ライフサイエンスコーパス: crescentic glomerulonephritis

1 e as the pre-emptive treatment in abrogating crescentic glomerulonephritis.

2 kade by nutlin-3a ameliorated all aspects of crescentic glomerulonephritis.

3 izations were needed to induce a necrotizing/crescentic glomerulonephritis.

4 autoimmune glomerulonephritis or necrotizing/crescentic glomerulonephritis.

5 corticoids, renal function in a rat model of crescentic glomerulonephritis.

6 sion of crescentic lesions in a patient with crescentic glomerulonephritis.

7 iquely susceptible to experimentally induced crescentic glomerulonephritis.

8 ng the kidneys, where they cause necrotizing crescentic glomerulonephritis.

9 elerating glomerulosclerosis in experimental crescentic glomerulonephritis.

10 (PEC) are major constituents of crescents in crescentic glomerulonephritis.

11 oto (WKY) rat shows marked susceptibility to crescentic glomerulonephritis.

12 of renal injury in an experimental model of crescentic glomerulonephritis.

13 s approach is effective in the prevention of crescentic glomerulonephritis.

14 proaches may be worth investigating in human crescentic glomerulonephritis.

15 amined the effect of an antibody to VLA-1 in crescentic glomerulonephritis.

16 CR1 also promoted influx of DC precursors in crescentic glomerulonephritis, a DC-dependent aggressive

17 ronounced in females, which developed severe crescentic glomerulonephritis accompanied by heavy prote

18 microscopic polyangiitis or necrotizing and crescentic glomerulonephritis achieve remission with the

19 apsing glomerulopathy variant), pauci-immune crescentic glomerulonephritis, acute interstitial nephri

20 city of Th17 cells in a mouse model of acute crescentic glomerulonephritis and in a mouse chronic mod

21 dose of 1600 microg/kg induced pauci-immune crescentic glomerulonephritis and lung hemorrhage in all

22 400 microg/kg of myeloperoxidase resulted in crescentic glomerulonephritis and lung hemorrhage in all

23 Crescentic glomerulonephritis and lung hemorrhage were M

24 s model so that all animals reliably develop crescentic glomerulonephritis and lung hemorrhage.

25 f patients with pauci-immune necrotizing and crescentic glomerulonephritis and systemic small vessel

26 both glomerular and interstitial fibrosis in crescentic glomerulonephritis and that neutralization of

27 atment experiment, all rats developed florid crescentic glomerulonephritis, and control rats showed m

28 li were also noted in amyloidosis, diabetes, crescentic glomerulonephritis, and diffuse endocapillary

29 ome, idiopathic pauci-immune necrotizing and crescentic glomerulonephritis, and related and overlappi

30 With the emergence of necrotizing/crescentic glomerulonephritis, approximately 0.15% of re

31 microscopic polyangiitis and necrotizing and crescentic glomerulonephritis associated with antineutro

32 l, illustrate that TLR4 stimulation triggers crescentic glomerulonephritis by effects on both the ada

33 the role of SPARC in mediating experimental crescentic glomerulonephritis by inducing passive nephro

34 We hypothesized that the MDM2 would drive crescentic glomerulonephritis by NF-kappaB-dependent glo

35 onstrated to be involved in animal models of crescentic glomerulonephritis (cGN) and are potential ta

36 Crescentic glomerulonephritis (Crgn) is a complex diseas

37 Crescentic glomerulonephritis (Crgn) is a complex disord

38 ion is effective in reducing the severity of crescentic glomerulonephritis even when treatment is sta

39 Crescentic glomerulonephritis (GN) is a devastating dise

40 phages to progressive renal injury in murine crescentic glomerulonephritis (GN).

41 splenocytes developed severe necrotizing and crescentic glomerulonephritis, granulomatous inflammatio

42 Excluding pauci-immune necrotizing and crescentic glomerulonephritis, IgG4 immunohistochemistry

43 Moreover, in patients with crescentic glomerulonephritis, IL-26 is expressed by ren

44 early along the mouse GBM in vivo, eliciting crescentic glomerulonephritis in Fcgr2b(-/-) mice suscep

45 ty of collagen-induced arthritis in mice and crescentic glomerulonephritis in rats, in part by decrea

46 everal susceptibility loci, Crgn1-Crgn7, for crescentic glomerulonephritis in the Wistar Kyoto (WKY)

47 ndance resulting from experimentally induced crescentic glomerulonephritis in these rats.

48 The residual genetic susceptibility to crescentic glomerulonephritis in WKY.LCrgn1,2 rats assoc

49 ANCA-positive patients with necrotizing and crescentic glomerulonephritis, including 69 with evidenc

50 Crescentic glomerulonephritis is an important cause of h

51 he migration of leukocytes into glomeruli in crescentic glomerulonephritis is fundamental to pathogen

52 e's syndrome (GPS) develop severe autoimmune crescentic glomerulonephritis, kidney failure, and lung

53 murine anti-MPO Abs derived from Spontaneous Crescentic Glomerulonephritis/Kinjoh mice.

54 Concomitant crescentic glomerulonephritis led to an initial mistaken

55 that ANCA cause pauci-immune necrotizing and crescentic glomerulonephritis (NCGN) and systemic small

56 evidence that MPO-ANCA cause necrotizing and crescentic glomerulonephritis (NCGN) and vasculitis.

57 o ANCA-associated vasculitis and necrotizing crescentic glomerulonephritis (NCGN).

58 development of pauci-immune necrotizing and crescentic glomerulonephritis (NCGN).

59 kidney and lung basement membranes, neither crescentic glomerulonephritis nor alveolitis ensued, lik

60 WKY) rat strain, we have identified multiple crescentic glomerulonephritis QTL (Crgn) and positionall

61 or and found increased life span, suppressed crescentic glomerulonephritis, reduced spleen size, and

62 ly progressive glomerulonephritis/idiopathic crescentic glomerulonephritis (RPGN/ICG), IgA nephropath

63 plenocytes into immune-deficient mice caused crescentic glomerulonephritis that could be completely b

64 In these mice, crescentic glomerulonephritis was induced with a previou

65 control IgG developed focal necrotizing and crescentic glomerulonephritis with a paucity of glomerul