ライフサイエンスコーパス: crystal violet

1 les with volumes as large as 461 A(3) (e.g., crystal violet).

2 nce was assessed by visual experiments using crystal violet.

3 Adherent cells were quantified using crystal violet.

4 95% relative to the energy transfer to free crystal violet.

5 le salt deoxycholate and the hydrophobic dye crystal violet.

6 ng likewise was decreased in the presence of crystal violet.

7 ch was verified using a low concentration of crystal violet (10(-)(5)M) as the probe molecule.

8 ur chromophores were examined: Rhodamine 6G, crystal violet, a cyanine dye, and a cationic donor-acce

9 t of energy transfer donors to the acceptor, crystal violet, a noncompetitive antagonist of the nAChR

10 iocin, and dyes such as ethidium bromide and crystal violet and increased accumulation of radioactive

11 It is concluded that crystal violet and other dyes of similar structure bind

12 Organic dyes such as crystal violet and Rhodamine B, the nucleobase cytosine,

13 Viability tests were performed with crystal violet and ROS tests with DCFH-DA.

14 Because of their optical properties, crystal violet and several of the other homologous dyes

15 istance to several antimicrobials, including crystal violet and streptomycin (this phenotype could al

16 ta sets {the reduction of chloranil by leuco crystal violet and the reduction of morphinone reductase

17 ith attached P. gingivalis were stained with crystal violet, and attachment was expressed based on dy

18 hree-dye mixture composed of methylene blue, crystal violet, and rhodamine 6G for positive ion mode d

19 ssembly techniques to fabricate a pattern of crystal violet as a standard reticle slide for assessing

20 e fluorescent NCIs ethidium, quinacrine, and crystal violet as well as [(3)H]thienylcyclohexylpiperid

21 age of different dye molecules (pyranine and crystal violet) as well as avidin through melittin induc

22 We used a crystal violet assay and confocal laser scanning microsc

23 ective inhibitors, estimated cell numbers by crystal violet assays, measured caspase activity by clea

24 tumor cell survival, as measured by MTT and crystal violet assays, regardless of IGF1 pre-treatment.

25 ducts necessary for biofilm development in a crystal violet-based assay involving 24-well tissue cult

26 ation by these M. catarrhalis strains in the crystal violet-based assay.

27 ly 3,000 transposon insertion mutants in the crystal violet-based biofilm assay system yielded six mu

28 Crystal violet binding blocked agonist-induced 22Na ion

29 with 1-2 negative charges within 8 A of the crystal violet binding site.

30 Following crystal violet biofilm assays for single metal ion solut

31 e to measure binding, we determined that one crystal violet bound per receptor with a dissociation co

32 The flux of the crystal violet cation across the membrane is simultaneou

33 In the presence of sbeta-CD, the crystal violet-containing buffer was blue and was deflec

34 In the absence of sbeta-CD, the crystal violet-containing buffer was reddish/purple and

35 the binding site location for the fluorophor crystal violet (CrV), a noncompetitive antagonist of the

36 fects, as well as bactericidal activity with crystal violet (CV) coated polyurethane.

37 ]arene (SC4) interacts with the aromatic dye crystal violet (CV) to form complexes with stoichiometri

38 e and gram-negative strains by staining with crystal violet (CV).

39 mpounds, N-methyl mesoporphyrin IX (NMM) and Crystal Violet (CV).

40 Lysine, peptides, crystal violet dye, and a biotin conjugate are found to

41 Anionic I3(-) reacts with cationic crystal violet dye, and the product is extracted into 1-

42 Brilliant green and crystal violet dyes were the molecular probes, and the e

43 henylmethane dyes (rose bengal, rhodamine B, crystal violet, ethyl violet, fast green fcf, and brilli

44 relative sensitivities are malachite green > crystal violet > quinaldine red > ascorbate reduction >

45 ing and chemical imaging of the cationic dye crystal violet in inked lines on glass and for lipid dis

46 absorbance and fluorescence spectroscopy of crystal violet in order to elucidate the binding mechani

47 potentials by the bound acceptor fluorophore crystal violet, its binding site was first localized wit

48 plification products are detected with leuco crystal violet (LCV) dye by eye without a need for instr

49 GCN), basic fuchsin leuconitrile (BFCN), and crystal violet leucomethyl (CVMe) and leucobenzyl (CVBn)

50 The photochemical reactions of crystal violet leuconitrile (CVCN) were investigated by

51 nt of a medical grade silicone incorporating crystal violet, methylene blue and 2 nm gold nanoparticl

52 ive SERS by measuring the areal densities of crystal violet molecules embedded in an ultrathin spin-o

53 same tissues with metachromatic dyes such as crystal violet or with the cotton dye Congo red (particu

54 rowth and was more susceptible to killing by crystal violet, osmotic shock, and select carbapenem ant

55 adical anion of 2-chloranthraquinone and the crystal violet radical, which display improved resolutio

56 The patterned crystal violet reticle was also used to diagnose issues

57 One dye with high affinity, crystal violet, revealed a greater than 200-fold fluores

58 d by using a microtiter plate assay with the crystal violet staining method, and the presence of the

59 y continuous passaging at a 1:3 split and by crystal violet staining of confluent dishes.

60 HMC adhered strongly (quantified using crystal violet staining) to collagen IV and collagen I (

61 ve, cell outgrowth ([3H]thymidine uptake and crystal violet staining) was also tested.

62 Crystal violet staining, acryflavin agglutination, and p

63 ree independent measures: Congo red binding, crystal violet staining, and confocal laser scanning mic

64 s were evaluated by fluorescence microscopy, crystal violet staining, and the MTS assay.

65 l strains were identified and quantitated by crystal violet staining.

66 mal and distal tubular cells, as observed by crystal violet staining.

67 are consistent with preferential binding of crystal violet to the desensitized conformation of the A

68 ollected and visualized with the addition of crystal violet to the separation buffer.

69 Cell proliferation was monitored by crystal violet uptake, and pericyte migration was assess

70 containing buffer was reddish/purple and the crystal violet was deflected cathodically in the chamber

71 from a neutral Tb3+ -chelate to nAChR-bound crystal violet was reduced 95% relative to the energy tr

72 This result indicated that crystal violet was strongly shielded from solvent when b

73 r example, spectra of glucose, arginine, and crystal violet were obtained with no observed interferen

74 pigment, and to regulate binding to the dye crystal violet, whereas motility, flagellar secretion, a