ライフサイエンスコーパス: crassa

1 point mutation (RIP) of repetitive DNA in N. crassa.

2 cognate codons are used for initiation in N. crassa.

3 n in the model filamentous fungus Neurospora crassa.

4 r of lignocellulolytic gene expression in N. crassa.

5 growth in the filamentous fungus Neurospora crassa.

6 een characterized in detail using Neurospora crassa.

7 ation in the multicellular fungus Neurospora crassa.

8 emic species, Leavenworthia alabamica and L. crassa.

9 ulation in the filamentous fungus Neurospora crassa.

10 of self-compatibility in L. alabamica and L. crassa.

11 ol repetitive selfish elements in Neurospora crassa.

12 esponses in the filamentous fungi Neurospora crassa.

13 ging of genetically engineered strains of N. crassa.

14 ll RNAs in the filamentous fungus Neurospora crassa.

15 f4 against F. graminearum but not against N. crassa.

16 mplexes in the filamentous fungus Neurospora crassa.

17 tation of blue-light responses in Neurospora crassa.

18 49 SSRs of 963 SSR types in the genome of N. crassa.

19 r HP1 localization and DNA methylation in N. crassa.

20 molecular markers for genetic studies in N. crassa.

21 otein from the filamentous fungus Neurospora crassa.

22 pathway in the filamentous fungus Neurospora crassa.

23 nation in the filamentous fungus, Neurospora crassa.

24 se chromosomes between N. tetrasperma and N. crassa.

25 -1, are essential for female fertility in N. crassa.

26 K9 in both mouse embryonic stem cells and N. crassa.

27 unching the pheromone response pathway in N. crassa.

28 nd meiotic silencing and RNA silencing in N. crassa.

29 an RDRP associated with RNA silencing in N. crassa.

30 the specific mark for DNA methylation in N. crassa.

31 om cosmid libraries of the fungus Neurospora crassa.

32 ponent of the circadian system in Neurospora crassa.

33 nance of regular hyphal growth in Neurospora crassa.

34 tant that is used in circadian studies in N. crassa.

35 lulolytic gene expression and activity in N. crassa.

36 y initiation in the fungal model, Neurospora crassa.

37 and acts at the step of bilayer fusion in N. crassa.

38 ation of nitroethane catalyzed by Neurospora crassa 2-nitropropane dioxygenase was investigated by me

39 Catalytic turnover of Neurospora crassa 2-nitropropane dioxygenase with nitroethane as su

40 ating functional variation of proteins in N. crassa, 3) there are different levels of evolutionary fo

41 In Neurospora crassa, a circadian rhythm of conidiation (asexual spore

42 ast, the histone modifications in Neurospora crassa, a convenient model organism for multicellular eu

43 In Neurospora crassa, a eukaryotic model system for studying blue-ligh

44 ication in the filamentous fungus Neurospora crassa, a simple and experimentally amenable model syste

45 In Neurospora crassa, a single H3K9 methyltransferase complex, called

46 In Neurospora crassa, a transcription factor, WCC, activates the trans

47 that the minimal functional domain of the N. crassa AAP corresponded closely to the region that was m

48 ur understanding of the light response in N. crassa, about which the most is known, and will then jux

49 NcLPMO9C from Neurospora crassa acts both on cellulose and on non-cellulose beta-

50 log in the filamentous ascomycete Neurospora crassa affects the circadian clock output, yielding a pa

51 show that the powerful tools available in N. crassa allow for a comprehensive system level understand

52 -stage over the last few decades--Neurospora crassa and Aspergillus nidulans.

53 ulans, Aspergillus fumigatus, and Neurospora crassa and expressed the genes as secreted proteins with

54 We show that N. crassa and F. graminearum respond differently to MtDef4

55 pecific expression and editing in Neurospora crassa and F. verticillioides Furthermore,F. graminearum

56 s growth of the ascomycete fungi, Neurospora crassa and Fusarium graminearum, at micromolar concentra

57 Two species of Fungiidae corals, Ctenactis crassa and Herpolitha limax, displaying YBD-like lesions

58 3K27me3 in the filamentous fungus Neurospora crassa and in other Neurospora species.

59 changes in gating the photic response of N. crassa and indicate that LOV-LOV homo- or heterodimeriza

60 te fungi Fusarium graminearum and Neurospora crassa and induces accumulation of reactive oxygen speci

61 The DEAD-box proteins CYT-19 in Neurospora crassa and Mss116p in Saccharomyces cerevisiae are broad

62 The DEAD-box proteins CYT-19 in Neurospora crassa and Mss116p in Saccharomyces cerevisiae are gener

63 e functional predictions of novel genes in N.crassa and other filamentous ascomycete species.

