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1 vely selected or non-selected were minimally crossreactive.
2                       To test whether highly crossreactive alphabeta T cell receptors (TCRs) produced
3                       These T cells showed a crossreactive and heteroclitic (stronger) response to de
4                                      Using a crossreactive antibody, we show that the embryonic expre
5 l benefits and treatment outcomes using less crossreactive antigens will enhance clinical and treatme
6 imed by higher amounts of self antigen or by crossreactive antigens.
7 ein present two clinically important and non-crossreactive antigens: core antigen (HBcAg), which appe
8 tic heart disease have provided evidence for crossreactive autoantibodies that target the dominant gr
9       To summarize, pathogenic mechanisms of crossreactive autoantibodies which target the valve in r
10 ry cells, formation of dysfunctional HDL and crossreactive autoantibodies.
11 ique natural experiment to determine whether crossreactive cellular immunity limits symptomatic illne
12 itol (GPI) anchor that is recognized by anti-crossreactive determinant antibodies.
13  responses to donor antigens could spread to crossreactive determinants on self-proteins, thus perpet
14 areas have antibodies in their sera that are crossreactive for both helminth and malaria parasites ra
15 e thymus contains many T cells that are very crossreactive for peptide and MHC and that subsequent ne
16 cific fusion inhibition activity; only 1 had crossreactive fusion inhibition activity against HIV-1/M
17 s IgE antibodies, are highly immunologically crossreactive glycoproteins exclusively expressed in pol
18 a now suggest that mutations associated with crossreactive material-negative fXI deficiency fall into
19 autoantibodies against collagen that are not crossreactive may form because of the release of collage
20             This is in part accounted for by crossreactive memory T cells, which can be employed in i
21 etermine the kinetics of strain-specific and crossreactive nAb emergence, we studied patient H, the s
22         The delayed appearance of high titer crossreactive nAbs in chronically infected patients sugg
23 the MN strain of HIV-1 (HIV-1/MN), and 2 had crossreactive neutralizing activity against the IIIB str
24                           Nine of the 13 had crossreactive neutralizing activity against the MN strai
25 lly infected with HCV develop high-titer and crossreactive neutralizing antibodies (nAbs), yet fail t
26                            In the absence of crossreactive neutralizing antibodies, CD8(+) T cells sp
27  rgp120 in MF59 can induce type-specific and crossreactive neutralizing antibody against B-subtype la
28 uick and simultaneous testing of potentially crossreactive NMBA and the identification of safe altern
29 allergy diagnostics enables the detection of crossreactive or species-specific allergen components.
30                                 In addition, crossreactive patterns were observed between different l
31  resulted from the presentation of the donor crossreactive peptide Kk 61-80 at the surface of recipie
32 a) production was measured by a specific non-crossreactive RIA.
33  overview of the various types of biomimetic crossreactive sensor arrays (also referred to as electro
34                                   Biomimetic crossreactive sensor arrays have been used to detect and
35 , FTK-OspA, in which the LFA-1alpha(L)/rOspA crossreactive T cell epitope was mutated to reduce the p
36 CDR1 and CDR2 amino acids dominated the most crossreactive TCR interface with MHC, including Vbeta8 4
37                                Thus, broadly crossreactive TCRs arise at low frequency in the pre-sel
38                         These data show that crossreactive TCRs can spotlight the evolutionarily cons
39                        Negative selection of crossreactive TCRs led to clonal deletion but also recyc
40 face of cells and also makes TCRs inherently crossreactive towards different peptides bound by the sa
41 t only of target enzyme inhibition, but also crossreactive with chemical xenobiotics that share molec
42    To identify HCV protein which possibly is crossreactive with M3R or which binds to this receptor,
43 ntracellular regions of the EGFR, but is not crossreactive with other HER-family members.
44 ut varied from noncrossreactive to extremely crossreactive with other peptides and MHCs.
45 able, and a nonspecific insulin immunoassay (crossreactive with proinsulin) was used.
46 eart valves revealed the presence of T cells crossreactive with streptococcal M protein and cardiac m
47  T cells specific for L144/R147 peptide were crossreactive with the native PLP-(139-151) peptide, pro

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