ライフサイエンスコーパス: cross-tolerance

1 ffective concentration of Cd was considered (cross-tolerance).

2 that first encounter an Ag in the periphery (cross-tolerance).

3 oss-priming) or in CD8+ T cell inactivation (cross-tolerance).

4 tion during lipopolysaccharide tolerance and cross tolerance.

5 ignaling is involved in the induction of TLR cross-tolerance.

6 tion of TRIM30alpha and LTbetaR-mediated TLR cross-tolerance.

7 erexpression, suggesting its crucial role in cross-tolerance.

8 TNF-alpha production, mimicking LPS-induced cross-tolerance.

9 LPS-exposed THP-1 monocytes was studied for cross-tolerance.

10 vitro monocytic cell-based endotoxin-induced cross-tolerance.

11 THP1 cells resulted in a state of flagellin cross-tolerance.

12 y closely correlates with the development of cross-tolerance.

13 hine, which may account for the lack of NSIA cross-tolerance.

14 DCs were required for both cross-priming and cross-tolerance.

15 g that LPS and mycobacterial products induce cross-tolerance.

16 o the in vivo phenomena of cross-priming and cross-tolerance.

17 the vehicle previously, indicating a lack of cross-tolerance.

18 LPS and IL-1 were found to induce a state of cross-tolerance against each other, while no such recipr

19 In addition, there is cross-tolerance and cross-dependence between mu, A1, and

20 Furthermore, cross-tolerance and cross-withdrawal between mu, A1, and

21 The observations of cross-tolerance and cross-withdrawal suggest that all th

22 rized human promonocytic THP-1 cells develop cross-tolerance and no longer respond to LTA-induced IL-

23 hment of an in vitro system for the study of cross-tolerance and show that dendritic cells (DCs) phag

24 ent from LPS with regard to the induction of cross-tolerance, and these actions may be mediated, at l

25 Pam3Cys and Ec LPS did not induce a state of cross-tolerance at the level of TNF-alpha.

26 he accentuating effect of ICB CHA suggesting cross-tolerance between adenosine agonists and cannabino

27 ections in patients with CLL and describes a cross-tolerance between endotoxins and tumors.

28 However, a paucity of information exists on cross-tolerance between hemorrhage and sepsis.

29 We also demonstrated that there was cross-tolerance between SP-A and LPS in THP-1 cells.

30 lease, and HLA-DR expression, and induce TLR cross-tolerance by decreased phosphorylation of MAPK pat

31 To date, LPS-induced cross-tolerance has not been examined regarding microRNA

32 Cross-tolerance, in which plants pre-treated with chitin

33 g-SP tolerance via both direct and indirect (cross-tolerance) mechanisms leading to prevention and ef

34 induce direct and most importantly indirect cross-tolerance of alloantigen-specific T cells.

35 ontaneous cytotoxic response attributable to cross-tolerance of several unrelated NK-activating recep

36 te that after high-dose cocaine SA, there is cross-tolerance of the DAT to other uptake blockers, but

37 ogenous antigens and can maintain tolerance (cross-tolerance) or induce immune responses (cross-primi

38 either the same (tolerance) or a different (cross-tolerance) oxidant gas.

39 f evidence that NOD2 stimulation activates a cross-tolerance response that downregulates and thus pre

40 opolysaccharide tolerance) or Gram-positive (cross tolerance) stimulus.

41 n of induced tolerance also provides induced cross-tolerance that is not restricted to pesticides wit

42 ingly appreciated that heat shock may induce cross-tolerance to a variety of stimuli based on in vitr

43 184 lost its analgesic activity and produced cross-tolerance to cannabinoid receptor (CB1) agonists i

44 to the anticonvulsant action of this drug or cross-tolerance to diazepam.

45 de (LPS) and flagellin may induce a state of cross-tolerance to each other.

46 ular O2.- and prevented tolerance to NTG and cross-tolerance to endogenous nitric oxide released by a

47 Induction of cross-tolerance to endotoxin by endogenous cytokines has

48 ration of a low dose of TNF-alpha can induce cross-tolerance to endotoxin by induction of endogenous

49 LPS-induced cross-tolerance to flagellin is also associated with a b

50 ce to endotoxin over 7 days, which conferred cross-tolerance to intestinal manipulation.

51 n kinase activity and subsequently conferred cross-tolerance to lipopolysaccharide stimulation.

52 d higher tolerance to carbaryl and increased cross-tolerance to malathion and cypermethrin but not to

53 o evidence was observed for a role of IDO or cross-tolerance to mycobacterial Hsp70, mycobacterial Hs

54 mic morphine administration but did not show cross-tolerance to NSIA.

55 so investigated the ability of LPS to induce cross-tolerance to postoperative ileus.

56 ception and the development of tolerance and cross-tolerance to the antinociceptive effects of opioid

57 tioning of the muscularis showed significant cross-tolerance to the functional, molecular, and leukoc

58 In addition, cross-tolerance to the inhibitory effect of the mu-opioi

59 cyclase and suggest a potential mechanism of cross-tolerance to the motor incoordinating effects of c

60 n on bone marrow-derived macrophages induced cross-tolerance to TLR4 and TLR9 ligands in vitro.

61 ages derived from these mice LTbetaR-induced cross-tolerance to TLR4 and TLR9 ligands was impaired.

62 state of hyporesponsiveness, referred to as cross-tolerance, to both homologous and heterologous lig

63 Cross-tolerance was also demonstrated following 3 days o

64 TNF-induced cross-tolerance was dependent on the kinase GSK3, which

65 Cross-tolerance was not a characteristic of adr1 plants,

66 Cross-tolerance was observed by TLR5 ligand flagellin an

67 han saline-infused rats indicates that kappa cross-tolerance was present.

68 r antagonist naloxone, and two-way analgesic cross-tolerance with morphine.

69 e effective with meditation due to a lack of cross-tolerance with opiate-based medications.