64 However, the means by which N. crassa and other filamentous fungi sense the presence of

65 However, the means by which N. crassa and other filamentous fungi sense the presence of

66 In Neurospora crassa and other filamentous fungi, light-dependent-spec

67 equence information available for Neurospora crassa and other fungi has greatly facilitated evolution

68 owing the divergence of S. cerevisiae and N. crassa and provides insight into the evolution of kinase

69 In Neurospora crassa and Saccharomyces cerevisiae, efficient splicing

70 In Neurospora crassa and Saccharomyces cerevisiae, the latter function

71 We then analyzed N. crassa and Schizosaccharomyces pombe telomerase reconsti

72 Recent work in Neurospora crassa and Sclerotinia sclerotiorum has illuminated how

73 er (PTC) function was analyzed in Neurospora crassa and wheat germ translation extracts using the tra

74 lly inoculated with the mycelium (Neurospora crassa), and following the initial incubation period, th

75 ulator of protoperithecial development in N. crassa, and double mutants carrying deletions of both vi

76 witches in the filamentous fungus Neurospora crassa, and found that one activates and two repress gen

77 e role of MAP kinase signaling in Neurospora crassa, and to identify downstream target genes of the p

78 mately 35% of genes marked by H3K27me3 in N. crassa are also H3K27me3-marked in Neurospora discreta a

79 aromyces cerevisiae and CYT-19 of Neurospora crassa are ATP-dependent helicases that function as gene

80 that of CMLE from the eukaryotic Neurospora crassa are completely different from that of PpCMLE, ind

81 st eukaryotes, the centromeric regions of N. crassa are rich in sequences that are related to transpo

82 The Neurospora crassa arg-2 uORF encodes the 24-residue arginine attenu

83 iae GCN4, S. cerevisiae CPA1, and Neurospora crassa arg-2, regulation by uORFs controls expression in

84 ng an in vivo tethering system in Neurospora crassa Artificial recruitment of the H3K9 methyltransfer

85 Here, we use Neurospora crassa as a model filamentous fungus to interrogate the

86 We have developed N. crassa as a model system where we can dissect the comple

87 Using the cellulolytic fungus Neurospora crassa as a model, we identified a xylodextrin transport

88 show that the P. syringae is able to use N. crassa as a sole nutrient source.

89 uorin expression were obtained in Neurospora crassa, Aspergillus niger and Aspergillus awamori by cod

90 f LAD from the filamentous fungus Neurospora crassa at 2.6 A resolution.

91 ierarchy of initiation at start codons in N. crassa (AUG >> CUG > GUG > ACG > AUA approximately UUG >

92 ggesting a minor role for this G alpha in N. crassa biology.

93 In the lowly bread mould, Neurospora crassa, biomolecular reactions involving the white-colla

94 the growth defect characteristic of dim-5 N. crassa but did not fully rescue the gross DNA hypomethyl

95 ungi, such as the model eukaryote Neurospora crassa, but is absent from the genomes of baker's yeast

96 , is needed for entry of this defensin in N. crassa, but not in F. graminearum.

97 T80 pathway is not involved in meiosis in N. crassa, but rather regulates the formation of female rep

98 l for light-mediated responses in Neurospora crassa, but the molecular mechanisms underlying gene ind

99 issues in the microbial eukaryote Neurospora crassa by using a "reverse-ecology" population genomic a

100 ture of the ring of c subunits in Neurospora crassa by using data from the crystal structure of the h

101 strial scale enzymes in the model system, N. crassa, by removing the endogenous negative feedback reg

102 l timing of the robust circadian clock in N. crassa can be disrupted in the dark when maintained in a

103 titution of NSP ubiquitylation in Neurospora crassa cell extracts.

104 In the filamentous fungus Neurospora crassa, cell fusion occurs during asexual spore germinat

105 Reconstitution of the N. crassa cellodextrin transport system in Saccharomyces ce

106 These data suggest that the N. crassa cellodextrin transporters act as "transceptors" w

107 ntified, including 10 of the 23 predicted N. crassa cellulases.

