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1 C- and non-BCC-infected patients to minimize cross infection.
2 more than 40% without increasing the risk of cross infection.
3 lides or aminoglycosides, further indicating cross-infection.
4 hould be instituted with measures to prevent cross-infection.
5 to C difficile infection, but who can cause cross-infection.
6 gy with respect to ribotype distribution and cross-infection.
7 g a multi-type Lotka-Volterra framework with cross-infection.
8 cia complex frequently occurs as a result of cross infection among individuals with cystic fibrosis.
9 cile as a possibly significant problem, with cross-infection and a distinct ribotype distribution, in
10 tive surveillance for Pseudomonas aeruginosa cross-infection at a large regional adult cystic fibrosi
11 Phylogenetic analyses revealed evidence for cross infection between the resting and activated CD4+ T
12 sign and ventilation) to limit P. aeruginosa cross-infection between patients with cystic fibrosis.
13 viremic individuals and provide evidence for cross-infection between these 2 cellular compartments.
14 vides a good approximation when the level of cross-infection between vector species is very small.
16 ients from non-colonised patients to prevent cross-infection has been recommended, there is little ev
18 ryngitis in a boarding school (serotype M5), cross-infection in a hospital burn unit (serotype M76),
19 prevent medical device-related infection and cross-infection in the hospital would yield benefit with
21 Otherwise, particularly when the level of cross-infection is high, the two-host, two-vector formul
25 This limited human epidemic resulted from cross-infection of prairie dogs by imported African rode
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