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1 lyses of biomarkers is to adjust for urinary creatinine.
2 oagulation, incidence of encephalopathy, and creatinine.
3 ong with decreased levels of ngal and plasma creatinine.
4 timated glomerular filtration rate and lower creatinine.
5 d at earlier time points compared with serum creatinine.
6 se of 50% or more from preprocedural to peak creatinine.
7 ty when assessed by serum cystatin C than by creatinine.
8 /5.67 micromol/L and 39.7/33.9 micromol/mmol creatinine.
9 ed equation (r(2) = 74.6%) was GFR = 45.9 x (creatinine(-0.836) ) x (urea(-0.229) ) x (international
10 Neurological and renal impairments (serum creatinine, 0.87+/-0.20; median, 0.80; interquartile ran
11 pe 2 diabetes mellitus and renal impairment (creatinine 1.5-3 mg/dL) who were candidates for coronary
12 atinine, 140.3 mumol/L) and ABOc recipients (creatinine, 140.2 mumol/L) (P = 0.99), with no significa
13 Graft function was similar between ABOi (creatinine, 140.3 mumol/L) and ABOc recipients (creatini
14 had an increased risk for doubled levels of creatinine (1985 events; adjusted hazard ratio [HR], 1.2
22 that is produced from the co-condensation of creatinine and amino acids as meats cook at high tempera
27 est association with 12-month eGFR, and POD5 creatinine and creatinine reduction between POD1 and POD
33 in neutrophil levels and increases in serum creatinine and low-density lipoprotein cholesterol level
34 ot associated with obesity, and that urinary creatinine and osmolality may be colliders on the causal
35 rs) urinary concentrations were obtained for creatinine and phenol metabolites: enterolactone, genist
36 endpoint was the change of serum bilirubin, creatinine and serum BUN levels before and after the fir
38 e difference between baseline and peak serum creatinine and staged according to Kidney Disease Improv
39 rs, we compared ICU discharge cystatin C and creatinine and their association with 1-year mortality.
40 a rapid increase in circulating amino acids, creatinine and urea compared with breast-fed infants.
41 otected against mild renal IRI, with reduced creatinine and urea levels compared with wild type litte
42 after reperfusion for renal function (serum creatinine and urea), complement deposition (C3b/c and C
43 phometry and the utility of measuring plasma creatinine and urinary albumin, has been almost entirely
45 s (111 had AKI, defined as doubling of serum creatinine) and ascertained outcomes in the correspondin
46 toxicity (hemoglobin, leukocytes, platelets, creatinine), and immunohistochemical analysis of the kid
47 of >/=30% decline in eGFR, doubling of serum creatinine, and AKI; however, apixaban did not have a st
49 xygen extraction, a lower decrement of serum creatinine, and higher levels of NGAL and ET-1 were asso
50 e sex, current smoking, statin use, elevated creatinine, and higher lipids were associated with great
51 -TG mice had even greater increases in urea, creatinine, and KIM-1 levels and more tubular injury and
52 , Sequential Organ Failure Assessment score, creatinine, and PCO2 were associated with higher high-se
54 also had elevated blood levels of sodium and creatinine, and reduced levels of random glucose and hae
55 D-fibroblast growth factor 23 (FGF23) axis, creatinine, and renal tubular reabsorption of phosphate
59 re compared with other renal injury markers (creatinine, aspartate transaminase, and heart-type fatty
60 ucose at admission >10.6 mmol/l (191 mg/dl), creatinine at admission >132.6 mumol/l (1.5 mg/dl), Thro
61 ) as well as smaller mean increases in serum creatinine at week 48 (0.01 mg/dL [0.00-0.02] vs 0.03 mg
63 l hazards models to quantify associations of creatinine-based eGFR, urine albumin-to-creatinine ratio
