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1 critical component in the immune response to Coxsackievirus infection.
2 hanisms of innate immune responses following coxsackievirus infection.
3 peroxynitrite in the host immune response to Coxsackievirus infection.
4 5 patients showed no evidence of intervening coxsackievirus infection.
5 0; P=.75), indicating no association between coxsackievirus infection and cardiac impairment.
6                                In studies of coxsackievirus infection and selenium deficiency in mice
7 extreme susceptibility of newborn infants to coxsackievirus infection and viral tropism for the CNS,
8  and adenovirus receptor, are susceptible to coxsackievirus infection, and can be used to predict ant
9 n conclusion, IFN-gamma protects from lethal coxsackievirus infection by activating macrophages in an
10                                              Coxsackievirus infection causes myocarditis and pancreat
11                                              Coxsackievirus infection causes severe pancreatitis and
12                                        Thus, coxsackievirus infections do not appear to initiate beta
13 hermore, a sex bias for severe sequelae from coxsackievirus infections has been observed in humans.
14                                              Coxsackievirus infections have been proposed as an envir
15                                     Although Coxsackievirus infection induces expression of inducible
16                         The host response to Coxsackievirus infection is complex, including T lymphoc
17            Dystrophin is also cleaved during Coxsackievirus infection of cultured myocytes and in inf
18  PABP is also proteolytically cleaved during coxsackievirus infection of HeLa cells.
19     Neonates are particularly susceptible to coxsackievirus infections of the central nervous system
20 These findings indicate that the timing of a coxsackievirus infection, rather than its simple presenc
21                        Serologic evidence of coxsackievirus infection was present in all children, wi

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