Na+, the cycle is completed by countertransport of K+.

19 l glutamate transport is achieved by the co-/countertransport of Na(+) and other ions down their conc

20 exchangers catalyze the electrically silent countertransport of Na+ and H+, controlling the transmem

21 tamic acid with three Na(+) ions followed by countertransport of one K(+) ion.

22 of three sodium ions and one proton and the countertransport of one potassium ion.

23 hree sodium ions and one proton, followed by countertransport of potassium.

24 atp1-mediated uptake is energized by efflux (countertransport) of intracellular reduced glutathione (

25 ontrols studies are consistent with an anion countertransport process using a relay mechanism and a k

26 e becomes competitive toward F and Britton's countertransport property disappears with these activato

27 he K(+) ion, which is believed to serve as a countertransport substrate for SERT, was the most effect

28 This behavior may reflect a limiting countertransported substrate needed to promote turnover

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