ライフサイエンスコーパス: crossbred

1 logical abnormalities on inbred (129/Ola) or crossbred (129/Ola x C57BL/6) genetic backgrounds.

2 milarity in function between ANK and NPP1 we crossbred Akp2(-/-) mice to ank/ank mice and found a par

3 To do so, we crossbred ank/ank mice with mice lacking Vanin-1 panteth

4 tion (AI) were conducted in 201 synchronized crossbred beef heifers.

5 le from Suino Nero Lucano (SNL) and a modern crossbred (CG) pigs, before and after cooking and in vit

6 mmunostaining of cytoskeletal proteins and a crossbred D(1) receptor null:YFP transgenic reporter lin

7 /-) and Rpe65(-/-) mice (n = 126) as well as crossbred Gnat1(-/-) mice lacking rod phototransduction

8 Retinas of Lrat(-/-), Rpe65(-/-), and crossbred Gnat1(-/-) mice tolerated prolonged high-dose

9 rporation in spontaneous prostate tumors, we crossbred Id mutant mice with the transgenic adenocarcin

10 Linkage analysis of a crossbred informative pedigree showed five obligate reco

11 Importantly, when crossbred into a mouse genetic model of CHF (alpha-myosi

12 levels of orally administered tranilast, 36 crossbred juvenile pigs were randomized to placebo or tr

13 Crossbred Karan Fries (KF) cows, among the best yielders

14 Female crossbred Landrace/Yorkshire/Duroc pigs (27-32 kg).

15 In the current study, we have crossbred M83 mice on a DJ-1 null background (M83-DJnull

16 Steatohepatitis score of crossbred mice (ob/ob/ko) was similar to that of ob/ob m

17 xidase system on activation of NF-kappaB, we crossbred mice deficient in p47(phox) with NF-kappaB rep

18 activation on amyloid pathology in vivo, we crossbred mice lacking CX3CR1 with the Alzheimer's mouse

19 ould increase survival in cardiomyopathy, we crossbred mice with Gq-associated cardiomyopathy and tho

20 e role of MYH9 deficiency in nephropathy, we crossbred Myh9-haploinsufficient mice (Myh9(+/-)) with H

21 esis in the absence of apoptosis in vivo, we crossbred NHEJ-deficient mice into a mutant p53R172P bac

22 Crossbred piglets were assigned to three groups, intracr

23 Seventy-six, crossbred, porcine reproductive and respiratory syndrome

24 ormation of intraneuronal tau inclusions, we crossbred previously described tau (T44) Tg mice overexp

25 A 2-month-old female crossbred puppy was submitted to necropsy with a history

26 igate the phenotype in a mammalian model, we crossbred SCA1 mice with mice over-expressing a molecula

27 Eighteen juvenile male crossbred swine were included.

28 CAR at the surface of the airway epithelium, crossbred these mice with mice that were genetically dev

29 we generated L2-cyclin D1 (L2D1(+)) mice and crossbred these with p53-deficient mice.

30 participation of TLR7 in atherosclerosis, we crossbred TLR7-deficient (Tlr7 (-/-)) mice with apolipop

31 d PF4-driven tTA-viral protein 16 (VP16) was crossbred to a responder line.

32 ) ) and wild-type mice (WT, NOS3(+/+) ) were crossbred to generate homozygous NOS3(-/-) (KO), materna

33 e generated in the authors' laboratories and crossbred to generate Sur/SSTR5 KO mice.

34 The families are crossbred to maximize levels of heterozygosity and inclu

35 Single Ccl2- and Cx3cr1-deficient mice were crossbred to obtain Ccl2(-/-)/Cx3cr1(-/-) mice.

36 ay, PC(+/-)/FXI(-/-) mice were generated and crossbred to produce double-deficient progeny (PC(-/-)/F

37 Three erd-affected dogs were crossbred to three prcd-affected dogs, and their progeny

38 er facilitate bladder autoimmunity study, we crossbred URO-OVA mice with OVA-specific CD8(+) TCR Tg m

39 xidative stress reactions using ApoE-/- mice crossbred with 12/15LO-deficient (12/15LO-/-) mice (12/1

40 TSLP transgenic mice were crossbred with animals deficient for FcgammaRIIb on the

41 ically deficient in CD11c were generated and crossbred with apolipoprotein E (apoE)-/- mice to genera

42 Cystatin C-deficient mice (Cyst C-/-) were crossbred with apolipoprotein E-deficient mice (ApoE-/-)

43 ion were more dramatic in NEP2 knockout mice crossbred with APP transgenic mice.

44 epitopes in 12/15-lipoxygenase knockout mice crossbred with atherosclerosis-prone apo E-deficient mic

45 cle-specific PGC-1alpha transgenic mice were crossbred with cardiac-specific calsequestrin transgenic

46 Kit(+/copGFP) mice were crossbred with diabetic Lep(+/ob) mice to generate compo

47 A floxed synectin knockin mouse line was crossbred with endothelial-specific (Tie2, Cdh5, Pdgfb)

48 ls were counted from progeny of BDNF-OE mice crossbred with green fluorescent protein (GFP) (gustduci

49 n of gangliosides, the GD3S mutant mice were crossbred with mice carrying a disrupted GalNAcT gene en

50 on of the disease, TSLP transgenic mice were crossbred with mice deficient for immunoglobulin-binding

51 When crossbred with mice lacking the other allele of tumor-su

52 shed cardiac function, Rab4 mutant mice were crossbred with mice overexpressing human beta2AR.

53 heir wild-type littermates (CBS((+/+))) were crossbred with mice that overexpress GPx-1 [GPx-1((tg+))

54 irected expression of AC type VI (ACVI) were crossbred with mice with cardiomyopathy induced by cardi

55 oma formation with 30 - 40% penetrance, were crossbred with p27+/- mice for two successive generation

56 ntial, a strain of tal-1 transgenic mice was crossbred with p53-/- mice; the survival rate in these a

57 3 stabilize each other, SRC-3(-/-) mice were crossbred with the liver-specific transthyretin (TTR)-IG