108 Here we identified Neurospora crassa centromeric DNA by chromatin immunoprecipitation

109 K [Osmotically Sensitive-2 (OS-2)] by the N. crassa circadian clock allows anticipation and preparati

110 omprehensive dynamic model of the Neurospora crassa circadian clock that incorporates its key compone

111 In the Neurospora crassa circadian clock, a protein complex of frequency (

112 The Neurospora crassa circadian negative element FREQUENCY (FRQ) exempl

113 Predicted key features of the N. crassa clock system are a dynamically frustrated closed

114 ble RNA and protein profiling data on the N. crassa clock.

115 Neurospora crassa colonizes burnt grasslands and metabolizes both c

116 Neurospora crassa colonizes burnt grasslands in the wild and metabo

117 nserved between S. cerevisiae and Neurospora crassa compared with that which has evolved in A. nidula

118 dentified in one of the double mutants of N. crassa conferred resistance to both bafilomycin and conc

119 nit c of the vacuolar ATPase from Neurospora crassa conferred strong resistance to bafilomycin but li

120 The 3D-solution structure of Neurospora crassa Cu(6)-metallothionein (NcMT) polypeptide backbone

121 The Neurospora crassa CYT-18 protein is a mitochondrial tyrosyl-tRNA sy

122 Comparisons with previous N. crassa CYT-18 structures and a structural model of the A

123 howed that one of these proteins, Neurospora crassa CYT-18, binds group I introns by using both its N

124 The Neurospora crassa DEAD-box protein CYT-19 is a mitochondrial RNA ch

125 have generated mutant strains of Neurospora crassa defective in six subunits, C, H, a, c, c', and c'

126 e of MEI3, the only RAD51/DMC1 protein in N. crassa, demonstrating independence from the canonical ho

127 the amino terminus that was unique to the N. crassa DGAT2 protein.

128 Two forms of N. crassa DGAT2 were tested: the predicted full-length prot

129 osition equivalent to Phe(281) of Neurospora crassa DIM-5 or Phe(1205) of human G9a allows the enzyme

130 chromatin, "heterochromatin." In Neurospora crassa, DNA methylation depends on trimethylation of Lys

131 ree eukaryotic NR from the fungus Neurospora crassa, documenting that Moco is necessary and sufficien

132 rom a favored carbon source to cellulose, N. crassa dramatically up-regulates expression and secretio

133 rbon source such as sucrose to cellulose, N. crassa dramatically upregulates expression and secretion

134 Most if not all paralogs in N. crassa duplicated and diverged before the emergence of R

135 The fl (fluffy) gene of Neurospora crassa encodes a binuclear zinc cluster protein that reg

136 a tritici, Magnaporthe oryzae and Neurospora crassa, exhibited PAMP activity, inducing cell death in

137 ed from one of its natural hosts, Neurospora crassa, exists in a multimeric form and has the ability

138 Using N. crassa expressing the Ca(2+) reporter aequorin, MsDef1,

139 Notably, the only other NMO from Neurospora crassa for which biochemical evidence is available lacks

140 e hydrophobin EAS from the fungus Neurospora crassa forms functional amyloid fibrils called rodlets t

141 res the relative contributions of Neurospora crassa G alpha subunits, gna-1, gna-2, and gna-3, in dir

142 Mutation or deletion of the N. crassa gene encoding subunit c' did not completely elimi

143 The protein encoded by the N. crassa gene was longer than that of U. ramanniana.

144 nication and fusion in the fungus Neurospora crassa Genetically identical germinating spores of this

145 In the filamentous fungus Neurospora crassa, genetically identical asexual spores (germlings)

146 o initiation of this project, the Neurospora crassa genome assembly contained only 3 of the 14 telome

147 istributions of the SSRs in the sequenced N. crassa genome differ systematically between chromosomes

148 The analysis of the N. crassa genome sequence also reveals that RIP has impacte

149 l selfish DNA, an analysis of the Neurospora crassa genome sequence reveals a complete absence of int

150 H3K27me3 covers 6.8% of the N. crassa genome, encompassing 223 domains, including 774 g

151 hich account for 71% of total SSRs in the N. crassa genome, using a Poisson log-linear model.

152 In N. crassa, germinating asexual spores (germlings) of identi

153 Thus, N. crassa germlings undergoing chemotropic interactions rap

154 as identified from expression analysis of N. crassa grown on pure cellulose.

