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1                                          LPA cross-desensitized both the alkenyl-GP and cyclic-PA res
2      TA-induced Ca(2+) flux in monocytes was cross-desensitized by an agonistic ligand MMK-1 specific
3 Ca(2+) mobilization in monocytes can also be cross-desensitized by an FPRL1-specific agonist.
4 Y1 receptor-selective antagonist and was not cross-desensitized by ATP.
5 MLP (FR), CXCR2 was cross-phosphorylated and cross-desensitized by C5a and fMLP.
6 ) mobilization by S3 and DeltaCCR1 were also cross-desensitized by CXCR1 and CXCR2 despite lack of re
7 ther C5aR nor FR was cross-phosphorylated or cross-desensitized by CXCR2 activation, although CXCR1 d
8 sitized by PAF, and cross-phosphorylated and cross-desensitized by fMet-Leu-Phe, C5a, and IL-8.
9 due, mediates prolonged signaling and is not cross-desensitized by OnM.
10 ctants were neither cross-phosphorylated nor cross-desensitized by PAF.
11 )A also induced sustained ERK activation and cross-desensitized Ca(2+) mobilization to CCR5 relative
12    In contrast to CXCR2, stimulation of 331T cross-desensitized Ca2+ mobilization by both FR and C5aR
13 ing the PAFRs along with CXCR1, IL-8 (CXCL8) cross-desensitized Ca2+ mobilization to PAF by all the r
14 CR2, but not CXCR2, activates PKCepsilon and cross-desensitizes CCR5, the data further suggest that s
15 Interestingly, CCR1 cross-phosphorylated and cross-desensitized CXCR2, but not CXCR1.
16 s and amyloid-beta were able to reciprocally cross-desensitize each other.
17                                        Lkn-1 cross-desensitized hMIP-1 alpha-induced calcium flux, bu
18      We previously demonstrated that cys-LTs cross-desensitized IL-4-primed primary human mast cells
19 CXCR1 or CXCR2 cross-phosphorylated CCR1 and cross-desensitized its ability to stimulate GTPase activ
20 wn to signal for calcium flux in a self- and cross-desensitizing manner, implicating a single recepto
21  to also influence the perception of pain by cross-desensitizing opioid G protein-coupled receptors f
22  sustained signals, cross-phosphorylated and cross-desensitized responses to CXCR1 as well as CXCR2.
23 lation of the mPAFR cross-phosphorylated and cross-desensitized responses to IL-8RA.
24 ular calcium stores and bradykinin failed to cross-desensitize the DADLE-evoked response, although bo
25 duced molecule 1 ligand chemokine completely cross-desensitized the calcium response of each other, i
26                                   Alkenyl-GP cross-desensitized the cyclic-PA response, but only part
27 ing an increase in intracellular calcium and cross-desensitizing the response of the receptor to CCL1
28 CR2, cross-phosphorylated CCR5 and CXCR4 and cross-desensitized their responsiveness to RANTES (regul
29 sitized slowly (over approximately 20-50 s), cross-desensitized with each other, were associated with
30 /Gro-alpha in neutrophils are heterologously cross-desensitized, with a hierarchy of resistance to co

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