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1 DHPRs) and RyRs within Ca(2+) release units (CRUs).
2 cial structures named calcium release units (CRUs).
3 ork of diffusively coupled Ca release units (CRUs).
4 t" (Ca sparks inducing Ca sparks in adjacent CRUs).
5 zed structures called calcium release units (CRUs).
6 ing of an ensemble of interacting stochastic CRUs.
7 e full pressures, referenced to the anatomic crus.
8 e administered cisatracurium to paralyse the crus.
9 elate sphincteric pressure with the anatomic crus.
10 pproximately 3-fold lower than that of young CRUs.
12 a region in posterior cerebellum (lobule VII crus 1) was engaged specifically when a temporal-spatial
14 o this end, multiple electrode recordings of crus 2a complex spike activity were obtained in awake ra
18 iple electrode recordings were obtained from crus 2a Purkinje cells, and carbenoxolone, a gap junctio
19 ); 2) stochastic Ca2+ release (or firing) of CRUs; 3) discrete, asymmetric distribution of CRUs along
21 tracts involved in limbic circuitry (fornix crus [AD, beta = 0.02 (P = .046)] and cingulum [RD, beta
22 RUs; 3) discrete, asymmetric distribution of CRUs along the longitudinal (separation distance of 2 mi
27 Ca cycling model in which Ca release units (CRUs) are locally coupled by Ca diffusion throughout the
29 a subsection of cerebellar gray matter (cere-crus) as well as a parametrically derived white matter-b
31 investigated motor fiber organization in the crus cerebri of the cerebral peduncle (ccCP) in the rhes
35 that expression of either isoform results in CRUs containing arrays of feet, indicating the ability o
37 ing failure of endolymphatic duct and common crus formation, accompanied by epithelial dilatation and
42 genome sequence of the hexaploid species E. crus-galli, i.e., a 1.27 Gb assembly representing 90.7%
43 ring capacity, and the spatial separation of CRUs help control the inherent instability of SR Ca2+ re
45 ebellar abnormalities, particularly in Right Crus I (RCrusI), are consistently reported in autism spe
47 r cortical Golgi cells and Purkinje cells in Crus I and II of the posterior lobe cerebellar hemispher
50 bellar lesion volume, cerebellar Lobules VI, Crus I and VIIIa atrophy being independent predictors of
54 tracers were microinjected into the folia of crus I of the cat cerebellum to investigate spatial loca
56 traction of total cerebellar, lobule IV, and Crus I volumes with additional X- or Y-chromosomes; X-sp
61 (p<0.001) and left lobule VI (p<0.01), left crus I, right VIIb and entire cerebellum (p<0.05 for eac
62 he cerebellar nuclei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI ipsilateral to t
69 S, anodal tDCS increased activation in right Crus I/II during semantic prediction and enhanced restin
71 ing activity in the human lateral cerebellar Crus I/II modulates the cerebral default mode network, w
72 al gyrus (z score = 4.31, P < .001), and the crus I/II of the cerebellum (z score = 3.77, P < .001),
74 deling, we hypothesize that right cerebellar Crus I/II supports prediction of upcoming sentence conte
76 y lower functional connectivity of the right Crus I/II with the left dorsolateral prefrontal cortex.
77 ght cerebellar lobules VI and VII (including Crus I/II) are engaged during a variety of language proc
78 erolateral portions of the cerebellum (right Crus I/II) contribute to language processing, but the na
79 owed marked atrophy in AD, ALS, FTD and PSP (Crus I/II), and MSA and PSP (lobules I-IV), respectively
81 ermis lobule VII or right lateral cerebellar Crus I/II, subregions that prominently couple to the dor
82 indazole attenuated the vascular response to crus II activation in wild-type mice but not in D2-null
83 stimulation induces patch-like activation of Crus II and GABAA antagonists fail to convert this patch
84 bing fibre responses were evoked in parts of crus II and paramedian lobule by stimulation of corticof
86 peripherally evoked patch-like responses in Crus II are aligned between parasagittal bands of EAAT4.
90 e show that the majority of granule cells in Crus II of the cerebrocerebellum receive sensory-evoked
91 tivity of neighboring individual MLIs in the Crus II region of awake female mice during two types of
94 clei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI ipsilateral to the conditioned e
95 ffect the climbing fibre responses evoked in crus II, and produced a relatively small reduction of th
97 xpansion of total cerebellum, flocculus, and Crus II-lobule VIIIB volumes in males) and SCA (contract
102 cell column, we recorded simultaneously from crus IIA areas and from left and right vermal lobule IX,
103 ranule cell layer (GCL) of cerebellar folium Crus IIa as freely moving rats engaged in a variety of n
104 of all four areas, i.e., the left and right crus IIA as well as the left and right lobule IX, can fi
105 ke synchrony observed between left and right crus IIA could indeed be mediated in part through couple
106 fractured tactile cerebellar map within the crus IIa folia of the cerebellar hemispheres reorganizes
107 the 1000-4500 Hz multiunit responses in the Crus IIa GCL of awake rats are correlated with tactile i
108 ossy fiber and climbing fiber projections to crus IIa in the lateral hemispheres of the rat cerebellu
109 mbing fiber activation of the left and right crus IIA in the rat can be explained by (1) bilateral in
110 mined in the tactilely responsive regions of crus IIa in the rat, the results show that SI influences
111 imultaneous recordings of the left and right crus IIA of the cerebellar cortex in the rat have demons
112 the tactile map in the granule cell layer of crus IIa reorganized, with representations of intact str
113 rents of the olivary regions that project to crus IIA was studied using Phaseolus vulgaris leucoagglu
115 e structural role of RyR3 in the assembly of CRUs in 1B5 cells independently expressing either RyR1 o
117 in three vineyards, representing different 'crus' in the cultivation areas of Barolo, Barbaresco and
118 cy of marrow competitive repopulation units (CRUs) increased approximately 2-fold from 2 months to 2
119 ayed afterdepolarizations, Ca release units (CRUs) interact with not just the immediately adjacent CR
121 tract-based spatial statistics suggested the crus of the fornix as a focus for this relationship.
122 ately 20 pA) through the Ca2+ release units (CRUs) of the sarcoplasmic reticulum (SR); 2) stochastic
123 alamus, right posterior cingulum, and fornix crus (seven studies; largest cluster, 980 voxels; z = 2.
127 -SR Ca diffusion from neighboring non-firing CRUs to the firing CRUs, which helps to maintain the loc
128 anization and RyR2 orphaning (5000 of 20 000 CRUs) uncovered a local mechanism of delayed subcellular
130 eract with not just the immediately adjacent CRUs via Ca diffusion, but also further CRUs via fast (
131 cent CRUs via Ca diffusion, but also further CRUs via fast ( approximately 0.1 ms) changes in Vm medi
132 ears of age, the BM homing efficiency of old CRUs was approximately 3-fold lower than that of young C
133 ceptible to RA treatment, whereas the common crus was particularly resistant, suggesting that the mol
135 the spark frequency one would observe if the CRUs were incommunicado; 2), the coupling strength, whic
136 a2+ currents through the Ca2+ release units (CRUs) were approximately 1-2 pA, producing sparks with p
137 oupling is governed by Ca(2+) release units (CRUs) whereby Ca(2+) influx via L-type Ca(2+) channels (
138 om neighboring non-firing CRUs to the firing CRUs, which helps to maintain the local SR Ca concentrat
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