ライフサイエンスコーパス: cresyl violet

1 rat, stained with FJ and counterstained with cresyl violet.

2 ally for the dendritic marker MAP-2, or with cresyl violet.

3 of vermis were sectioned and stained with 1% cresyl violet.

4 s of 10-micron coronal sections stained with cresyl violet.

5 All brains were stained with Cresyl violet, a Nissl stain.

6 was assessed in brain sections stained with cresyl violet and animals with misplaced lesions were ex

7 Cresyl violet and dimethylaminoazobenzene are Raman labe

8 alog, PP3, were examined histologically with cresyl violet and iron stain to assess the degree of dam

9 P-32 staining) leading to neurodegeneration (cresyl violet and neuronal nuclei staining) associated w

10 , postfixed and examined histologically with cresyl violet and Perl's iron stain to assess the degree

11 al staining but extensive neurodegeneration (cresyl violet and silver staining) when evaluated 4 days

12 Cryosections of LG were stained with cresyl violet, and acinar cells and ductal epithelial ce

13 t on fixed tissues using stains for neurons (cresyl violet), astrocytes (GFAP), microglia (Iba1), glu

14 were stained with hematoxylin & eosin (H&E), Cresyl violet, Bielschowsky silver stain, or immunohisto

15 Brains were harvested and stained with cresyl violet, caspase-3, and TUNEL to detect morphologi

16 rin 343 (C343)-TiO(2) nanoparticles (NP) and Cresyl Violet (CV(+))-TiO(2) NP systems, using time-corr

17 y interaction of a benzo-phenoxazine ligand (Cresyl Violet, CV) with antiparallel and (3 + 1) hybrid

18 dded, hippocampal sections were stained with cresyl violet for Nissl substance and immunolabeled for

19 articles and the long wavelength fluorophore cresyl violet, has been used for the determination of co

20 pars reticulata (TH immunohistochemistry and Cresyl violet histochemistry) by 28 days after ischemia/

21 variety of methodologies: cytoarchitecture (cresyl violet), histochemistry (peanut agglutinin), immu

22 cts of these treatments were evaluated using cresyl violet (Nissl) staining.

23 g and nuclear morphology were assessed using Cresyl violet (Nissl) staining.

24 rfused, and brain sections were stained with cresyl violet or immunolabeled with NeuN (for neuronal c

25 nglion cells (flat preparations stained with cresyl violet or retrograde labeling with a neurotracer)

26 LC), cytochrome oxidase (CO) histochemistry, cresyl violet, or demonstration of TCAs by placement of

27 X-100 and CoQ10 causes the MLs lysis and the cresyl violet oxidation, obtaining a decrease in the flu

28 er sensitized trans-cis isomerization, using cresyl violet perchlorate as the sensitizer, also led to

29 ion technique using fiducial markers such as cresyl violet, Ponceau S, and bromophenol blue that poss

30 Retinas were stained with cresyl violet, retinal cell-specific markers, and a huma

31 Cresyl violet stain demonstrated massive loss of neurons

32 Counting of cresyl violet stained sections showed an apparent increa

33 sing an image analysis system (BioQuant) and cresyl violet stained sequential sections from the foreb

34 ions were evaluated for area of tissue loss (Cresyl-violet stained sections) and the number of GFAP i

35 ts of two adult rat spinal cords on adjacent cresyl violet-stained and in situ hybridization sections

36 Molecular fingerprint comparison of cresyl violet-stained CA1 and CA3 pyramidal neurons micr

37 on cells were counted by light microscopy in cresyl violet-stained retina sections, and the percentag

38 es immediately after I/R injury and counting cresyl violet-stained retinal ganglion cell layer cells

39 Using still images from cresyl-violet-stained material, we present an adaptation

40 For both methods, Arc cresyl violet staining (cell density) and NPY and Y1 rec

41 Brain tissue loss determined using cresyl violet staining and astrocyte hypertrophy and pro

42 brains of Tg and Wt mice as assessed by both Cresyl violet staining and by TUNEL staining for DNA fra

43 Neuronal damage, as assessed by cresyl violet staining and DNA fragmentation analysis, w

44 -induced neurodegeneration, as visualized by cresyl violet staining and quantified in 20 serially sta

45 developed a modified alcohol-based, buffered cresyl violet staining protocol that provides reproducib

46 n the present study, immunohistochemical and Cresyl violet staining showed that the noradrenergic neu

47 neuronal death using silver impregnation and cresyl violet staining.

48 30 and later at > or =6 months of age using cresyl violet, Timm, and rapid Golgi staining and immuno

49 Cells were also stained with cresyl violet to detect morphological changes characteri