64 sease (CKD) resulting from the inaccuracy of creatinine-based estimates of glomerular filtration rate
65 1), proadrenomedullin (beta=0.171; P<0.001), creatinine (beta=0.118; P=0.003), sodium (beta=0.101; P=
66 ultaneous analysis of immunosuppressants and creatinine can replace conventional venous sampling in d
68 eved Css,avg >/=2mg/L; but for patients with creatinine clearance >/=80mL/min target attainment was <
69 individuals with cardiovascular disease and creatinine clearance >30 ml/min) and examined post hoc t
70 n a non-dialysis day), >80% of patients with creatinine clearance <80mL/min achieved Css,avg >/=2mg/L
73 data from 214 adult critically-ill patients (creatinine clearance 0-236mL/min; 29 receiving renal rep
74 sed a transient rise in plasma Pi levels and creatinine clearance and an increase in phosphaturia wit
75 parathyroidectomized rats also led to higher creatinine clearance and lower plasma calcium levels but
79 5 times the upper limit of normal or less, a creatinine clearance of at least 30 mL/min, and a Karnof
85 nt-related concern, then patients with lower creatinine clearance values of > 30 mL/min should be inc
86 polycystic kidney disease (ADPKD; estimated creatinine clearance, >/=60 ml per minute), the vasopres
88 enal function criteria should enable liberal creatinine clearance, unless the investigational agent i
93 ake, ES rats still showed a lower mean serum creatinine concentration and less albuminuria, as well a
94 eatinine plasma concentration in mumol/L) = (creatinine concentration in DBS in mumol/L)/0.73, with a
95 ion rate, manifested by an increase in serum creatinine concentration or oliguria, and classified by
99 zard model adjusted for age, diabetes, serum creatinine concentration, urinary albumin concentration,
101 < 0.001) and presented with higher admission creatinine concentrations (1.21 +/- 0.09 vs 0.81 +/- 0.0
102 ined data on age, sex, height, weight, serum creatinine concentrations, and results for GFR from chro
103 ocioeconomic, lifestyle factors, and urinary creatinine concentrations, BPA, but not BPF or BPS, was
105 ge glomerular filtration rate estimated from creatinine consistently overestimated follow-up glomerul
106 ssociations of natural logarithm transformed creatinine-corrected urinary vanadium (Ln-vanadium) conc
107 or obesity-related outcomes, controlling for creatinine could induce collider stratification bias.
109 computed tomography (CT) and for whom serum creatinine (Cr) levels were obtained within 72 hours bef
114 L/min/1.73 m2, calculated using the combined creatinine-cystatin C CKD-Epidemiology Collaboration Equ
117 stopathologic findings, with increased serum creatinine detected only in the ReninAAV-treated db/db m
118 During ICU admission, serum cystatin C and creatinine diverged, so that by ICU discharge, almost tw
119 s (KDIGO grade 1: n = 3, grade 3: n = 1) but creatinine/eGFR returned to baseline values in all patie
120 I) equations, we compared GFR estimated from creatinine (eGFRcreat), cystatin C (eGFRcys), and both (
123 ndrome; and a composite of doubling of serum creatinine, ESRD, or death between 100 Rtx-treated patie
124 Levels of oxygen consumption, extraction, creatinine fall and fractional excretion of sodium were
127 re observed with lung function, haemoglobin, creatinine, glucose levels or resting blood pressure mea
128 pressure, body mass index, and cholesterol, creatinine, glucose, insulin, triglycerides, and urea le
129 l species (human serum albumin, neurotensin, creatinine, glycine, and alanine) were retained in the s
130 arette use, diabetes, hypertension, terminal creatinine greater than 1.5 mg/dL and AB blood type.
