ライフサイエンスコーパス: cpm

1 cpm-1285 entered HL-60 tumor cells, bound Bcl-2 protein,

2 9 versus 18,141 +/- 334 and 38,919 +/- 1,021 cpm for clfA agr, coa agr, and the single agr mutant, re

3 8.19 to -1.43 [P=0.005]; from 100 cpm, -5.04 cpm; 95% confidence interval, -8.44 to -1.65 [P=0.004];

4 er relative to control islets (0.25 +/- 0.06 cpm/microunits).

5 interval, -8.19 to -1.43 [P=0.005]; from 100 cpm, -5.04 cpm; 95% confidence interval, -8.44 to -1.65

6 t compressions and rates closer to 85 to 100 cpm are significantly associated with improved survival

7 OR-dependent uptake of glucose (3467 +/- 102 cpm/mg protein in 95% oxygen versus 2100 +/- 112 cpm/mg

8 g/mL CTGF, and from 30+/-43 cpm to 429+/-102 cpm at 48 hours.

9 BALMs from IL-10(-/-) mice and 4082 +/- 1036 cpm for BALMs from CFTR(-/-) mice, but <200 cpm with BAL

10 mg protein in 95% oxygen versus 2100 +/- 112 cpm/mg protein in control).

11 and 54 dag compared to Namikonga (10 and 117 cpm respectively).

12 5I-mouse CSF-1 or human rCSF-1, and the 125I-cpm or human CSF-1 concentrations were measured in fetal

13 ere 2,238 cpm/microCi (82.8 cpm/MBq) and 129 cpm/microCi (4.8 cpm/MBq), respectively (reconstructed s

14 ymphocyte reaction (690+/-119 vs. 3850+/-148 cpm, P<0.01).

15 hionine to a specific activity of 2 x 10(15) cpm/mol.

16 13 cpm for control DCs versus 17,428 +/- 152 cpm for NB-treated DCs; P < 0.05).

17 of lactacystin-treated islets (0.52 +/- 0.16 cpm/microunits) was 2-fold greater relative to control i

18 ound of 50 cpm and the GAPDH signals of 1700 cpm were independent of stimulation with PDGF or serum.

19 incubated with perchlorate (15,184 +/- 1,173 cpm versus 2,837 +/- 187 cpm).

20 vivors and nonsurvivors, 90+/-17 and 79+/-18 cpm, respectively; P=0.0033).

21 cific activity (125I-QLSPYPFDL, 3.5 x 10(18) cpm per mole) we show that at a peptide concentration (5

22 e (15,184 +/- 1,173 cpm versus 2,837 +/- 187 cpm).

23 - 42.6 cpm; 14 at 100 microM, 101.4 +/- 35.2 cpm) compared to the enzyme control.

24 cpm for BALMs from CFTR(-/-) mice, but <200 cpm with BALMs from either type of +/+ mouse.

25 h anti-CD3 as a mitogen showed 9202 +/- 2107 cpm [(3)H]thymidine incorporation for BALMs from IL-10(-

26 Sensitivities of PET and SPECT were 2,238 cpm/microCi (82.8 cpm/MBq) and 129 cpm/microCi (4.8 cpm/

27 om 506+/-108 counts per minute to 2764+/-240 cpm by 50 ng/mL CTGF, and from 30+/-43 cpm to 429+/-102

28 alpha-dystroglycan to laminin (6955 +/- 250 cpm/well) was similar to that measured for merosin (7440

29 d cholesterol (6238 +/- 273 and 5128 +/- 277 cpm/ microg, respectively) failed to equilibrate, indica

30 c superoxide generation declined (3124+/-280 cpm/mg FA versus 5898+/-474 cpm/mg placebo; P=0.001).

31 y SPECT imaging (2.2 +/- 0.4 vs. 1.4 +/- 0.3 cpm/mm(2)/injected dose, respectively, P < 0.05).

32 e frequency was significantly greater than 3 cpm in all regions of the stomach.

33 re elevated in diabetic rats (74.8+/-8x10(3) cpm/10 mg protein) compared with controls (38.1+/-8x10(3

34 tein) compared with controls (38.1+/-8x10(3) cpm/10 mg protein; P<0.01).

35 ed tachygastria in 2 patients into regular 3-cpm slow waves.

36 induced by UV-EC (vehicle: 21,813 +/- 7,302 cpm; RA; 5,299 +/-635 cpm) (n = 3).

37 mutant (8,504 +/- 831 versus 18,141 +/- 334 cpm), significant residual fibrinogen binding capacity r

38 ng reduced system sensitivity (from 75 to 34 cpm/microCi).

39 peptide concentrations yielded a Vmax of 343 cpm/min/reaction for the wild-type peptide, and Vmax val

40 /-240 cpm by 50 ng/mL CTGF, and from 30+/-43 cpm to 429+/-102 cpm at 48 hours.

41 ined (3124+/-280 cpm/mg FA versus 5898+/-474 cpm/mg placebo; P=0.001).

