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1 DHPRs) and RyRs within Ca(2+) release units (CRUs).
2 cial structures named calcium release units (CRUs).
3 ork of diffusively coupled Ca release units (CRUs).
4 t" (Ca sparks inducing Ca sparks in adjacent CRUs).
5 zed structures called calcium release units (CRUs).
6 ing of an ensemble of interacting stochastic CRUs.
7 e full pressures, referenced to the anatomic crus.
8 e administered cisatracurium to paralyse the crus.
9 elate sphincteric pressure with the anatomic crus.
10 pproximately 3-fold lower than that of young CRUs.
11                      Purkinje cells (PCs) in Crus 1 represent whisker movement via linear changes in
12 a region in posterior cerebellum (lobule VII crus 1) was engaged specifically when a temporal-spatial
13 eria included three CRs, 12 unconfirmed CRs (CRus), 16 PRs, 26 with SD, four with PD, and 21 NA.
14 o this end, multiple electrode recordings of crus 2a complex spike activity were obtained in awake ra
15 recordings of CSs and simple spikes (SSs) in crus 2a of anesthetized rats.
16      We used multiple electrode recording of crus 2a Purkinje cell complex spikes (CSs) in ketamine-x
17 x spike (CS) activity were obtained from 236 crus 2a Purkinje cells in anesthetized rats.
18 iple electrode recordings were obtained from crus 2a Purkinje cells, and carbenoxolone, a gap junctio
19 ); 2) stochastic Ca2+ release (or firing) of CRUs; 3) discrete, asymmetric distribution of CRUs along
20  the local SR Ca concentration of the firing CRUs above a critical level to sustain firing.
21  tracts involved in limbic circuitry (fornix crus [AD, beta = 0.02 (P = .046)] and cingulum [RD, beta
22 RUs; 3) discrete, asymmetric distribution of CRUs along the longitudinal (separation distance of 2 mi
23 regulators have on the intrinsic activity of CRUs and on the coupling between them.
24 ccule, and the endolymphatic duct and common crus are invariably fused.
25 tion provides some isolation from neighbors, CRUs are not incommunicado.
26                       Calcium release units (CRUs) are junctions between the sarcoplasmic reticulum (
27  Ca cycling model in which Ca release units (CRUs) are locally coupled by Ca diffusion throughout the
28 Logan graphical analysis with the cerebellum crus as a reference region.
29 a subsection of cerebellar gray matter (cere-crus) as well as a parametrically derived white matter-b
30 Rs) and triadin, two essential components of CRUs, but no RyRs (or feet).
31 investigated motor fiber organization in the crus cerebri of the cerebral peduncle (ccCP) in the rhes
32 rona radiata (CR), internal capsule (IC) and crus cerebri of the cerebral peduncle (ccCP).
33                           In skeletal muscle CRUs contain two isoforms of the sarcoplasmic reticulum
34                                              CRUs contain two proteins essential to e-c coupling: dih
35 that expression of either isoform results in CRUs containing arrays of feet, indicating the ability o
36                                A clusters of CRUs, each producing 10 pA simultaneously, can produce s
37 ing failure of endolymphatic duct and common crus formation, accompanied by epithelial dilatation and
38 hytoalexin momilactone A are found in the E. crus-galli genome, respectively.
39                   Barnyardgrass (Echinochloa crus-galli) is a pernicious weed in agricultural fields
40                   Barnyardgrass (Echinochloa crus-galli) is an invasive plant that is difficult to co
41 rice paddy weed, barnyard grass (Echinochloa crus-galli).
42  genome sequence of the hexaploid species E. crus-galli, i.e., a 1.27 Gb assembly representing 90.7%
43 ring capacity, and the spatial separation of CRUs help control the inherent instability of SR Ca2+ re
44 IS for all lobules in subregions VI and left crus I (p<0.05).
45 ebellar abnormalities, particularly in Right Crus I (RCrusI), are consistently reported in autism spe
46 genetically younger parts of the cerebellum, crus I and crus II.
47 r cortical Golgi cells and Purkinje cells in Crus I and II of the posterior lobe cerebellar hemispher
48 s of the cerebellar hemispheres bilaterally (Crus I and lobule VI).