131 renal function, defined as a rise in plasma creatinine >/=26.5 mumol/l or 50% higher than the admiss
132 te <60 mL/min per 1.73 m(2) or urine albumin/creatinine >30 mg/g) and available echocardiogram-derive
134 r SGF definitions, postoperative day (POD) 7 creatinine had the strongest association with 12-month e
135 0.66 to 0.89; p < 0.001), doubling of serum creatinine (HR: 0.62; 95% CI: 0.40 to 0.95; p = 0.03), a
136 llows the fast and accurate determination of creatinine in real samples with minimal sample manipulat
137 of this novel sensor is tested by measuring creatinine in real urine samples (N=50) and comparing th
140 with acute kidney injury (defined by a serum creatinine increase during hospitalization > 0.3 mg/dL o
141 vents-syncope, pulmonary embolism, and serum creatinine increase-in 3 patients were determined to be
150 eletal muscle injury, evidenced by increased creatinine kinase and lactate dehydrogenase, as well as
153 gher resting heart rate, older age, elevated creatinine, larger left atrial volume index, and larger
155 as stage 2 or 3 acute kidney injury (plasma creatinine level >/=2 times the baseline level or urine
156 hylaxis without severe kidney disease (serum creatinine level >3 mg/dL; glomerular filtration rate <1
157 idney disease (OR, 2.05; 95% CI, 1.15-3.64), creatinine level (per SD increase; OR, 1.13; 95% CI, 1.0
159 acute kidney injury according to the plasma creatinine level alone failed to identify acute kidney i
160 ic kidney disease (CKD) in children, such as creatinine level and cystatin C-derived estimated glomer
163 O criteria and was based on changes in serum creatinine level from hospital days 0 to 2 through hospi
164 arameters were also associated with a raised creatinine level in the donor before organ retrieval.
165 n per cohort) had a mean (SD) baseline serum creatinine level of 1.0 (0.2) mg/dL and more than 20% ha
166 mal level, <142 U/L [2.37 mukat/L]), a serum creatinine level of 93 mumol/L (reference range, 79-125
168 hat GFR was significantly greater, and serum creatinine level was significantly lower in TRPC6 defici
169 io [OR], 1.644; P = 0.021), whereas elevated creatinine level was the only factor associated with adv
170 iltration rate (eGFR), doubling of the serum creatinine level, acute kidney injury (AKI), and kidney
171 STAT activity and resulted in reduced serum creatinine level, albuminuria, and renal histologic chan
173 lbuminuria, persistent doubling of the serum creatinine level, end-stage renal disease, or death due
174 ain (NFL), the ratio of N-acetylaspartate to creatinine levels (a magnetic resonance spectroscopy neu
176 oup had higher serum blood urea nitrogen and creatinine levels and a longer electrocardiographic QTc
177 statistically significant decreases in serum creatinine levels compared with levels in animals given
178 ) and with the ratio of N-acetylaspartate to creatinine levels in parietal gray matter (r = -0.352 an
179 ow BD induction, superoxide, MDA, and plasma creatinine levels increased further, whereas GPx activit
180 /=7 days) was estimated using serum urea and creatinine levels of 1,448 samples collected from 1,177
181 underlying kidney disease or abnormal serum creatinine levels on hospital days 0 to 2 were among tho
182 nexin 43+/-) had proteinuria, BUN, and serum creatinine levels significantly lower than those of wild
184 esulted in significant increases in urea and creatinine levels, a small (P<0.05) reduction in ejectio
185 avbeta5 antibody significantly reduced serum creatinine levels, diminished renal damage detected by h
187 presentation at time of index biopsy, serum creatinine levels/renal function over 24 months of follo
188 rence >94 (males) or >80 (females) cm, serum creatinine <1.2 mg/dL, and normoalbuminuria were randomi
189 accuracy when applied to subclinical cases (creatinine, <25% increase from baseline) or had minimal