42 al response (13 at 1 microM, 320.3 +/- 124.5 cpm; 13 at 50 microM, 308.8 +/- 139.9 cpm; 13 at 100 mu

43 - 139.9 cpm; 13 at 100 mu M, 446.0 +/- 153.5 cpm) and no response (14 at 1 microM, 104.9 +/- 52.6 cpm

44 sing benzoyl-gly-gly-gly as substrate (10(5) cpm, 5 mM).

45 HLECs were incubated with 30 nmol (5 x 10(5) cpm/ micromol) of HNE in 2 mL Krebs-Hansleit buffer for

46 consisting of 100 microg POPC LUV, 9 x 10(5)cpm (approximately 15 pmol) Dol-P-Man, 100 nmol YNPTSV,

47 The nonhomologous control background of 50 cpm and the GAPDH signals of 1700 cpm were independent o

48 with Ad-ARR(2)PB/hNIS was no more than 2,500 cpm, demonstrating the tissue specificity of this constr

49 /- 52.6 cpm; 14 at 50 microM, 103.3 +/- 42.6 cpm; 14 at 100 microM, 101.4 +/- 35.2 cpm) compared to t

50 no response (14 at 1 microM, 104.9 +/- 52.6 cpm; 14 at 50 microM, 103.3 +/- 42.6 cpm; 14 at 100 micr

51 ults show, 14C peaks were detected down to 6 cpm, which is approximately 20 times more sensitive than

52 osed to cardiac-profile cyclic stretch at 60 cpm continuously accumulated KDR mRNA in a transcription

53 Cardiac profile cyclic stretch at 60 cpm increased VEGF mRNA expression in a time- and magnit

54 ligosaccharides containing approximately 600 cpm/ng.

55 c mixed leukocyte reaction (33,508 +/- 1,613 cpm for control DCs versus 17,428 +/- 152 cpm for NB-tre

56 icle: 21,813 +/- 7,302 cpm; RA; 5,299 +/-635 cpm) (n = 3).

57 ns per minute (cpm), and 21.7% had rates <70 cpm.

58 ding to a signal from the c-fos probe of 700 cpm.

59 roCi (82.8 cpm/MBq) and 129 cpm/microCi (4.8 cpm/MBq), respectively (reconstructed spatial resolution

60 ified cholesterol = 280 +/- 37 and 458 +/- 8 cpm/ microg, respectively).

61 f PET and SPECT were 2,238 cpm/microCi (82.8 cpm/MBq) and 129 cpm/microCi (4.8 cpm/MBq), respectively

62 esidues and a specific activity of 7 x 10(8) cpm/hr per mg.

63 (absolute mean difference from 85 cpm, -4.81 cpm; 95% confidence interval, -8.19 to -1.43 [P=0.005];

64 nsurvivors (absolute mean difference from 85 cpm, -4.81 cpm; 95% confidence interval, -8.19 to -1.43

65 124.5 cpm; 13 at 50 microM, 308.8 +/- 139.9 cpm; 13 at 100 mu M, 446.0 +/- 153.5 cpm) and no respons

66 d high-specific-activity probes (2.6 x 10(9) cpm/microgram) with reduced radiolytic decay and a usefu

67 low conditions (mean pressure, 100 mm Hg, 90 cpm, approximately 200 mL/min) alter the concentration o

68 ek after delivery (676+/-223 versus 453+/-93 cpm/mg; P<0.02); this was associated with significantly

69 r to that measured for merosin (7440 +/- 970 cpm/well).

70 In contrast, cpm-1285 had little effect on normal human peripheral bl

71 Furthermore, cpm-1285 had in vivo activity in slowing human myeloid l

72 [3H]thymidine incorporation (cpm/microg protein) in the transplanted grafts at 7 days

73 for VLP] than for the control group (median cpm: 107,049 for PHA, 2,111 for Flu, and 2,068 for VLP).

74 bert) (733 and 1660 read counts per million (cpm)) at 45 dag and 54 dag compared to Namikonga (10 and

75 s calculated based on the counts per minute (cpm) bound.

76 iescence in the fundus, 3 cycles per minute (cpm) pacemaker activity in corpus and antrum, and a prox

77 closer to 85 to 100 compressions per minute (cpm) than nonsurvivors (absolute mean difference from 85

78 sion rates were <80 compressions per minute (cpm), and 21.7% had rates <70 cpm.

79 gens of more than 100 000 counts per minute (cpm).

80 ent HPV infection [median counts per minute (cpm): 72,849 for PHA, 1,241 for Flu, and 727 for VLP] th

81 lumes of PA (accelerometer count per minute, cpm) and moderate-vigorous intensity PA (MVPA), and the

82 cl-2 binding peptide, cell permeable moiety (cpm)-1285, was designed by chemically attaching a fatty

83 Liposomal radioactivity (cpm/mg) was greatly enhanced in the liver (11-fold) duri

84 mice (P <.05), as was the bound 125I-rmTPO (cpm) per million PLT (P <.05).