155 ssion data, the secretome associated with N. crassa growth on Miscanthus and cellulose was determined

156 N. crassa H3K27me3-marked genes are less conserved than unm

157 In the fungus Neurospora crassa, H3K9me3 and 5mC are catalyzed, respectively, by

158 We found that L. alabamica and L. crassa had high species-level genetic diversity (H(e)=0.

159 Unlike S. cerevisiae, N. crassa has a single isoform of the a subunit.

160 Neurospora crassa has been a model organism for the study of circad

161 Neurospora crassa has been for decades a principal model for filame

162 The filamentous fungus Neurospora crassa has been shown to be missing homologs of a number

163 Neurospora crassa has been utilized as a model organism for studyin

164 The eukaryotic filamentous fungus Neurospora crassa has proven to be a dependable model system for th

165 l other than the model ascomycete Neurospora crassa--has been neglected, leaving this type of questio

166 nisms in Aspergillus nidulans and Neurospora crassa have been intensively studied, leading to importa

167 nd function of DNA methylation in Neurospora crassa have led to a greater understanding of heterochro

168 Genetics studies of Neurospora crassa have revealed that a DNA methyltransferase (DIM-2

169 pombe and the filamentous fungus Neurospora crassa have served as important model systems for RNAi r

170 ponent of the quelling pathway in Neurospora crassa, have rapidly diverged in evolution at the amino

171 ted mutants of each of the four classical N. crassa HDAC genes and tested their effect on histone ace

172 A locus DNA methylation (DLDM) in Neurospora crassa Here we show that the conserved exonuclease ERI-1

173 tion experiments, a heterocomplex between N. crassa HET-C1 and PhcA was associated with phcA-induced

174 In the filamentous fungus Neurospora crassa, HET-C regulates a conserved programmed cell deat

175 We further evaluated the role of the N. crassa homolog of IME2, a kinase involved in initiation

176 urth component, Neurospora protein 55 (an N. crassa homolog of p55/RbAp48), is critical for H3K27me3

177 24 proteins, whereas CBH-2 depends on the N. crassa homolog of yeast Erv29p.

178 s able to attach and extensively colonize N. crassa hyphae, while an Escherichia coli control showed

179 stal structures of an enzyme from Neurospora crassa in the resting state and of a copper(II) dioxo in

180 ined by translating mRNAs in a homologous N. crassa in vitro translation system or in rabbit reticulo

181 on deletion of Puf4 in filamentous fungi (N. crassa) in contrast to the increase upon Puf3 deletion i

182 c screen of the ascomycete fungus Neurospora crassa, in which dynein is nonessential.

183 The biological clock of Neurospora crassa includes interconnected transcriptional and trans

184 Ectopic expression of phcA in N. crassa induced HI and cell death that was dependent on t

185 We show that osmotic stress in N. crassa induced the phosphorylation of a eukaryotic elong

186 ning microscopy that a LPMO (from Neurospora crassa) introduces carboxyl groups primarily in surface-

187 Neurospora crassa is a heterothallic filamentous fungus with two ma

188 The filamentous fungus Neurospora crassa is a model laboratory organism, but in nature is

189 The eukaryotic model organism Neurospora crassa is an excellent system to study evolution and bio

190 This hyphal type in Neurospora crassa is being used as a model for studies on hyphal se

191 The model filamentous fungus Neurospora crassa is capable of utilizing a variety of carbohydrate

192 Internalization of MtDef4 in N. crassa is energy-dependent and involves endocytosis.

193 romatin in the filamentous fungus Neurospora crassa is marked by cytosine methylation directed by tri

194 The fluffy (fl) gene of Neurospora crassa is required for asexual sporulation and encodes a

195 tion/translation feedback loop in Neurospora crassa is the protein FREQUENCY (FRQ), shown here shown

196 N. crassa is typically found on woody biomass and is common

197 A model filamentous fungus, Neurospora crassa, is a multinucleate system used to elucidate mole

198 Using a population of 110 wild N. crassa isolates, we investigated germling fusion between

199 ted a number of site-directed variants of N. crassa LAD that are capable of utilizing NADP(+) as cofa

200 ils of rosette leaves, has shown that the L. crassa LFY ortholog, LcrLFY, rescues most aspects of flo

201 showed that the splicing of some Neurospora crassa mitochondrial group I introns additionally requir

202 The bifunctional Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (CYT-18 pro

203 cture of a C-terminally truncated Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (CYT-18 pro

204 The Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (mtTyrRS; C

205 One such protein, the Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (TyrRS; CYT

206 Moreover, expression of Neurospora crassa MMM1, which naturally lacks a long N-terminal ext

207 A N. crassa mutant carrying deletions for both transporters i

208 Previously, we have shown that a N. crassa mutant carrying deletions of three beta-glucosida

209 Here, we show that an N. crassa mutant carrying deletions of two genes encoding e

210 the Schizosaccharomyces pombe and Neurospora crassa Nbp2p orthologues and the high conservation of th

211 rresponding dicer-like genes from Neurospora crassa [Ncdcl-1 (50.5%); Ncdcl-2 (38.0%)] and Magnaporth

212 ity of a fast fungal kinesin from Neurospora crassa (NcKin).

