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1                                   The use of crevicular AST activity to monitor periodontal disease p
2 udy examined conditions under which gingival crevicular blood (GCB) could be used to obtain a useful
3 l diabetes screening approach using gingival crevicular blood (GCB) could be used to test for hemoglo
4 GCF), a marker of neutrophil influx into the crevicular environment, has previously been shown to be
5 ociation with plaque index (PI) and gingival crevicular fluid (GCF) after OHR for 1 week.
6 le is known about MMP reductions in gingival crevicular fluid (GCF) after therapy.
7 and local inflammatory mediators in gingival crevicular fluid (GCF) among patients with untreated chr
8  testing, histological examination, gingival crevicular fluid (GCF) analysis for the levels of C-telo
9 at in chronic periodontitis (CP) by gingival crevicular fluid (GCF) analysis of myeloperoxidase (MPO)
10 eri-implant crevicular fluid (PICF)/gingival crevicular fluid (GCF) and a selection of subgingival/su
11                      At each visit, gingival crevicular fluid (GCF) and blood were collected for dete
12 pecific FN fragments are present in gingival crevicular fluid (GCF) and can be used as markers for pe
13 ere to delineate cyto/chemokines in gingival crevicular fluid (GCF) and evaluate systemic levels of e
14 evels of stress-related hormones in gingival crevicular fluid (GCF) and extent/severity of periodonta
15     PAF levels were investigated in gingival crevicular fluid (GCF) and gingival tissue (GT) samples
16    LTB4 levels were investigated in gingival crevicular fluid (GCF) and gingival tissue (GT) samples
17                                 The gingival crevicular fluid (GCF) and monocytic secretion of prosta
18  surgery on growth factor levels in gingival crevicular fluid (GCF) and periodontal healing.
19 the present study is to investigate gingival crevicular fluid (GCF) and plasma acute-phase cytokines,
20 s study is to investigate levels of gingival crevicular fluid (GCF) and plasma TGM-2 and oxidative st
21 nitial periodontal treatment on the gingival crevicular fluid (GCF) and salivary levels of 8-hydroxy-
22 ng pregnancy on periodontal health, gingival crevicular fluid (GCF) and serum cytokines, and pregnanc
23 This study is performed to evaluate gingival crevicular fluid (GCF) and serum levels of soluble recep
24  evaluate YKL-40 and IL-6 levels in gingival crevicular fluid (GCF) and serum of patients with period
25 nti-inflammatory cytokine levels in gingival crevicular fluid (GCF) and serum of rheumatoid arthritis
26 mor necrosis factor (TNF)-alpha, in gingival crevicular fluid (GCF) and serum samples between women w
27 idative stress index (OSI) in their gingival crevicular fluid (GCF) and serum were recorded.
28                                     Gingival crevicular fluid (GCF) and subgingival plaque were colle
29 growth factor-beta 1 (TGF-beta1) in gingival crevicular fluid (GCF) and the periodontal status of sub
30 ith apoptosis in serum, saliva, and gingival crevicular fluid (GCF) and their association with period
31 is-specific molecular signatures in gingival crevicular fluid (GCF) and to confirm clinical diagnoses
32 s and RANKL/OPG ratios in serum and gingival crevicular fluid (GCF) are investigated in patients with
33           Volumetric alterations in gingival crevicular fluid (GCF) are widely accepted to be associa
34 implant crevicular fluid (PICF) and gingival crevicular fluid (GCF) as surrogate measures of immune f
35 tudy is to determine whether either gingival crevicular fluid (GCF) bacterial gingivain/gingipain or
36 his study were to measure levels of gingival crevicular fluid (GCF) biomarkers and subgingival bacter
37                                     Gingival crevicular fluid (GCF) biomarkers associated with bone r
38  study are to: 1) determine whether gingival crevicular fluid (GCF) chemerin is a novel predictive ma
39 n unbiased metabolomic profiling of gingival crevicular fluid (GCF) collected from healthy, gingiviti
40 Aa), Tannerella forsythia (Tf), and gingival crevicular fluid (GCF) concentration of interleukin (IL)
41 this study was to compare serum and gingival crevicular fluid (GCF) concentrations of sICAM-1 in smok
42 ue, suggesting that their levels in gingival crevicular fluid (GCF) could be increased at gingivitis
43     It may be hypothesized that the gingival crevicular fluid (GCF) cytokine profile in patients with
44 l periodontal therapy (NSPT) on the gingival crevicular fluid (GCF) cytokine profile in sites with st
45 he study are to determine serum and gingival crevicular fluid (GCF) endocan levels in the pathogenesi
46                                     Gingival crevicular fluid (GCF) flow was significantly lower in t
47 F) samples from the new implant and gingival crevicular fluid (GCF) from adjacent teeth were sampled
48 ence of Th1-associated cytokines in gingival crevicular fluid (GCF) from LagP patients and the high l
49 -derived growth factor (PDGF-BB) in gingival crevicular fluid (GCF) from localized intrabony defects
50 ingival fibroblasts in vitro and in gingival crevicular fluid (GCF) in a randomized controlled split-
51 mpare the levels of resistin in the gingival crevicular fluid (GCF) in health, chronic periodontitis
