ライフサイエンスコーパス: cranial nerve palsy

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1 tral nervous system disorders and peripheral cranial nerve palsies.

2 f current neuro-imaging guidelines for third cranial nerve palsies.

3 .3%), conjunctival microvasculopathy (2.3%), cranial nerve palsies (2%), herpes zoster ophthalmicus (

4 There were more cranial nerve palsies (22 vs 0) in the endarterectomy gr

5 l symptoms were painful radiculitis (65.9%), cranial nerve palsy (43.4%), and headache (28.3%).

6 mours; abnormal gait and coordination (78%), cranial nerve palsies (52%), pyramidal signs (33%), head

7 days and up to 3 years; clinical outcome of cranial nerve palsy after PED placement; angiographic ev

8 Botulism manifests with cranial nerve palsies and flaccid paralysis in children

9 By excluding patients with third cranial nerve palsies and those with GCA, the incidence

10 ially avoiding the surgical complications of cranial nerve palsy and hematoma.

11 e composite outcome of death, stroke, MI, or cranial nerve palsy during the periprocedural period (OR

12 ly benign causes of third, fourth, and sixth cranial nerve palsies in children, but a study from a te

13 ) had local side effects that included third cranial nerve palsy in 6 (40%), orbital edema in 3 (20%)

14 lower incidence of myocardial infarction and cranial nerve palsy in patients undergoing stenting.

15 There was one event of cranial nerve palsy in the stenting group compared with

16 = 2), small cerebral infarction (n = 2), and cranial nerve palsy (n = 1).

17 lvement (lymphoma cells in the CSF [n = 23], cranial nerve palsy [n = 9], both features [n = 4]), rep

18 cedural MI (OR: 0.45; 95% CI: 0.27 to 0.75); cranial nerve palsy (OR: 0.07; 95% CI: 0.04 to 0.14); an

19 s in an atraumatic sample or the presence of cranial nerve palsy) status.

20 ed with lower rates of periprocedural MI and cranial nerve palsy than CEA.

21 f other causes for isolated fourth and sixth cranial nerve palsies was 4.7% (3/64).

22 in a nontraumatic sample or the presence of cranial nerve palsy) was present.

23 all SF-36 domains, but periprocedural MI or cranial nerve palsy were not.

24 is, etiology, work-up and treatment of third cranial nerve palsies, while incorporating information f

25 93.8%) completely recovered from symptoms of cranial nerve palsy within a median of 3.5 months.

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