213 e have characterized an LPMO from Neurospora crassa (NcLPMO9C; also known as NCU02916 and NcGH61-3).

214 We demonstrate that an LPMO from Neurospora crassa, NcLPMO9C, indeed degrades various hemicelluloses

215 s, such as BarA and TorS; and the Neurospora crassa Nik-1 (Os-1) sensor that contains a tandem array

216 fied, which may implicate mitochondria in N. crassa nonself recognition and PCD.

217 CdCl2 was contacted with supernatants of N. crassa obtained after growth in urea-containing medium.

218 associated DNA (RAD) mapping for use with N. crassa oligonucleotide microarrays.

219 In this study, we show that the Neurospora crassa osmosensing MAPK pathway, essential for osmotic s

220 ing the model filamentous fungus, Neurospora crassa, our microfluidic system enabled direct visualiza

221 In Neurospora crassa, pairing of homologous DNA segments is monitored

222 The Neurospora crassa photoreceptor Vivid tunes blue-light responses an

223 The filamentous fungus Neurospora crassa played a central role in the development of twent

224 the nonreducing end product formed by an N. crassa PMO is a 4-ketoaldose.

225 alyses of purified QDE-1 polymerases from N. crassa (QDE-1(Ncr)) and related fungi, Thielavia terrest

226 hat the model cellulolytic fungus Neurospora crassa relies on a high-affinity cellodextrin transport

227 Using Neurospora crassa repeat-induced point mutation (RIP) as a model sy

228 how the model filamentous fungus Neurospora crassa responds to the three main cell wall polysacchari

229 the introns of the model organism Neurospora crassa revealed a different organization at the 3' end o

230 equence of the filamentous fungus Neurospora crassa reveals a gene number very much higher than those

231 three eukaryotic model organisms, Neurospora crassa, Saccharomyces cerevisiae, and Candida albicans,

232 The excellent fit of the N. crassa sequence to the E. hirae structure and the degree

233 of Schizosaccharomyces pombe and Neurospora crassa show that these types of enzymes are involved in

234 crystalline precipitates on the hyphae of N. crassa showed that the main elements present in the crys

235 mponent of the circadian clock in Neurospora crassa, shows daily cycles that are exquisitely sensitiv

236 Neurospora crassa sports features of heterochromatin found in highe

237 y in yeast, and we also characterized the N. crassa STE12 homolog pp-1.

238 cts derived from MacroD-deficient Neurospora crassa strain exhibit a major reduction in the ability t

239 isms (SNPs) between the reference Neurospora crassa strain Oak Ridge and the Mauriceville strain (FGS

240 ur algorithm on a real dataset of Neurospora crassa strains, using the genetic and geographic distanc

241 e restriction site polymorphisms from two N. crassa strains: Mauriceville and Oak Ridge.

242 erived from S. cerevisiae OXA1 or Neurospora crassa SU9, both coding for hydrophobic mitochondrial pr

243 d the related protein Cyt-19 from Neurospora crassa suggest that these proteins form a subclass of DE

244 Studies with the Neurospora crassa synthetase (CYT-18 protein) showed that splicing

245 analysis affirmed that the reconstituted N. crassa telomerase synthesizes TTAGGG repeats with high p

246 The unique N. crassa TER 5'-splice site sequence is evolutionarily con

247 the canonical 5'-splice site GUAUGU, the N. crassa TER intron contains a non-canonical 5'-splice sit

248 t molecular coevolution of LcrLFY and the L. crassa TFL1 ortholog, LcrTFL1, contributed to the evolut

249 al new PMO families in the fungus Neurospora crassa that are likely to be active on novel substrates.

