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1 taltic reflex activity, and proliferation of crypt epithelial cells.
2 ly expressed in the proliferating intestinal crypt epithelial cells.
3 ly higher in intestinal villus compared with crypt epithelial cells.
4 Tegaserod increased proliferation of crypt epithelial cells.
5 s also were unable to induce swelling in the crypt epithelial cells.
6 s, hematopoietic cells, and gastrointestinal crypt epithelial cells.
7 ulator primarily expressed in the intestinal crypt epithelial cells.
8 ithelial fibroblasts and in villous, but not crypt, epithelial cells.
9 antiapoptotic protein bcl-2 was increased in crypt epithelial cells after growth hormone treatment.
10 solateral membrane K+ channels of intestinal crypt epithelial cells also participate in secretagogue-
12 ced apoptosis was detected only in wild-type crypt epithelial cells, and not in the p21-deficient col
13 anges is a significant loss of proliferative crypt epithelial cells as revealed by BrdU or Ki67 stain
14 l mucosa by Northern analysis and located in crypt epithelial cells as well as in myenteric neurons b
15 Receptor density was greater in surface vs crypt epithelial cells, but no significant differences w
16 This suggests that colon cancer and possibly crypt epithelial cells can modulate the effects of Cdx2
17 oped significantly greater colon surface and crypt epithelial cell colonization, surface epithelial c
18 tion of lamina propria mononuclear cells and crypt epithelial cells, crypt expansion and villus blunt
20 is was paralleled by decreased expression of crypt epithelial cell genes involved in cell cycle, prol
23 2 are rapidly induced after IR in intestinal crypt epithelial cells in mice, but CUGBP2 protein expre
24 s localization along the lateral membrane of crypt epithelial cells in normal human colonic epitheliu
25 ensely expressed in a patchy distribution in crypt epithelial cells in proximity to lymphocytic infil
26 was no difference in the number of apoptotic crypt epithelial cells in the grafts of control and CD8-
27 ibodies directed against NKCC from the human crypt epithelial cell line, T84, we define its surface l
29 the apical and lateral membranes of colonic crypt epithelial cells, mediates N-formylpeptide-depende
33 ption factor that is highly expressed in the crypt epithelial cells of the intestine and plays a crit
35 tial gradients of these factors insures that crypt epithelial cell proliferation and development proc
36 6 and Stat3 activation, as well as increased crypt epithelial cell proliferation in normal colon muco
39 ypts, villus epithelium, or undifferentiated crypt epithelial cells, suggesting a lineage-specific ro
41 e-sensitive) uptake of 3H-5-HT by intestinal crypt epithelial cells was found by radioautography.
42 itis activity and rescued MLH1 expression in crypt epithelial cells, which was associated with increa
43 ced apoptosis and increased proliferation of crypt epithelial cells while increasing apoptosis of lam
44 ctance regulator was internalized in colonic crypt epithelial cells without a change in overall expre
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