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1 o exclusively yield the C4 coupling product, crotonaldehyde.
2 the mutagenic and carcinogenic properties of crotonaldehyde.
3 om cigarette smoke and polluted air, such as crotonaldehyde, acrolein, and oxidizing agents such as h
4 rchain cross-link formation by the analogous crotonaldehyde adduct (2b) was evaluated in a 5'-dC-2b s
5 ially incorporated opposite the acrolein and crotonaldehyde adducts, and dTTP incorporation was prefe
7 alpha,beta-unsaturated aldehydes, acrolein, crotonaldehyde, and prenal, over Pt(111) at Torr pressur
8 ng adducts derived from butadiene, acrolein, crotonaldehyde, and styrene, and examined for effects on
9 g 1,N(2)-deoxyguanosine adducts of acrolein, crotonaldehyde, and trans-4-hydroxynonenal can form cros
10 ine interstrand cross-links arising from the crotonaldehyde- and acetaldehyde-derived R- and S-alpha-
11 a-unsaturated aldehydes such as acrolein and crotonaldehyde are common environmental pollutants prese
13 ermidine directly reacts with AA to generate crotonaldehyde (CrA), most likely via an enamine aldol c
14 e (dG) with enals, including acrolein (Acr), crotonaldehyde (Cro), pentenal (Pen), heptenal (Hep), an
16 cyclic adducts, has shown that acrolein- and crotonaldehyde-derived 1,N2-propanodeoxyguanosine adduct
23 aturated carbonyl compounds like acrolein or crotonaldehyde in acetone-d(6) generates metastable (E)-
24 that acrolein was 2000-fold more potent than crotonaldehyde in blocking DNA binding to an NF-kappaB c
25 ld: 16%) and B (1b, overall yield: 34%) from crotonaldehyde in nine and seven steps, respectively, ha
29 d aldehydes in cigarette smoke (acrolein and crotonaldehyde) inhibited production of interleukin-2 (I
31 cy site on the TiO2 surface, the C=O bond of crotonaldehyde is activated, by charge transfer, for hyd
32 arified by carrying out the hydrogenation of crotonaldehyde over both Pt(111) and Pt(100) single crys
34 ata to experiments performed with 2-butenal (crotonaldehyde) shows that EpB and 2-butenal decompose t
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