192 n depending on the prevailing methodology of creatinine measurement, and used linear regression to mo
193 ontrol groups had similar prepregnancy serum creatinine measurements (0.70+/-0.20 versus 0.69+/-0.10
194 a retrospective analysis using the Stockholm creatinine measurements database, which contains informa
195 could be improved by replacing non-targeted creatinine measurements with a standard clinical creatin
196 simple formula-lactate dehydrogenase (U/L) x creatinine (mg/dL)/thrombocytes (10(9) cells per L)-term
197 n with high urinary FSH values (>11.5 mIU/mg creatinine [n = 69]) did not have a significantly differ
198 clinical pregnancy loss in women with higher creatinine- normalized concentrations of MEP, MBP, MEOHP
199 or frontal cortex correlated positively with creatinine-normalized Delta9-tetrahydrocannabinol (THC)
200 nship between cadmium exposure, evaluated by creatinine-normalized urinary cadmium concentration, and
201 1.88; 95% CI, 1.19-2.99), abnormal baseline creatinine (odds ratio, 2.48; 95% CI, 1.59-3.88), and in
202 graft outcomes were similar with mean 1-year creatinine of 1.03 +/- 0.45 versus 0.99 +/- 0.6 (P = 0.4
203 reserved with NEVKP demonstrated lower serum creatinine on days 1 to 7 (P < 0.05) and lower peak valu
205 was progression CKD, defined as doubling of creatinine or decrease in estimated glomerular filtratio
207 st-to-height ratio were observed when either creatinine or osmolality were used to standardize or as
208 lution, including standardization by urinary creatinine, osmolality, and flow rates, and inclusion of
210 tical concentrations of ATP, ADP, AMP, cAMP, creatinine phosphate and ATP:AMP ratio were increased by
212 0.71-0.76), giving the conversion formula: (creatinine plasma concentration in mumol/L) = (creatinin
213 cystatin C (Pnoninferiority < 0.0001), serum creatinine (Pnoninferiority = 0.0004), and measured glom
216 gorised into quartiles (Q; Q1: </=0.84 mug/g creatinine, Q2: 0.84-1.40 mug/g creatinine, Q3: 1.40-2.9
217 /=0.84 mug/g creatinine, Q2: 0.84-1.40 mug/g creatinine, Q3: 1.40-2.96 mug/g creatinine, Q4: >2.96 mu
218 4-1.40 mug/g creatinine, Q3: 1.40-2.96 mug/g creatinine, Q4: >2.96 mug/g creatinine, with the lowest
220 FR] <60 mL/min/1.73m2), albuminuria (albumin/creatinine ratio >/=3 mg/mmol), and proximal renal tubul
221 of patients with baseline urinary albumin-to-creatinine ratio >/=30 mg/g, urinary albumin-to-creatini
222 r 1.73 m(2)), and a 24-hour urine protein-to-creatinine ratio >/=800 mg/g to TGF-beta1 mAb (2-, 10-,
223 tubular dysfunction (retinol-binding protein/creatinine ratio >2.93mug/mmol and/or fractional phospha
227 hibited a substantial increase in albumin-to-creatinine ratio (ACR) and serum creatinine level and mo
228 s of creatinine-based eGFR, urine albumin-to-creatinine ratio (ACR), and dipstick proteinuria with th
229 and fourth quartiles of urinary endotrophin:creatinine ratio (ECR) were independently associated wit
230 7.23; P<0.001); >/=0.30 g/g urine protein-to-creatinine ratio (HR, 2.44; 95% CI, 1.47 to 4.09; P<0.00
231 n (P < 0.001), the increase of urine albumin creatinine ratio (P < 0.01), the fall in glomerular filt
233 clearance) was associated with urine albumin/creatinine ratio (UACR) post-HTx in a subgroup of patien
236 en urine albumin excretion (urine albumin-to-creatinine ratio [UACR]) and genomic regions harboring v
238 glomerular filtration rate, urine albumin-to-creatinine ratio and urine protein-to-creatinine ratio)
239 across categories of eGFR and urine albumin-creatinine ratio at baseline and across the 2 doses stud
240 retion, assessed according to the albumin-to-creatinine ratio calculated from three early-morning uri
241 atinine ratio >/=30 mg/g, urinary albumin-to-creatinine ratio decreased more with canagliflozin 100 m
243 agulated thromboembolism; a urine protein to creatinine ratio of less than 1; and measurable disease.