52 IL)-11 and IL-17 and their ratio in gingival crevicular fluid (GCF) in patients with AgP.
53 al parameters of disease and on the gingival crevicular fluid (GCF) inflammatory mediator interleukin
54  of eight oral pathogens, levels of gingival crevicular fluid (GCF) interleukin-1beta (IL-1beta), pro
55 clinical periodontal parameters and gingival crevicular fluid (GCF) levels of 15-epi-lipoxin A4 (15-e
56 IgG) titers to 17 bacteria, and the gingival crevicular fluid (GCF) levels of 16 inflammatory mediato
57        The current study determines gingival crevicular fluid (GCF) levels of fibrosis-related IL-6-t
58 e of this study was to test whether gingival crevicular fluid (GCF) levels of IL-1beta and tumor necr
59                                     Gingival crevicular fluid (GCF) levels of interleukin (IL)-1beta
60 clinical periodontal parameters and gingival crevicular fluid (GCF) levels of interleukin (IL)-1beta,
61                  Serum, saliva, and gingival crevicular fluid (GCF) levels of interleukin (IL)-6, IL-
62 her glycemic control was related to gingival crevicular fluid (GCF) levels of interleukin-1beta (IL-1
63 tcome of a chairside test measuring gingival crevicular fluid (GCF) levels of the enzyme aspartate am
64                                     Gingival crevicular fluid (GCF) levels of the polymorphonuclear l
65                      Four different gingival crevicular fluid (GCF) markers from 14 patients were cha
66                           Different gingival crevicular fluid (GCF) matrix metalloproteinase (MMP)-8
67  this study is to explore different gingival crevicular fluid (GCF) matrix metalloproteinase-8 (MMP-8
68 al and microbiologic parameters and gingival crevicular fluid (GCF) matrix metalloproteinases-8 (MMP-
69 levels of immunoglobulin A (IgA) in gingival crevicular fluid (GCF) may be "protective", while increa
70       The analysis of biomarkers in gingival crevicular fluid (GCF) may be helpful in forecasting pat
71 e of inflammatory products found in gingival crevicular fluid (GCF) may be of value in evaluating bot
72  Biochemical markers harvested from gingival crevicular fluid (GCF) may be useful to identify and pre
73             Estimation of serum and gingival crevicular fluid (GCF) MCP levels could be a reliable in
74 tudy is to investigate salivary and gingival crevicular fluid (GCF) nitric oxide (NO) metabolism and
75  with their corresponding levels in gingival crevicular fluid (GCF) obtained from the same participan
76 nzyme-linked immunosorbent assay in gingival crevicular fluid (GCF) of 24 individuals with periodonti
77 agenase activity in the gingival tissues and crevicular fluid (GCF) of adult periodontitis subjects i
78 to determine visfatin levels in the gingival crevicular fluid (GCF) of healthy individuals and patien
79 nvestigate the levels of sTREM-1 in gingival crevicular fluid (GCF) of individuals without periodonti
80 , 2 microbiostatic proteins, in the gingival crevicular fluid (GCF) of normal children.
81 eviously demonstrated that IgG from gingival crevicular fluid (GCF) of patients with chronic periodon
82 hat ADAM8 levels are also raised in gingival crevicular fluid (GCF) of patients with periodontal dise
83 factor-kappaB ligand (RANKL) in the gingival crevicular fluid (GCF) of patients with periodontitis af
84  that TACE would be elevated in the gingival crevicular fluid (GCF) of persons with periodontitis.
85 radation and bone resorption in the gingival crevicular fluid (GCF) of the same vulnerable subjects.
86 c tromethamine mouthrinse (0.1%) on gingival crevicular fluid (GCF) prostaglandin E2 (PGE2) concentra
87 f alveolar bone, and measurement of gingival crevicular fluid (GCF) prostaglandin E2 (PGE2) levels we
88 ne naphthylamine [BANA] assay), and gingival crevicular fluid (GCF) pyridinoline cross-linked carboxy
89                                     Gingival crevicular fluid (GCF) samples and clinical parameters,
90 ollowing: 1) biochemical content in gingival crevicular fluid (GCF) samples from sites of gingival re
91 Clinical periodontal parameters and gingival crevicular fluid (GCF) samples were collected at baselin
92 ssment of periodontal measurements, gingival crevicular fluid (GCF) samples were taken at one incisor
93  multiple inflammatory mediators in gingival crevicular fluid (GCF) samples.
