ライフサイエンスコーパス: cross-desensitization

1 so showed homologous desensitization but not cross desensitization.

2 d the receptor homologously but there was no cross-desensitization.

3 GXM's inhibitory effect appeared to involve cross-desensitization.

4 osinophil intracytoplasmic Ca2+ and complete cross-desensitization.

5 Moreover, the A2a receptor-induced cross-desensitization also reduced the susceptibility of

6 Cross-desensitization among receptors for peptide chemoa

7 By promoting receptor cross-desensitization and cointernalization, such functi

8 Ca2+ release, similar dose-response curves, cross-desensitization, and partial inhibition of release

9 signals, including receptor internalization, cross-desensitization, and phospholipase D activation, a

10 Because chemotaxis and cross-desensitization are exclusively mediated by G(i),

11 ermore, inhibition of p38 MAPK prevented the cross-desensitization as well as cointernalization of mi

12 to maintained stimuli and may contribute to cross desensitization between chemical and electrical st

13 Cross-desensitization between capsaicin and ATP was Ca2+

14 ndicate the existence of reciprocal receptor cross-desensitization between CD4 and CCR5 induced by tw

15 These experiments demonstrate reciprocal cross-desensitization between CRF and stress using LC el

16 There was no cross-desensitization between galectin-3 and any of the

17 h either DAMGO or clonidine induced a mutual cross-desensitization between micro and alpha2A receptor

18 To examine cross-desensitization between receptors for lipid and pe

19 gnaling mechanisms, we evaluated patterns of cross-desensitization between SAA and other leukocyte ch

20 We have studied cross-desensitization between these two agents in rat is

21 mPAFR was also resistant to cross-desensitization by peptide chemoattractants at the

22 riments were designed to investigate whether cross-desensitization develops between CRF and stress.

23 Mediator release showed a similar cross-desensitization effect.

24 ggested that a hierarchy of sensitivities to cross-desensitization exists for the chemoattractant GPC

25 Cross-desensitization experiments using THP-1 cells sugg

26 In cross-desensitization experiments, among the numerous ch

27 responses to ATP (10 microM) suggesting that cross-desensitization had occurred between capsaicin and

28 A peripheral site of nicotine cross-desensitization is suggested via a nAChR-mediated

29 tle mu receptor endocytosis, induced neither cross-desensitization nor internalization of alpha2A rec

30 r to that induced by serotonin, and complete cross-desensitization occurred between the InsP3 and 5-H

31 ated equivalent PLCbeta3 phosphorylation and cross-desensitization of Ca2+ mobilization by FR, C5aR,

32 Furthermore, we found that the cross-desensitization of CCR1 by FPR1 was associated wit

33 and found that the mechanism of MOR-induced cross-desensitization of CCR5 involves the activation of

34 or dominant negative mutants suppresses the cross-desensitization of CCR5.

35 Further, CCL1 and CCL18 induced heterologous cross-desensitization of CCR8-transfected cells and huma

36 tudy the role of receptor phosphorylation in cross-desensitization of chemoattractant receptors, M2CX

37 okine receptor regulation further we studied cross-desensitization of chemokine receptors in normal P

38 acetylcholine receptors (nAChRs) in nicotine cross-desensitization of chemonociceptive responses of t

39 ) mobilization, suggesting that NMDA induces cross-desensitization of CXCR2.

40 This cross-desensitization of CXCR4 was manifested by the inh

41 porting the idea that TCR signaling leads to cross-desensitization of CXCR4.

42 on(-/-)) also showed decreased CCR5-mediated cross-desensitization of G protein activation and Ca(2+)

43 ide hydrolysis and cross-phosphorylation and cross-desensitization of IL-8RA.

44 horylation and cross-phosphorylation but not cross-desensitization of its Ca2+ mobilization by fMLP o

45 /G protein uncoupling, its susceptibility to cross-desensitization of its Ca2+ response by fMLP and C

46 The data herein suggest that cross-desensitization of PAFR by peptide chemoattractant

47 pioid receptor (MOR) is capable of mediating cross-desensitization of several chemokine receptors inc

48 The magnitude of cross-desensitization of the Akt or extracellular signal

49 urosporine blocked cross-phosphorylation and cross-desensitization of the PAFR by peptide chemoattrac

50 TrkA-expressing neurons to NGF resulted in a cross-desensitization of turning responses induced by a

51 Chemokine receptor cross-desensitization provides an important mechanism to

52 Cross-desensitization studies and single-receptor transf

53 ive calcium flux in primary lymphocytes, and cross-desensitization studies indicate that MCP-2 acts v

54 Finally, the micro-alpha2A cross-desensitization was absent in dorsal root ganglion

55 The cross-desensitization was closely associated with simult

56 rs, homologous desensitization was seen, but cross-desensitization was not observed.

57 kely a normal cellular process that leads to cross-desensitization, which is exploited by the B subun

58 ion, taurine-induced current showed complete cross-desensitization with glycine-activated currents bu

59 nsitization to capsaicin is due to selective cross-desensitization with the heteromeric P2X receptors

60 ous desensitization of FP and also exhibited cross-desensitization, with PGF2alpha resulting in a max