245 ogical measures FE and urinary metabolite-to-creatinine ratio was lower, but could be improved by rep
246 min-to-creatinine ratio and urine protein-to-creatinine ratio) did not (Rho = -0.222; -0.137; -0.070
247 emographics, baseline eGFR, urine albumin-to-creatinine ratio, comorbidity, and measures of mineral m
251 atients with AASK-N, with 24-hour protein-to-creatinine ratios (milligrams per milligram) ranging fro
252 onic renal injury, increased urinary albumin/creatinine ratios, and increased tissue oxidative stress
253 renal lesions, and measured urinary albumin/creatinine ratios, tissue oxidative stress levels, and A
254 ated CS-induced increases in urinary albumin/creatinine ratios, tissue oxidative stress levels, endot
257 values in the upper third of the albumin-to-creatinine ratios; 443 were randomly assigned in a place
258 with 12-month eGFR, and POD5 creatinine and creatinine reduction between POD1 and POD2 demonstrated
259 S-DGF to a definition that combines impaired creatinine reduction in the first 48 hours or greater th
262 0.004 compared with 0.041 +/- 0.004 mumol/g creatinine, respectively; P = 0.0009) and had 52% lower
263 .6 +/- 0.1 compared with 1.0 +/- 0.1 mumol/g creatinine, respectively; P = 0.002), 63% less hexadeute
264 +/- 0.02 compared with 0.13 +/- 0.02 mumol/g creatinine, respectively; P = 0.002), and 58% less d6-al
266 using a standardized definition -i.e., serum creatinine rise of >/=0.3 mg/dL (26.5 mcmol/L) or >/=50%
268 , bilirubin, international normalized ratio, creatinine scores), complete abstinence was independentl
269 ears with type 1 or type 2 diabetes, a serum creatinine (SCr) level of 1.3-3.3 mg/dl for women and 1.
273 ients in rate of decline was lower in plasma creatinine than in ALS functional rating scale-Revised (
274 etween plasma hemoglobin level and change in creatinine that varied by age (overall [R = 0.12; p < 0.
275 improved laboratory variables (bilirubin and creatinine), the short-term mortality (up to day 14) of
276 ing Pearson's correlation coefficient, serum creatinine-to-serum cystatin C ratio was found to be the
277 ulses or in case of return to baseline serum creatinine together with reduction of donor-specific HLA
278 doxyl sulfate, p-cresol sulfate, kynurenine, creatinine, urate) include two "drug" transporters of th
279 7 was associated with higher levels of serum creatinine, uric acid, calcium and lower urine pH level.
281 that included age, sex, and discharge serum creatinine value alone (integrated discrimination improv
283 older age, female sex, higher baseline serum creatinine value, albuminuria, greater severity of acute
284 with a marginal functional benefit in 1-year creatinine values (P = .044), with adjusted averages of
286 nts, 194 (2.3%) were excluded due to missing creatinine values, no or an incomplete coronary angiogra
287 e diagnosis of the index admission and serum creatinine values: 1) acute kidney injury, 2) pneumonia,
288 tients were excluded if their baseline serum creatinine was >1.2mg/dL or they were receiving renal re
290 ance as estimated from mass, age, and plasma creatinine was a significant predictor of BFI on the tot
293 ter onset of therapy-induced increased serum creatinine was not superior to standard care and resulte
294 nine and cystatin C levels, higher discharge creatinine was then associated with lower long-term mort
297 up glomerular filtration rate estimated from creatinine, whereas ICU discharge glomerular filtration
298 with other target molecules, such as urea or creatinine, while maintaining a low detection limit (0.0
299 ease (CKD) monitoring in primary care, serum creatinine with estimated glomerular filtration rate and
300 1.40-2.96 mug/g creatinine, Q4: >2.96 mug/g creatinine, with the lowest quartile set as reference) w
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