94 in reaction was performed using the gingival crevicular fluid (GCF) samples.
95 livered antibiotics on the level of gingival crevicular fluid (GCF) stromelysin (SL) and tissue inhib
96 he hypothesis to be tested was that gingival crevicular fluid (GCF) TGF-beta1 production was altered
97 in are higher and more sustained in gingival crevicular fluid (GCF) than in serum.
98 clines reach steady-state levels in gingival crevicular fluid (GCF) that are several-fold higher than
99  Secondary purposes were to compare gingival crevicular fluid (GCF) volume and GCF concentrations of
100 ndex (PI), gingival index (GI), and gingival crevicular fluid (GCF) volume from days 0 to 21 were sig
101 GI), bleeding on probing (BOP), PD, gingival crevicular fluid (GCF) volume, and total amount and conc
102 nical evaluation was performed, and gingival crevicular fluid (GCF) was collected for selected sites
103 on probing (BOP) were measured, and gingival crevicular fluid (GCF) was collected from five deepest p
104                                     Gingival crevicular fluid (GCF) was collected in a cohort of 44 A
105 e mean concentration of 0.5% CLM in gingival crevicular fluid (GCF) was estimated by reverse-phase hi
106                       The volume of gingival crevicular fluid (GCF) was quantified, and interleukin-1
107                                     Gingival crevicular fluid (GCF) was sampled for the analysis of m
108     Levels of visfatin in serum and gingival crevicular fluid (GCF) were explored in patients with pe
109                          Samples of gingival crevicular fluid (GCF) were obtained 1, 3, 7, 14, and 21
110 status and the enzymatic profile of gingival crevicular fluid (GCF) when given as adjunct to periodon
111       Beta-glucuronidase (betaG) in gingival crevicular fluid (GCF), a marker of neutrophil influx in
112                             Saliva, gingival crevicular fluid (GCF), and blood samples from 30 patien
113 rotic cells, has been identified in gingival crevicular fluid (GCF), and elevated levels are associat
114 sion patterns of ghrelin in saliva, gingival crevicular fluid (GCF), and gingival tissue, as well as
115 gingival temperature, flow rates of gingival crevicular fluid (GCF), and neutrophil emigration into G
116 he aim of this study is to evaluate gingival crevicular fluid (GCF), saliva, and plasma levels of IL-
117     This study investigates whether gingival crevicular fluid (GCF), saliva, and plasma levels of int
118 nd TIMP-1 levels were determined in gingival crevicular fluid (GCF), saliva, and serum by immunofluor
119 im of this study is to evaluate the gingival crevicular fluid (GCF), saliva, and serum concentrations
120                                     Gingival crevicular fluid (GCF), saliva, and serum levels of IL-6
121 macokinetic data were obtained from gingival crevicular fluid (GCF), saliva, and serum of adult perio
122 17A/F, and IL-17E (IL-25) levels in gingival crevicular fluid (GCF), saliva, and serum of non-obese f
123                                     Gingival crevicular fluid (GCF), serum, and saliva samples were o
124 as well as gingival tissue (GT) and gingival crevicular fluid (GCF), was obtained from a total of 35
125 ta on microbial plaque composition, gingival crevicular fluid (GCF)-interleukin (IL)-1beta levels, an
126 strates are different in saliva vs. gingival crevicular fluid (GCF).
127 nti-PC and anti-oxLDL production in gingival crevicular fluid (GCF).
128 ino acid, enters the oral cavity in gingival crevicular fluid (GCF).
129 dy are to comparatively analyze the gingival crevicular fluid (GCF)/peri-implant sulcus fluid (PISF)
130 ytokine levels were measured in peri-implant crevicular fluid (PICF) and gingival crevicular fluid (G
131 ecific inflammatory cytokine in peri-implant crevicular fluid (PICF) compared with healthy patients;
132                                 Peri-implant crevicular fluid (PICF) samples from the new implant and
133 udy assesses cytokine levels in peri-implant crevicular fluid (PICF)/gingival crevicular fluid (GCF)
134 uld enhance DOX availability in the gingival crevicular fluid after its release from the collagen mem
135 rails to reduce MMP activity in the gingival crevicular fluid and gingival tissues, with a resultant
136 iodontitis have type-1 cytokines in gingival crevicular fluid and high titers of IFN-gamma-dependent
137 IgG2 reactive with P. gingivalis in gingival crevicular fluid and serum.
138 and is detected at higher levels in gingival crevicular fluid and tissue specimens.
