ライフサイエンスコーパス: crush injury

1 to a level equivalent of that observed with crush injury.

2 ation in animal models following axotomy and crush injury.

3 protected from degeneration following nerve crush injury.

4 emyelination is severely delayed after nerve-crush injury.

5 neration following retro-orbital optic nerve crush injury.

6 arly axonal regeneration after sciatic nerve crush injury.

7 of mouse sciatic nerve distal segment after crush injury.

8 egrowth tapered off around 2 weeks after the crush injury.

9 s gangrene, necrotizing fasciitis, and acute crush injury.

10 ation and delayed RGC loss after optic nerve crush injury.

11 axons into the spinal cord after dorsal root crush injury.

12 ced regrowth of axons after an in vivo nerve crush injury.

13 lumbar spinal cord, following sciatic nerve crush injury.

14 10 is rapidly expressed by macrophages after crush injury.

15 nces regeneration of the sciatic nerve after crush injury.

16 ce (C57BL/6J) were unilaterally subjected to crush injury.

17 ances motor nerve regeneration after a nerve crush injury.

18 after 7 d in vivo after a peripheral axonal crush injury.

19 nerves starting between 2 and 3 weeks after crush injury.

20 us following either sciatic nerve section or crush injury.

21 unctional recovery after incomplete cervical crush injury; (2) either of these cell types is preferab

22 After large crush injuries across the entire spinal cord, ependyma-d

23 y after nerve transection, crush injury, and crush injury after a previous "conditioning" lesion.

24 ave now investigated whether a sciatic nerve crush injury alters the behavioral response in rats to t

25 C57Bl/6 mice received crush injuries and were allowed to survive for 6 weeks t

26 ating that APC can diminish complications of crush injury and leukocyte damage to lung and other tiss

27 terminating inflammation after sciatic nerve crush injury and promoting regeneration.

28 reviously suggested in nerve transection and crush injuries, and now demonstrated in CCI neuropathy,

29 lowing sterile injury, ischemia reperfusion, crush injury, and autoimmune-mediated tissue damage.

30 egenerative ability after nerve transection, crush injury, and crush injury after a previous "conditi

31 In the carotid crush injury animals, biodistribution analysis confirmed

32 After crush injury, animals received either 8.3 mg/kg (332,000

33 cidence may be increased as a result of mass crush injury casualties or prolonged evacuation times.

34 ple forms of sciatic nerve injury, including crush injury, chronic constriction injury, and axotomy.

35 Equivalent moderate crush injuries combined with ablation of reactive astroc

36 sory nerve regeneration achieved after nerve crush injury compared with untreated diabetic rats.

37 RGCs subjected to optic nerve crush injury demonstrated more rapid neurite outgrowth i

38 ral hood ipsilateral or contralateral to the crush injury elicited synaptic responses in RS neurons o

39 uctures and swirling debris inflict numerous crush injuries, fractures, and serious wounds.

40 s surviving blunt and penetrating trauma and crush injuries have subsequent complications that lead t

41 Moderate crush injuries in control mice caused focal tissue disru

42 ered by i.v. injection 1 hr post-spinal-cord crush injury in an effort to prevent inflammatory angiog

43 Remyelinated axons were evident 20 d after crush injury in control mice, yet were largely absent in

44 mmation and regeneration after sciatic nerve crush injury in mice.

45 regeneration, using a model of sciatic nerve crush injury in mice.

46 Sciatic nerve crush injury in rats induced expression of the ER chaper

47 tion, acute kidney injury by gentamicin, and crush injury in spinal cord cells.

48 ronal death in Nf1+/- mice after optic nerve crush injury is also attenuated by rolipram treatment in

49 nal regrowth into the distal zone of a nerve crush injury is not markedly impaired in cyclin D1-/- mi

50 Optic nerve crush injury leads to rapid elevation of DLK protein, fi

51 when combined with retro-orbital optic nerve crush injury, lengthy growth of severed retinal ganglion

52 After crush injury, LRP-1 is lost from the axoplasm and substa

53 Similarly, in the optic nerve crush injury model, MAB228 and AG490 neutralizes dominan

54 Wistar rats (n=26) underwent either carotid crush injury (mural thrombosis model) or embolic stroke

55 ), myonecrosis due either to polymyositis or crush injuries (n = 12), and septic shock (n = 6); resul

56 wine model, atherosclerosis was developed by crush injury of one carotid and one femoral artery and i

57 n effects of the growth factor artemin after crush injury of the dorsal spinal nerve roots in rats.

58 Following a unilateral crush injury of the right trigeminal root, escape behavi

59 Following unilateral crush injury of the right trigeminal root, HRP labeling

60 Deceleration and crushing injuries of the chest may traumatize the thorac

61 Mice received complete crush injury or control laminectomy at either thoracic l

62 ognition of potential complications, such as crushing injuries or nerve damage, must be sought.

63 ing with or without a right atrial free wall crush injury, respectively.

64 of MMP-9-PEX into sciatic nerves, 24 h after crush injury, robustly increased phosphorylation of ERK1

65 In response to crush injury, sciatic nerves in scLRP1(-/-) mice showed

66 t of adult mice with LiCl after facial nerve crush injury stimulated the expression of myelin genes,

67 cell body to axon predominantly after nerve crush injury, suggesting that it encodes a growth-associ

68 ofile of laser ablation is more similar to a crush injury than the precision removal of individual ce

69 Following optic nerve crush injury, the mpz:egfp transgene was strongly up-reg

70 After dorsal root crush injury, the ROCKII(-/-) mice recovered use of the

71 overy occurred in mice after a sciatic nerve crush injury, there was little return of motor function

72 Within 3 days after an optic nerve crush injury to one eye, primary transcript levels of NF

73 Although remote tissue ischemia, such as a crush injury to the hindlimb, may result in P-selectin-m

74 After crush injury to the optic nerve, elevated intraocular pr

75 s were generated and subjected to unilateral crush injury to the sciatic nerve.

76 One day sciatic nerve crush injury triggered a robust increase in UPR-associat

77 Crush injury up-regulates kon expression downstream of N

78 Using an infraorbital optic nerve crush injury, we show that reducing beta-catenin-depende

79 Spinal cord crush injuries were produced at T9 in two inbred strains

80 Crush injuries were produced at the T8 level by using an

81 n in the dorsal horn reverts to normal after crush injury when regeneration occurs but persists after

82 Except in one instance of crush injury with progressive intracerebral damage, and

83 ession profiles during development and after crush injury with those of Egr2Lo/Lo Schwann cells revea