49 erived primarily from reduced gray matter in crus I and lobules VI, VIIB, and VIIIA.
50 bellar lesion volume, cerebellar Lobules VI, Crus I and VIIIa atrophy being independent predictors of
51  on the effective connectivity of cerebellar crus I during visual attention.
52                           The involvement of Crus I is consistent with an emerging picture in which i
53  that zone, consistent with the existence in crus I of olivo-cortico-nuclear microcomplexes.
54 tracers were microinjected into the folia of crus I of the cat cerebellum to investigate spatial loca
55 n of the connections from V5 to PPC and from crus I to V5 by attention.
56 traction of total cerebellar, lobule IV, and Crus I volumes with additional X- or Y-chromosomes; X-sp
57 sensitivity of V5 populations to inputs from crus I was increased under attention.
58 t superior cerebellum (hemispheric lobule VI/Crus I) impairs verbal working memory performance.
59 llum, specifically in hemispheric lobule VI, Crus I, and VIIb.
60 rdination were variably associated with HVI, Crus I, Crus II, HVII B and/or HIX.
61  (p<0.001) and left lobule VI (p<0.01), left crus I, right VIIb and entire cerebellum (p<0.05 for eac
62 he cerebellar nuclei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI ipsilateral to t
63 e to the peripherally generated responses in Crus I.
64 w stimulation evokes a beam-like response in Crus I.
65 as primarily correlated with degeneration of Crus I.
66 erse kinematic transformation and sparing of Crus I.
67 recordings were obtained from the D1 zone of crus I.
68 xist within each of the zones present within crus I.
69 S, anodal tDCS increased activation in right Crus I/II during semantic prediction and enhanced restin
70 l connectivity of executive function-related Crus I/II in the cerebellum was analysed.
71 ing activity in the human lateral cerebellar Crus I/II modulates the cerebral default mode network, w
72 al gyrus (z score = 4.31, P < .001), and the crus I/II of the cerebellum (z score = 3.77, P < .001),
73 tive sentences increased activation in right Crus I/II of the cerebellum.
74 deling, we hypothesize that right cerebellar Crus I/II supports prediction of upcoming sentence conte
75        We found that activation within right Crus I/II was enhanced when semantic predictions were ma
76 y lower functional connectivity of the right Crus I/II with the left dorsolateral prefrontal cortex.
77 ght cerebellar lobules VI and VII (including Crus I/II) are engaged during a variety of language proc
78 erolateral portions of the cerebellum (right Crus I/II) contribute to language processing, but the na
79 owed marked atrophy in AD, ALS, FTD and PSP (Crus I/II), and MSA and PSP (lobules I-IV), respectively
80 ependent signal in the posterior cerebellum (Crus I/II).
81 ermis lobule VII or right lateral cerebellar Crus I/II, subregions that prominently couple to the dor
82 indazole attenuated the vascular response to crus II activation in wild-type mice but not in D2-null
83 stimulation induces patch-like activation of Crus II and GABAA antagonists fail to convert this patch
84 bing fibre responses were evoked in parts of crus II and paramedian lobule by stimulation of corticof
85 irs located mainly in the A2 and C1 zones in crus II and the paramedian lobule.
86  peripherally evoked patch-like responses in Crus II are aligned between parasagittal bands of EAAT4.
87 ce, upper lip stimulation increased BFcrb in crus II by 32 +/- 2%.
88               The field potentials evoked in crus II by upper lip stimulation did not differ between
89 put from Purkinje cells located primarily in Crus II of the ansiform lobule.
90 e show that the majority of granule cells in Crus II of the cerebrocerebellum receive sensory-evoked
91 tivity of neighboring individual MLIs in the Crus II region of awake female mice during two types of
92                            Purkinje cells in Crus II showed continuous firing at relatively high rate
93                                              Crus II, a region of the cerebellar cortex that receives
94 clei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI ipsilateral to the conditioned e
95 ffect the climbing fibre responses evoked in crus II, and produced a relatively small reduction of th
96 n were variably associated with HVI, Crus I, Crus II, HVII B and/or HIX.