139           Measures of gingival inflammation, crevicular fluid aspartate aminotransferase (AST) levels
140          Bioassay of doxycycline in gingival crevicular fluid associated with presence of ABDS-D gave
141 ted whole-mouth salivary flow rate, gingival crevicular fluid biomarkers).
142 leeding on probing (BOP) as well as gingival crevicular fluid bone marker assessment (ICTP) and micro
143 e if levels of IL-1alpha protein in gingival crevicular fluid can be correlated to patient genotype.
144 performed a metabolomic analysis of gingival crevicular fluid collected from healthy, gingivitis, and
145                     The analysis of gingival crevicular fluid components offers great potential as a
146 s of gingipain activity detected in gingival crevicular fluid could implicate a role for gingipains i
147 be used for measuring ions from the gingival crevicular fluid directly into the peri-odontal pocket,
148 ics at concentrations detectable in gingival crevicular fluid do not dramatically reduce total bacter
149                             The reduction in crevicular fluid flow at control sites suggested that cl
150 ol sites, a significant decrease in gingival crevicular fluid flow rate was evident at the conclusion
151 galactose-deficient IgG in sera and gingival crevicular fluid from periodontal disease patients, comp
152  segments of FN and used to analyze gingival crevicular fluid from periodontal disease sites in syste
153 nd between DHA and control for both gingival crevicular fluid hsCRP (-5.3 ng/mL, standard error [SE]
154 s) and local inflammatory response (gingival crevicular fluid IL-1beta) and derive periodontal comple
155 e loss, which correlated with lower gingival crevicular fluid levels of proinflammatory mediators (e.
156 ponse to a major human salivary and gingival crevicular fluid LPA species, 18:1, and that they would
157 l parameters and cytokine levels in gingival crevicular fluid of dogs with experimental periodontitis
158 parameters and cytokine profiles in gingival crevicular fluid of patients with moderate-to-severe chr
159 nes were evaluated in the serum and gingival crevicular fluid of shallow and deep sites by enzyme-lin
160                                 Peri-implant crevicular fluid samples collected from 164 participants
161 ected the karilysin gene in >90% of gingival crevicular fluid samples containing T. forsythia obtaine
162  in the pathogenesis of periodontal disease, crevicular fluid samples from LAgP patients were found t
163                                     Gingival crevicular fluid samples from six healthy, six gingiviti
164                                     Gingival crevicular fluid samples were analyzed for changes in hi
165 Marginal and subgingival plaque and gingival crevicular fluid samples were collected at baseline; aft
166 Marginal and subgingival plaque and gingival crevicular fluid samples were collected from 15 current
167                                     Gingival crevicular fluid samples were used to monitor daily drug
168  examination; blood, subgingival plaque, and crevicular fluid specimen collection; and medical and de
169 ive attachment levels (RAL); and 5) gingival crevicular fluid volume (GCF).
170                        In addition, gingival crevicular fluid was analyzed for an inflammation index
171                                     Gingival crevicular fluid was collected before treatment, after 1
172     The mean concentration of MF in gingival crevicular fluid was estimated by reverse-phase high-per
173                          Plaque and gingival crevicular fluid were collected for trypsin-like enzyme
174 specific immunoglobulin G levels in gingival crevicular fluid were measured in primates immunized wit
175                          Samples of gingival crevicular fluid were obtained 1, 3, 7, 14, and 21 days
176 l and inflammatory cytokines in the gingival crevicular fluid, and a synergistic effect was observed,
177  are secreted--specifically saliva, gingival crevicular fluid, and airway surface fluid--differ great
178  effect on alveolar bone, effect on gingival crevicular fluid, and effect on salivary flow.
179 e examined whether IgG in serum and gingival crevicular fluid, and IgG locally produced by plasma cel
180 inase (MMP) activity in gingival tissues and crevicular fluid, in association with clinical efficacy,
181  8 selected inflammatory markers measured in crevicular fluid.
182 up to 70% of serum concentration in gingival crevicular fluid.
183                                              Crevicular fluids from localized juvenile periodontitis
184 t level, and bleeding on probing, as well as crevicular levels of interleukin-1beta and tumor necrosi
185  of AP induces a downregulation of amplified crevicular neutrophil activity.
186 lysis of betaG in saliva may be a measure of crevicular neutrophil influx for the whole mouth.
187 rinse enhances the antibacterial function of crevicular neutrophils.
188 orrelation between gingival inflammation and crevicular pH, which reportedly varies from 6.5 to 8.5.

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