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1 cognizes the genome of ssRNA viruses such as Coxsackievirus B.
2 not quantitatively altered by infection with coxsackievirus B.
3            The shared cellular receptor, the Coxsackievirus B-Adenovirus receptor (CAR), for the two
4 n addition, the identification of the common coxsackievirus B and adenovirus receptor has offered an
5 tion by vaccination supports the notion that coxsackievirus B and adenovirus vaccines may help reduce
6         Previous studies have suggested that coxsackievirus B (CVB) activates CD8(+) T cells in vivo,
7  adenovirus receptor (CAR) mediates entry of coxsackievirus B (CVB) and adenovirus (Ad).
8 al dissemination in released EMVs.IMPORTANCE Coxsackievirus B (CVB) can cause a number of life-threat
9                                              Coxsackievirus B (CVB) is a common cause of acute and ch
10                                              Coxsackievirus B (CVB) is a common enterovirus that can
11                                         Many coxsackievirus B (CVB) isolates bind to human decay-acce
12 ly shown that CTLs play conflicting roles in coxsackievirus B (CVB) myocarditis; they assist in contr
13 alized host factor that negatively regulates coxsackievirus B (CVB) replication through its control o
14 ental murine infections and in cell culture, coxsackievirus B (CVB) RNA can persist for weeks in the
15 eptors for poliovirus, human rhinovirus, and coxsackievirus B (CVB) serve to bind the viruses to targ
16                                              Coxsackievirus B (CVB), a member of the enterovirus fami
17 enteroviruses, including echovirus 11 (E11), coxsackievirus B (CVB), and enterovirus 71 (EV71), and t
18                 These viruses, which include coxsackievirus B (CVB), poliovirus (PV), and enterovirus
19 sed to assay the tissues for the presence of coxsackievirus B (CVB).
20 lasma gondii, herpes simplex virus (HSV), or coxsackievirus B (CVB).
21 sponses to candidate viral pathogens such as coxsackievirus B (CVB).
22                                              Coxsackieviruses B (CVBs) are etiological agents of huma
23                                              Coxsackievirus B infections are suspected environmental
24                                         Many coxsackievirus B isolates bind to human decay-accelerati
25                            pDC activation by Coxsackievirus B requires the presence of specific antiv
26 tive agents, and specifically identified the coxsackievirus B serogroup as the leading culprit.

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