ライフサイエンスコーパス: crocidolite asbestos

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1 to increase worldwide because of exposure to crocidolite asbestos.

2 nucleus determines cell fate in response to crocidolite asbestos.

3 d that incubation with low concentrations of crocidolite asbestos (0.5-1.25 microg/cm(2)) resulted in

4 Crocidolite asbestos also induced AP-1 transactivation i

5 (MET5A) exposed to various concentrations of crocidolite asbestos and man-made vitreous fibers (MMVF-

6 esothelial (RPM) cells, RPM cells exposed to crocidolite asbestos, and rat mesotheliomas, subsets of

7 In in vitro studies, crocidolite asbestos caused a dose- and time-dependent i

8 cell signaling, we evaluated the effects of crocidolite asbestos, EGF and H2O2, on MAPK activation i

9 Crocidolite asbestos elicits oxidative stress and cell p

10 ls and in cells after mechanical wounding or crocidolite asbestos exposure.

11 room air (sham-exposed) or to chrysotile or crocidolite asbestos fibers.

12 57/BL6 mice were exposed to 7-mg/m(3) air of crocidolite asbestos for 5 and 30 days, the times requir

13 te asbestos was addressed by instillation of crocidolite asbestos in a series of wild-type or SPARC-n

14 ew of the transcriptional changes induced by crocidolite asbestos in A549 human lung epithelial cells

15 Intratracheal injection of crocidolite asbestos in mice leads to pulmonary inflamma

16 We show here that crocidolite asbestos induces the DNA repair enzyme, apur

17 enitor cells of lung cancers, we report that crocidolite asbestos initially depletes intracellular gl

18 Crocidolite asbestos is known to cause cellular damage,

19 terations in gene expression associated with crocidolite asbestos or cristobalite silica exposures in

20 ells to nitric oxide (NO) in the presence of crocidolite asbestos resulted in a marked decrease in in

21 ole of SPARC in the in vivo lung response to crocidolite asbestos was addressed by instillation of cr

22 Activation of activator protein (AP-1) by crocidolite asbestos was examined in vitro in a JB6 P+ c

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