ライフサイエンスコーパス: creatine phosphate

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1 a-lipoic acid prevented the reduction in SCG creatine phosphate.

2 m, and increased the intracellular stores of creatine phosphate.

3 s a stimulatory role and processing requires creatine phosphate.

4 Creatine and creatine phosphate act as a buffer system for the regene

5 in p50 and attenuated the decline in cardiac creatine phosphate/adenosine triphosphate (PCr/ATP), per

6 onophosphate by the use of adenylate kinase, creatine phosphate, and creatine kinase in a single step

7 ATP, creatine phosphate, and lactate were measured in sciatic

8 in glucose, upper glycolytic intermediates, creatine phosphate, and the amino acids glutamine and se

9 by changes in whole-tissue concentrations of creatine phosphate ATP, or AMP, but they leave open the

10 showed reduced cardiac function and reduced creatine phosphate:ATP (16% reduction), but ssTnI TAC he

11 A higher creatine phosphate:ATP ratio in diabetic kidney cortices

12 mitochondrial capacity using the changes in creatine phosphate concentration ([PCr]) and pH as detec

13 city estimates on the kinetics of changes in creatine phosphate content ([PCr]) during recovery from

14 ely, high [MgATP]/low [MgADP] [10 mM MgATP + creatine phosphate (CP) + creatine kinase (CK)] or low [

15 In vivo LV creatine phosphate (CP) and adenosine triphosphate (ATP)

16 The effects of creatine phosphate (CP) and inorganic phosphate (Pi) on

17 ac muscles, the effects of ATP, ADP, Pi, and creatine phosphate (CP) on the rate of force redevelopme

18 reatine phosphokinase and ATPase activities, creatine phosphate (CP) was, surprisingly, found to be s

19 t of Pi on SR function was reversed by 10 mM creatine phosphate (CP).

20 In normals, LV myocardial creatine phosphate (CP)/ATP ratios were 2.10 +/- 0.10, 2

21 ce and decreased cytidine, uracil, fumarate, creatine phosphate, creatine, and choline.

22 By far, the creatine phosphate/creatine kinase (CP/CK) system, which

23 asma was inhibited by aspirin, ADP scavenger creatine phosphate/creative phosphokinase (CP/CPK), and

24 less than half its baseline value before the creatine phosphate (CrP) pool is 18% depleted.

25 ing mitochondrial metabolism and cytoplasmic creatine-phosphate energy stores, completing the packagi

26 ared with reported 31P NMR analyses of total creatine phosphate flux to estimate that each creatine p

27 ithin the intermembrane space in the form of creatine phosphate, (ii) is prevented by the outer mitoc

28 Release of creatine phosphate kinase (CPK) in the samples of corona

29 Myocardial ATP levels were normal, but creatine phosphate levels were 35% lower in LVH patients

30 reatine phosphate flux to estimate that each creatine phosphate molecule produced undergoes about 50

31 th changes in whole-tissue concentrations of creatine phosphate or adenine nucleotides; however, it d

32 cyclase, or by 3-isobutyl-1-methylxanthine, creatine phosphate, or creatine kinase.

33 The myocardial concentrations of ATP and creatine phosphate (PCr) and the rate of ATP synthesis t

34 ATP and creatine phosphate (PCr) are prime myocardial high-energ

35 The effect of creatine phosphate (PCr) on sarcoplasmic reticulum (SR)

36 on was also inhibited by the creatine kinase/creatine phosphate system that removes ADP from the chan

37 thickening while favoring higher myocardial creatine phosphate to creatine ratios (0.14 +/- 0.03 vs.

38 Myocardial creatine phosphate-to-ATP ratios were significantly lowe

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