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1 interaction of pathogen strains, mediated by cross immunity.
2 repeatedly interpreted as inconsistent with cross-immunity.
3 tions expected in the presence or absence of cross-immunity.
4 We suggest that this may be an indicator of cross-immunity.
5 e been insufficient to identify even intense cross-immunity.
6 Pertussis vaccine may induce cross-immunity.
7 strains into a single parameter representing cross-immunity.
8 distinct serotypes, resulting in inefficient cross-immunity.
9 genotypes, determining the degree of strain cross-immunity.
10 er of antigenic types that interact via host cross-immunity.
11 n be modeled through coinfection or complete cross-immunity.
12 se lpf phase-off variants were able to evade cross-immunity.
13 otherwise identical strains compete through cross-immunity.
14 formulation, assuming polarized immunity and cross-immunity act to reduced transmission probability.
16 still propagate in environments with reduced cross-immunity among different strain groups, even after
17 tructed to evaluate how different degrees of cross-immunity among M. tuberculosis groups could affect
18 f immune-mediated interactions, arising from cross-immunity and antibody-dependent enhancement, betwe
19 rogenitor are most successful under moderate cross-immunity and frequent re-infections, and (3) the i
20 er replacement depends on the specificity of cross-immunity and on the underlying pathogen mutation r
21 idence that T cells are responsible for this cross-immunity and that cross-stimulation of T cells als
22 ibility associated to each influenza strain, cross-immunity and the timing of the onset of the second
27 cent epidemiological modelling suggests that cross-immunity between RSV, HMPV and human parainfluenza
28 lpf phase variation is a mechanism to evade cross-immunity between Salmonella serotypes, thereby all
31 ation of LP fimbriae is a mechanism to evade cross-immunity between serotypes Enteritidis and Typhimu
32 gen of B. parapertussis confers asymmetrical cross-immunity between the causative agents of whooping
35 Increasing population density or decreasing cross-immunity could not fully explain the observed patt
37 longitudinal survey to determine the effect cross-immunity has on the prevalence of multiple infecti
38 he interaction of viral replication rate and cross-immunity imprint host population immunity, which i
39 and we address the role of host mobility and cross-immunity in shaping possible dominance/co-dominanc
40 structure (e.g. particular host behaviours, cross-immunity, interspecific competition) could be affe
43 d wave can occur whenever R 01 < 1.5 or when cross-immunity levels are less than 0.58 for our estimat
44 se this information to explore the impact of cross-immunity levels on the dynamics of the second wave
47 ination, strain competition mediated through cross-immunity structures the parasite population into a
48 ree modes to discuss the relationships among cross-immunity, the basic reproductive rates of each str
49 of two strains of influenza interacting via cross-immunity to simulate two temporal waves of influen
50 y mixed populations, our model confirms that cross-immunity to strains sharing alleles at antigenic l
51 ccinology approaches could enhance intrinsic cross-immunity to these paramyxoviruses and approaches t
52 nt epitopes typically increases the range of cross-immunity values in which chaotic strain dynamics a
53 rain disease transmission model with perfect cross immunity where environmental transmission is broad
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