97 xpansion of total cerebellum, flocculus, and Crus II-lobule VIIIB volumes in males) and SCA (contract
98  or Y-chromosomes; X-specific contraction of Crus II-lobule VIIIB).
99 rea 46 project to granule cells primarily in Crus II.
100 ers (EAATs) generates beam-like responses in Crus II.
101  younger parts of the cerebellum, crus I and crus II.
102 cell column, we recorded simultaneously from crus IIA areas and from left and right vermal lobule IX,
103 ranule cell layer (GCL) of cerebellar folium Crus IIa as freely moving rats engaged in a variety of n
104  of all four areas, i.e., the left and right crus IIA as well as the left and right lobule IX, can fi
105 ke synchrony observed between left and right crus IIA could indeed be mediated in part through couple
106  fractured tactile cerebellar map within the crus IIa folia of the cerebellar hemispheres reorganizes
107  the 1000-4500 Hz multiunit responses in the Crus IIa GCL of awake rats are correlated with tactile i
108 ossy fiber and climbing fiber projections to crus IIa in the lateral hemispheres of the rat cerebellu
109 mbing fiber activation of the left and right crus IIA in the rat can be explained by (1) bilateral in
110 mined in the tactilely responsive regions of crus IIa in the rat, the results show that SI influences
111 imultaneous recordings of the left and right crus IIA of the cerebellar cortex in the rat have demons
112 the tactile map in the granule cell layer of crus IIa reorganized, with representations of intact str
113 rents of the olivary regions that project to crus IIA was studied using Phaseolus vulgaris leucoagglu
114                         First, more areas of crus IIa were nonresponsive in animals lesioned later in
115 e structural role of RyR3 in the assembly of CRUs in 1B5 cells independently expressing either RyR1 o
116                                      Special CRUs in cardiac muscle are constituted by SR domains bea
117  in three vineyards, representing different 'crus' in the cultivation areas of Barolo, Barbaresco and
118 cy of marrow competitive repopulation units (CRUs) increased approximately 2-fold from 2 months to 2
119 ayed afterdepolarizations, Ca release units (CRUs) interact with not just the immediately adjacent CR
120            The muscle fascicles of the right crus of diaphragm which form the esophageal hiatus are a
121 tract-based spatial statistics suggested the crus of the fornix as a focus for this relationship.
122 ately 20 pA) through the Ca2+ release units (CRUs) of the sarcoplasmic reticulum (SR); 2) stochastic
123 alamus, right posterior cingulum, and fornix crus (seven studies; largest cluster, 980 voxels; z = 2.
124 fronto-occipital fasciculus, and left fornix crus (six studies; 323 voxels; z = 1.7; P = .001).
125  the canal pouch from the prospective common crus to a canal-like fate.
126 release and local recruitment of neighboring CRUs to fire more synchronously.
127 -SR Ca diffusion from neighboring non-firing CRUs to the firing CRUs, which helps to maintain the loc
128 anization and RyR2 orphaning (5000 of 20 000 CRUs) uncovered a local mechanism of delayed subcellular
129 ng, while the distal peak separated from the crus/upper-peak by 1.1 cm between FI and FE.
130 eract with not just the immediately adjacent CRUs via Ca diffusion, but also further CRUs via fast (
131 cent CRUs via Ca diffusion, but also further CRUs via fast ( approximately 0.1 ms) changes in Vm medi
132 ears of age, the BM homing efficiency of old CRUs was approximately 3-fold lower than that of young C
133 ceptible to RA treatment, whereas the common crus was particularly resistant, suggesting that the mol
134                                  One enzyme, CruS, was only distantly related to CrtD desaturases, wa
135 the spark frequency one would observe if the CRUs were incommunicado; 2), the coupling strength, whic
136 a2+ currents through the Ca2+ release units (CRUs) were approximately 1-2 pA, producing sparks with p
137 oupling is governed by Ca(2+) release units (CRUs) whereby Ca(2+) influx via L-type Ca(2+) channels (
138 om neighboring non-firing CRUs to the firing CRUs, which helps to maintain the local SR Ca concentrat
139  diffusively coupled Ca(2)(+) release units (CRUs) with fixed refractory period.

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