250 ation by IME-2 of a cell death pathway in N. crassa that functions in concert with the VIB-1 cell dea

251 TER 3'-end cleavage mechanism in Neurospora crassa that is distinct from that found specifically in

252 mponent of the circadian clock of Neurospora crassa that regulates the abundance of its core transcri

253 In Neurospora crassa, the circadian clock generates daily rhythms in t

254 In Neurospora crassa, the frq, wc-1, and wc-2 genes encode components

255 m graminicola, the model organism Neurospora crassa, the human pathogen Sporothrix schenckii, and the

256 In the filamentous fungus Neurospora crassa, the IME2 homolog (ime-2) is not required for mei

257 In Neurospora crassa, the interactions between products of the frequen

258 Among natural accessions of Neurospora crassa, there is significant variation in clock phenotyp

259 ated to transposable elements; however, in N crassa these sequences have been heavily mutated.

260 In Neurospora crassa, three allelic specificities at the het-c locus a

261 ombe, and one filamentous fungus, Neurospora crassa-three species that arguably are not representativ

262 h wild-type and mutant strains of Neurospora crassa to gain insight into the role of heterochromatin

263 y studied circadian oscillator of Neurospora crassa to inactivation of the frq gene.

264 used the model filamentous fungus Neurospora crassa to search for uncharacterized transcription facto

265 he NPS6 ortholog from the saprobe Neurospora crassa to the Deltanps6 strain of C. heterostrophus rest

266 and constitutive heterochromatin, Neurospora crassa, to explore possible interactions between element

267 A screen of a N. crassa transcription factor deletion collection identifi

268 methods refined our understanding of the N. crassa transcriptional response to cellulose and demonst

269 Analyses of the 5'-leader regions in the N. crassa transcriptome revealed examples of highly conserv

270 Here, we show that in N. crassa, two cellodextrin transporters, CDT-1 and CDT-2,

271 is suggests that the widespread and basal N. crassa-type spliceosomal cleavage mechanism is more ance

272 meiosis in the filamentous fungus Neurospora crassa, unpaired genes are identified and silenced by a

273 In Neurospora crassa, unpaired genes are silenced by a mechanism calle

274 ecular Cell, Lee et al. show that Neuropsora crassa uses several Dicer-dependent and -independent pat

275 his system, derived from genes in Neurospora crassa, uses the transcriptional activator QF to induce

276 We initially identified TER from Neurospora crassa using a novel deep-sequencing-based approach, and

277 have successfully applied BiFC in Neurospora crassa using two genes involved in meiotic silencing by

278 Neurospora crassa utilizes DNA methylation to inhibit transcription

279 In Neurospora crassa, VIVID (VVD), a small LOV domain containing blue-

280 r L. alabamica was 0.065 vs 0.206 and for L. crassa was 0.084 vs 0.189).

281 2 gene encoding for the enzyme in Neurospora crassa was cloned, expressed in Escherichia coli, and th

282 The complex from Neurospora crassa was composed of Tob55-Sam50, Tob38-Sam35, and Tob

283 s that showed expression differences when N. crassa was cultured on ground Miscanthus stems as a sole

284 heterologous expression method in Neurospora crassa was developed as a step toward connecting regiose

285 earch, the urease-positive fungus Neurospora crassa was investigated for the biomineralization of cal

286 eotide (nt) SSRs, the most common SSRs in N. crassa, was significantly biased in exons.

287 k in the circadian model organism Neurospora crassa We show that, in a ras2-deficient strain, the per

288 p, H3K9me, and DNA methylation in Neurospora crassa, we built and tested mutants of the putative H3S1

289 nt in the model ascomycete fungus Neurospora crassa, we show that genetic diversity is maintained by

290 Different PMOs isolated from Neurospora crassa were found to generate oxidized cellodextrins mod

291 merly Mortierella) ramanniana and Neurospora crassa were introduced into maize using an embryo-enhanc

292 while maintaining light responsiveness in N. crassa when held in a steady metabolic state using biore

293 ly active in the meiotic cells of Neurospora crassa, where they evaluate the mutual identity of homol

294 nctional genomics resources available for N. crassa, which include a near-full genome deletion strain

295 is study, we employed LPMO9C from Neurospora crassa, which is active toward cellulose and soluble bet

296 n by a fungal TPP riboswitch from Neurospora crassa, which is mostly located in a large intron separa

297 Previous research with Leavenworthia crassa, which produces solitary flowers in the axils of

298 he most is known, and will then juxtapose N. crassa with A. nidulans, which, as will be described bel

299 1 (HDA1) mutant (hda-1) strain of Neurospora crassa with inactivated histone deacetylase 1.

300 cells of the model fungal system, Neurospora crassa, with droplet microfluidics and the use of a fluo